Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Vijaya Balakrishnan"'
Autor:
Conan Kinsey, Vijaya Balakrishnan, Michael R. O’Dell, Jing Li Huang, Laurel Newman, Christa L. Whitney-Miller, Aram F. Hezel, Hartmut Land
Publikováno v:
Cell Reports, Vol 7, Iss 4, Pp 1143-1155 (2014)
Mutations in p53 and RAS potently cooperate in oncogenic transformation, and correspondingly, these genetic alterations frequently coexist in pancreatic ductal adenocarcinoma (PDA) and other human cancers. Previously, we identified a set of genes syn
Externí odkaz:
https://doaj.org/article/064bdbf94f78439b863c1ce0d9866588
Autor:
Harry A. Stern, Aslihan Ambeskovic, Hartmut Land, Matthew N. McCall, Vijaya Balakrishnan, Helene R. McMurray, Jordan Aldersley, Laurel Newman, Bradley N. Smith
Malignant cell transformation and the underlying genomic scale reprogramming of gene expression require cooperation of multiple oncogenic mutations. Notably, this cooperation is reflected in the synergistic regulation of downstream genes, so-called c
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::9d4472310f5f10d332c5593348abfdf1
https://doi.org/10.1101/2020.10.07.328542
https://doi.org/10.1101/2020.10.07.328542
Autor:
Jing Li, Jiyong Zhao, Stephen A. Spence, Brian Poligone, Joel Shapiro, Richard Burack, Craig T. Jordan, Raymond J. Deshaies, Hartmut Land, Randy Rossi, Luojing Chen, Mary Pulvino, Carla Casulo, David Oleksyn, Jonathan W. Friedberg, Vijaya Balakrishnan, George Compitello, Steven H. Bernstein
Publikováno v:
Oncotarget
In searching for small-molecule compounds that inhibit proliferation and survival of diffuse large B-cell lymphoma (DLBCL) cells and may, therefore, be exploited as potential therapeutic agents for this disease, we identified the commonly used and we
Autor:
Vijaya Balakrishnan, Michael R. O'Dell, Hartmut Land, Christa L. Whitney-Miller, Laurel Newman, Conan G. Kinsey, Jing Li Huang, Aram F. Hezel
Publikováno v:
Cell Reports, Vol 7, Iss 4, Pp 1143-1155 (2014)
SummaryMutations in p53 and RAS potently cooperate in oncogenic transformation, and correspondingly, these genetic alterations frequently coexist in pancreatic ductal adenocarcinoma (PDA) and other human cancers. Previously, we identified a set of ge
Autor:
Vijaya Balakrishnan, Kinsey Conan, Hartmut Land, Jing Li Huang, Aram F. Hezel, Michael R. O'Dell, Laurel Newman, Christa L. Whitney-Miller
Publikováno v:
Cancer Research. 75:PR09-PR09
A) Pancreatic ductal adenocarcinoma (PDA) depends on a marked reprogramming of metabolic pathways, including the acquisition of autophagy dependence, for survival and growth. How common mutations in PDA cause autophagy dependence as well as the timin