Zobrazeno 1 - 10
of 18
pro vyhledávání: '"Verónica Zaldivar"'
Autor:
Jimena Ferraris, Daniela Betiana Radl, Sandra Zárate, Gabriela Jaita, Guadalupe Eijo, Verónica Zaldivar, Carmen Clapp, Adriana Seilicovich, Daniel Pisera
Publikováno v:
PLoS ONE, Vol 6, Iss 7, p e21806 (2011)
The anterior pituitary is under a constant cell turnover modulated by gonadal steroids. In the rat, an increase in the rate of apoptosis occurs at proestrus whereas a peak of proliferation takes place at estrus. At proestrus, concomitant with the max
Externí odkaz:
https://doaj.org/article/dfcebb6554f44fe1b8938d90d5bae660
Autor:
María Laura Magri, Guadalupe Eijo, Verónica Zaldivar, Jimena Ferraris, Gabriela Jaita, Daniel Pisera, Daniela Radl, Sandra Zárate, Adriana Seilicovich, Valeria Romina Boti
Publikováno v:
Journal of Neuroendocrinology. 23:651-659
Nuclear factor-kappa B (NF-κB), an important pro-inflammatory factor, is a crucial regulator of cell survival. Both lipopolysaccharide (LPS) and tumour necrosis factor (TNF)-α activate NF-κB signalling. Oestrogens were shown to suppress NF-κB act
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::4a444e94d5786f0eace79dd3b9192f29
https://doi.org/10.1111/j.1365-2826.2011.02157.x
https://doi.org/10.1111/j.1365-2826.2011.02157.x
Autor:
Daniel Pisera, Sandra Zárate, Verónica Zaldivar, Adriana Seilicovich, Guadalupe Eijo, Jimena Ferraris, Daniela Radl, Gabriela Jaita, María Laura Magri
Publikováno v:
Neuroendocrinology. 93:106-113
Background: Estrogens are recognized modulators of pituitary cell renewal, sensitizing cells to mitogenic and apoptotic signals. Tumor necrosis factor-α (TNF-α) is a proinflammatory cytokine that plays an important role in tissue homeostasis modula
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a79b03fa0b49ba55b18f706fac40fec2
https://doi.org/10.1159/000323760
https://doi.org/10.1159/000323760
Autor:
L. Ferrari, Gabriela Jaita, Jimena Ferraris, Guadalupe Eijo, Sandra Zárate, Daniela Radl, Verónica Zaldivar, Daniel Pisera, Adriana Seilicovich
Publikováno v:
Endocrine. 39:21-27
We have previously reported that Fas activation induces apoptosis of anterior pituitary cells from rats at proestrus but not at diestrus and in an estrogen-dependent manner. In this study, we evaluated the effect of Fas activation on apoptosis of lac
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6fa1d674976d3068315f159b6237df74
https://doi.org/10.1007/s12020-010-9407-4
https://doi.org/10.1007/s12020-010-9407-4
Autor:
Sandra Zárate, Daniel Pisera, Guadalupe Eijo, Daniela Radl, Adriana Seilicovich, María Laura Magri, Verónica Zaldivar, Gabriela Jaita, Jimena Ferraris
Publikováno v:
Neuroendocrinology. 90:292-300
Background: Estrogens are recognized as acting as modulators of pituitary cell renewal, sensitizing cells to mitogenic and apoptotic signals, thus participating in anterior pituitary homeostasis during the estrous cycle. The balance of pro- and antia
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1f596d04b3b57d7dd3b9cac2544e4dc4
https://doi.org/10.1159/000235618
https://doi.org/10.1159/000235618
Autor:
Sandra Zárate, Maria G. Castro, Verónica Zaldivar, L. Ferrari, Marianela Candolfi, Adriana Seilicovich, Gabriela Jaita, Daniel Pisera
Publikováno v:
Endocrinology. 146:736-743
We previously reported that TNF-alpha-induced apoptosis of lactotropes is estrogen dependent and predominant at proestrus. Here we observed that TNF-alpha (50 ng/ml) failed to induce apoptosis of anterior pituitary cells from ovariectomized rats cult
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bf7d72977d7b544e2b07297c26cecc42
https://doi.org/10.1210/en.2004-1276
https://doi.org/10.1210/en.2004-1276
Autor:
Adriana Seilicovich, Verónica Zaldivar, Sandra Zárate, Gabriela Jaita, Marianela Candolfi, Daniel Pisera
Publikováno v:
Neuroendocrinology. 80:83-91
We previously reported that tumor necrosis factor-α (TNF-α) inhibits cell proliferation whereas it stimulates apoptosis of anterior pituitary cells in an estrogen-dependent manner. Also, we showed that nitric oxide (NO) mediates the inhibitory effe
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bcfbed59b3ff8ab935cd4d82166aa0a5
https://doi.org/10.1159/000081313
https://doi.org/10.1159/000081313
Autor:
Andrés E. Carrasco, Verónica Zaldivar, Silvia L. López, Adriana Seilicovich, A. De Laurentiis, S. Theas, Marianela Candolfi
Publikováno v:
Neuroendocrinology. 74:82-86
Tumor necrosis factor-α (TNF-α) is a pleiotropic cytokine that markedly affects neuroendocrine functions. This cytokine is expressed in the anterior pituitary where its receptors are also present. Nitric oxide (NO) is synthesized in gonadotropes an
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::eae97ef05b982727bfa3d0864036e7ac
https://doi.org/10.1159/000054673
https://doi.org/10.1159/000054673
Autor:
Adriana Seilicovich, Getal Jaita, L. Magri, Verónica Zaldivar, Sandra Zárate, Daniela Radl, Daniel Pisera
In this review, we analyze the action of estrogens leading to the remodeling of the anterior pituitary gland, especially during the estrous cycle. Proliferation and death of anterior pituitary cells a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::5fe5a1e3b0af0d25f377974338cef9b2
https://doi.org/10.1159/000318491
https://doi.org/10.1159/000318491
Autor:
Sandra Zárate, Verónica Zaldivar, Gabriela Jaita, Guadalupe Eijo, Daniela Radl, Adriana Seilicovich, Jimena Ferraris, Daniel Pisera
Publikováno v:
American journal of physiology. Endocrinology and metabolism. 296(4)
It is now accepted that estrogens not only stimulate lactotrope proliferation but also sensitize anterior pituitary cells to proapoptotic stimuli. In addition to their classical mechanism of action through binding to intracellular estrogen receptors
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d5737ce07d99b890a06f3dbcbe83960c
https://pubmed.ncbi.nlm.nih.gov/19158323
https://pubmed.ncbi.nlm.nih.gov/19158323