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Autor:
Flaviene F. Torres, Victoria S. Bernardo, Danilo G.H. Silva, Jéssika V. Okumura, Claudia R. Bonini-Domingos
Publikováno v:
Hematology, Transfusion and Cell Therapy, Vol 44, Iss 2, Pp 156-162 (2022)
Introduction: Studies have shown that the loss of the FOXO3 transcriptional function is involved in the pathophysiology of some chronic erythroid disorders, including beta-thalassemia (β-thal). Therefore, the single nucleotide polymorphism (SNP) rs3
Externí odkaz:
https://doaj.org/article/ca111f8e9f8745f285f2faa993b71e86