Zobrazeno 1 - 10
of 11
pro vyhledávání: '"Vijole Ottosson"'
Autor:
Lars Rönnblom, Kristina Gemzell-Danielsson, Gunnar Bergman, Sven-Erik Sonesson, Marie Wahren-Herlenius, Maija-Leena Eloranta, Malin Hedlund, Gudny Ella Thorlacius, Margarita Ivanchenko, Vijole Ottosson, Nikolaos Kyriakidis, Linda Lagnefeldt, Joanna Tingström, Allan Sirsjö, Anders A Bengtsson, Emma Aronsson, Alexander Espinosa
Publikováno v:
RMD Open, Vol 6, Iss 1 (2020)
Objective In utero exposure of the fetus to Ro/La autoantibodies may lead to congenital heart block (CHB). In the mother, these autoantibodies are associated with activation of the type I interferon (IFN)-system. As maternal autoantibodies are transf
Externí odkaz:
https://doaj.org/article/a7f6f921fc11498f9c20e3fe8af9f0a7
Autor:
Eva Sverremark-Ekström, Marie Wahren-Herlenius, Sophia Björkander, Katarina Bremme, Joanna Tingström, Malin Hedlund, Gudny Ella Thorlacius, Margarita Ivanchenko, Lauro Meneghel, Kristina Gemzell-Danielsson, Vijole Ottosson, Sven-Erik Sonesson, Karine Chemin, Amina Ossoinak
Publikováno v:
Annals of the Rheumatic Diseases. 80:194-202
ObjectiveCongenital heart block (CHB) with immune cell infiltration develops in the fetus after exposure to maternal Ro/La autoantibodies. CHB-related serology has been extensively studied, but reports on immune-cell profiles of anti-Ro/La-exposed ne
Autor:
Sabrina Meisgen, Malin Hedlund, Aurelie Ambrosi, Lasse Folkersen, Vijole Ottosson, David Forsberg, Gudny Ella Thorlacius, Luca Biavati, Linn Strandberg, Johannes Mofors, Daniel Ramskold, Sabrina Ruhrmann, Lauro Meneghel, William Nyberg, Alexander Espinosa, Robert Murray Hamilton, Anders Franco-Cereceda, Anders Hamsten, Tomas Olsson, Lois Greene, Per Eriksson, Kristina Gemzell-Danielsson, Stina Salomonsson, Vijay K Kuchroo, Eric Herlenius, Ingrid Kockum, Sven-Erik Sonesson, Marie Wahren-Herlenius
Publikováno v:
Annals of the Rheumatic Diseases
ObjectiveNeonatal lupus erythematosus (NLE) may develop after transplacental transfer of maternal autoantibodies with cardiac manifestations (congenital heart block, CHB) including atrioventricular block, atrial and ventricular arrhythmias, and cardi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::648421d9fa4958f253f6b7284d8ea0d8
https://hdl.handle.net/11250/3035404
https://hdl.handle.net/11250/3035404
Autor:
Sven Erik Sonesson, Vijole Ottosson, Lars Rönnblom, Allan Sirsjö, Gunnar Bergman, Anders A. Bengtsson, Malin Hedlund, Kristina Gemzell-Danielsson, Marie Wahren-Herlenius, Maija-Leena Eloranta, Nikolaos C Kyriakidis, Gudny Ella Thorlacius, Margarita Ivanchenko, Joanna Tingström, Linda Lagnefeldt, Alexander Espinosa, Emma Aronsson
Publikováno v:
RMD Open
RMD Open, Vol 6, Iss 1 (2020)
RMD Open, Vol 6, Iss 1 (2020)
ObjectiveIn utero exposure of the fetus to Ro/La autoantibodies may lead to congenital heart block (CHB). In the mother, these autoantibodies are associated with activation of the type I interferon (IFN)-system. As maternal autoantibodies are transfe
Autor:
Malin Hedlund, Madhanagopal Anandapadamanaban, Lars Ottosson, Vijole Ottosson, A. Ambrosi, Maria Sunnerhagen, Ariela Hoxha, Amelia Ruffatti, Sven-Erik Sonesson, Marie Wahren-Herlenius
Publikováno v:
Clinical and Experimental Immunology. 186:284-291
Summary Congenital heart block (CHB) is a potentially lethal condition characterized by a third-degree atrioventricular block (AVB). Despite anti-Ro52 antibodies being detected in nearly 90% of mothers of affected children, CHB occurs in only 1–2%
Autor:
Claus Johansen, Lars Iversen, Alexander Espinosa, Hanne Vinter, Line Raaby, Vijole Ottosson, Marie Wahren-Herlenius, Ane Langkilde
Publikováno v:
Vinter, H, Langkilde, A, Ottosson, V, Espinosa, A, Wahren-Herlenius, M, Raaby, L, Johansen, C & Iversen, L 2017, ' TRIM21 is important in the early phase of inflammation in the imiquimod-induced psoriasis-like skin inflammation mouse model ', Experimental Dermatology, pp. 713-720 . https://doi.org/10.1111/exd.13269
Tripartite motif-containing protein 21 (TRIM21) regulates pro-inflammatory cytokines and type I interferons and acts as an autoantigen in certain autoimmune diseases, but TRIM21 has not been investigated in psoriasis. It has been suggested that TRIM2
Autor:
Ariela Hoxha, Stina Salomonsson, Gunnar Bergman, Aurélie Ambrosi, Joanna Tingström, Vijole Ottosson, Amanda Skog, Marie Wahren-Herlenius
Aim To investigate the correlation between maternal autoantibodies and age at diagnosis of isolated complete atrioventricular (AV) block (CAVB) and to study signs of late progression of foetal immune-mediated insults in cases of postnatally diagnosed
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::59ff6055796fd6ace6a676e1abb81a9a
http://hdl.handle.net/11577/3342698
http://hdl.handle.net/11577/3342698
Autor:
Maria Sunnerhagen, Ariela Hoxha, A. Ruffatti, Marta Tonello, Vijole Ottosson, Marie Wahren-Herlenius, Leonardo Punzi, M. Anandapadmanaban
Publikováno v:
Annals of the Rheumatic Diseases. 72:A462.3-A463
Background The presence of maternal autoantibodies directed against the amino acids (aa) 200–239 stretch of Ro52 (Ro52-p200) is associated with high risk of foetal CHB to develop. Objectives To investigate the role of anti-Ro52 antibodies in the pa
Autor:
Leonardo Punzi, T. Del Ross, Marie Wahren-Herlenius, Malin Hedlund, A. Ruffatti, Ariela Hoxha, Vijole Ottosson, Lars Ottosson, Sven-Erik Sonesson
Publikováno v:
Annals of the Rheumatic Diseases. 72:A78.3-A79
Background Congenital atrioventricular block (CHB) depend on Ro/SSA and/or La/SSB-autoantibody mediated inflammation and subsequent fibrosis of the atrioventricular node. Despite the association of maternal Ro/SSA and La/SSB autoantibodes with congen
Autor:
Myriam Facchinetti, Susanna Brauner, Ariela Hoxha, Antonia Calligaro, A. Ruffatti, Marie Wahren-Herlenius, Maria Favaro, Leonardo Punzi, Vijole Ottosson
Publikováno v:
Annals of the Rheumatic Diseases. 72:A256.1-A256
Background Congenital atrioventricular block (CHB) is the most frequent manifestation of neonatal lupus and depends on anti-Ro/SSA and/or anti-La/SSB-mediated inflammation and subsequent fibrosis of the atrioventricular node. No therapy has yet been