Zobrazeno 1 - 10
of 46
pro vyhledávání: '"Viktoria, Denes"'
Autor:
Etelka Pöstyéni, Viktoria Denes, Adrienn Mester, Andrea Kovács-Valasek, Robert Gábriel, György Sétáló
Publikováno v:
International Journal of Molecular Sciences, Vol 22, Iss 444, p 444 (2021)
International Journal of Molecular Sciences
Volume 22
Issue 1
International Journal of Molecular Sciences
Volume 22
Issue 1
Retinal aging is the result of accumulating molecular and cellular damage with a manifest decline in visual functions. Somatostatin (SST) and pituitary adenylate cyclase-activating polypeptide (PACAP) have been implicated in neuroprotection through r
Autor:
Etelka Pöstyéni, György Sétáló, Andrea Kovács-Valasek, Robert Gábriel, Adrienn Mester, Viktoria Denes
Publikováno v:
Cells Tissues Organs
Imbalance of homeostasis causes permanent changes in the body with time. The central nervous system is especially prone to these changes since it possesses limited regenerative capacity. In the retina, neurons are damaged during the aging process, an
Autor:
Viktoria Denes, Andrea Kovács-Valasek, Zsolt Nyisztor, Orsolya Hideg, Robert Gábriel, Gergely Berta
Publikováno v:
Neuroscience. 385:59-66
PACAP1–38, a ubiquitous and multifunctional regulator has been in the focus of neurotoxicity research due to its impressive neuroprotective potential. Although the literature extensively demonstrated its repressive effect on the apoptotic machinery
Autor:
Kathleen Marinelli, James K. Kubilus, Peter Geck, Andrew Makarovskiy, Christine Kyritsis, Viktoria Denes
Publikováno v:
Cancer Research. 80:2714-2714
Introduction: Progression into metastatic disease is a critical step in most malignancies. Cancer cells may enter circulation very early (circulating tumor cells or CTCs), but distant metastases are disseminated only by cancer stem cells (CSCs) that
Autor:
Krisztina Szabadfi, Andrea Kovács-Valasek, Robert Gábriel, György Sétáló, Andrea Tamas, Dóra Reglődi, Aliz Szabo, Balint Szalontai, Viktoria Denes, Peter Kiss
Publikováno v:
Neuroscience. 348:1-10
Pituitary adenylate cyclase activating polypeptide (PACAP) is a neurotrophic and neuroprotective peptide. PACAP and its receptors are widely distributed in the retina. A number of reports provided evidence that PACAP is neuroprotective in retinal deg
Publikováno v:
Journal of Clinical Medicine, Vol 8, Iss 9, p 1488 (2019)
Journal of Clinical Medicine
Journal of Clinical Medicine
Emerging from the depths of evolution, pituitary adenylate cyclase-activating polypeptide (PACAP) and its receptors (i.e., PAC1, VPAC1, VPAC2) are present in multicellular organisms from Tunicates to humans and govern a remarkable number of physiolog
Autor:
Monika Lakk, Gergely Berta, Viktoria Denes, Peter Geck, Zsolt Nyisztor, Zoltan Godri, Robert Gábriel, Orsolya Hideg
Publikováno v:
Investigative ophthalmologyvisual science. 60(2)
Purpose PACAP1-38, a member of the secretin/glucagon superfamily, is expressed in the developing retina with documented neuroprotective effects. However, its function in retinal cell differentiation has yet to be elucidated. Our goals, therefore, wer
Publikováno v:
Cancer Research. 79:3766-3766
INTRODUCTION One of the unsolved problems of cancer is its progression into systemic, metastatic disease. Malignant cells are released into circulation very early (circulating tumor cells or CTCs), but distant metastases are disseminated only by canc
Autor:
Monika Lakk, István Karádi, Pál Jáksó, Laszlo Mandel, Peter Geck, Andrew Makarovskiy, Viktoria Denes, Szabolcs Szappanos, László Gráf
Publikováno v:
International Journal of Cancer. 136:1528-1536
Cancer hypoxia correlates with therapeutic resistance and metastasis, suggesting that hypoxic adaptation is a critical survival advantage for cancer stem cells (CSCs). Hypoxic metabolism, however, may be a disadvantage in aerobic circulation as the e
Publikováno v:
Cell and Tissue Research. 355:279-288
Pituitary adenylate cyclase-activating polypeptide (PACAP), a member of the secretin/glucagon/vasoactive intestinal peptide family, exerts various effects on neuronal development as mediated by the differential expression of PAC1 receptor (PAC1-R) is