Zobrazeno 1 - 3
of 3
pro vyhledávání: '"Vincent Cirotteau"'
Autor:
Layal Maatouk, Anne-Claire Compagnion, Maria-Angeles Carrillo-de Sauvage, Alexis-Pierre Bemelmans, Sabrina Leclere-Turbant, Vincent Cirotteau, Mira Tohme, Allen Beke, Michaël Trichet, Virginie Bazin, Bobby N. Trawick, Richard M. Ransohoff, François Tronche, Bénédicte Manoury, Sheela Vyas
Publikováno v:
Nature Communications, Vol 9, Iss 1, Pp 1-15 (2018)
Inflammation is a component of Parkinson’s disease. Here the authors show that activation of Toll-like receptor 9 controlled by microglial glucocorticoid receptor signaling, contributes to dopamine neuron loss in a model of Parkinson’s disease.
Externí odkaz:
https://doaj.org/article/7e620af317d44ae895a31e6a05dc755b
Autor:
Layal Maatouk, Anne-Claire Compagnion, Maria-Angeles Carrillo-de Sauvage, Alexis-Pierre Bemelmans, Sabrina Leclere-Turbant, Vincent Cirotteau, Mira Tohme, Allen Beke, Michaël Trichet, Virginie Bazin, Bobby N. Trawick, Richard M. Ransohoff, François Tronche, Bénédicte Manoury, Sheela Vyas
Publikováno v:
Nature Communications, Vol 9, Iss 1, Pp 1-1 (2018)
The originally published version of this Article contained an error in the subheading “Microglial GR does not affect DN loss triggered by TLR4 and TLR7,” which was incorrectly given as “Microglial GR does affect DN loss triggered by TLR2 and TL
Externí odkaz:
https://doaj.org/article/74bed7d28ef741b9857243929cdbec2e
Autor:
Bobby N. Trawick, Alexis-Pierre Bemelmans, Sheela Vyas, Vincent Cirotteau, Mira Tohme, Virginie Bazin, Sabrina Leclere-Turbant, Bénédicte Manoury, Maria-Angeles Carrillo-de Sauvage, Layal Maatouk, Michaël Trichet, Anne-Claire Compagnion, Allen Beke, Richard M. Ransohoff, François Tronche
Publikováno v:
Nature Communications, Vol 9, Iss 1, Pp 1-1 (2018)
Nature Communications
Nature Communications
Inflammation is a characteristic feature of Parkinson’s disease (PD). We examined the role of TLR9 and its regulation by glucocorticoid receptors (GRs) in degeneration of substantia nigra dopamine neurons (DNs). TLR9 agonist, CpG-ODN, induced DN de