Zobrazeno 1 - 10 of 13 pro vyhledávání: '"Ventricular Remodeling/drug effects"'
1
2-Arachidonoylglycerol mobilizes myeloid cells and worsens heart function after acute myocardial infarction
Autor: Christian Weber, Sébastien Lenglet, Aurélien Thomas, Michael Horckmans, Estelle Lauer, Sabine Steffens, Daniel Hering, Raquel Guillamat-Prats, Maximilian J. Schloss
Publikováno v: Cardiovascular research, vol. 115, no. 3, pp. 602-613
Cardiovascular Research
Cardiovascular Research, Vol. 115, No 3 (2019) pp. 602-613
Cardiovascular Research, 115(3), 602-613. Oxford University Press
Aims Myocardial infarction (MI) leads to an enhanced release of endocannabinoids and a massive accumulation of neutrophils and monocytes within the ischaemic myocardium. These myeloid cells originate from haematopoietic precursors in the bone marrow
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ad380123a8c898bbdb72fb5e6061b4dd
https://pubmed.ncbi.nlm.nih.gov/30629130
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2
Cyclosporine before PCI in Acute Myocardial Infarction
Autor: Mewton, Nathan, Bergerot, Cyrille, Ovize, Michel, Bernardi, Paolo, DI LISA, Fabio
Publikováno v: New England Journal of Medicine
New England Journal of Medicine, Massachusetts Medical Society, 2016, 374 (1), pp.90. ⟨10.1056/NEJMc1514192⟩
International audience
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d57c599c5df6d116c74f4d1a0bf7e8e8
https://pubmed.ncbi.nlm.nih.gov/26735999
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3
Targeted Deletion of the Extracellular Signal-Regulated Protein Kinase 5 Attenuates Hypertrophic Response and Promotes Pressure Overload–Induced Apoptosis in the Heart
Autor: Jun-Ichi Abe, Delvac Oceandy, Sukhpal Prehar, Arthur H. Weston, Min Zi, Xin Wang, Wei Liu, Elizabeth J. Cartwright, Tomomi E E Kimura, Jiawei Jin, Ludwig Neyses
Publikováno v: Circulation Research, 106(5), 961-70. United States (2010).
Rationale: Mitogen-activated protein kinase (MAPK) pathways provide a critical connection between extrinsic and intrinsic signals to cardiac hypertrophy. Extracellular signal-regulated protein kinase (ERK)5, an atypical MAPK is activated in the heart
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5476c4ce0d2bf7f78d0759469703b8d3
https://doi.org/10.1161/circresaha.109.209320
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4
Selective activation of angiotensin AT2 receptors attenuates progression of pulmonary hypertension and inhibits cardiopulmonary fibrosis
Autor: Bruce, E, Shenoy, V, Rathinasabapathy, A, Espejo, A, Horowitz, A, Oswalt, A, Francis, J, Nair, A, Unger, T, Raizada, M K, Steckelings, Ulrike Muscha, Sumners, C, Katovich, M J
Publikováno v: Bruce, E, Shenoy, V, Rathinasabapathy, A, Espejo, A, Horowitz, A, Oswalt, A, Francis, J, Nair, A, Unger, T, Raizada, M K, Steckelings, U M, Sumners, C & Katovich, M J 2015, ' Selective activation of angiotensin AT 2 receptors attenuates progression of pulmonary hypertension and inhibits cardiopulmonary fibrosis ', British Journal of Pharmacology, vol. 172, no. 9, pp. 2219-2231 . https://doi.org/10.1111/bph.13044
Background and Purpose Pulmonary hypertension (PH) is a devastating disease characterized by increased pulmonary arterial pressure, which progressively leads to right-heart failure and death. A dys-regulated renin angiotensin system (RAS) has been im
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid_dedup__::b3e044c48f01f109844d8f467705bbdd
https://europepmc.org/articles/PMC4403089/
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5
Soluble epoxide hydrolase inhibition improves coronary endothelial function and prevents the development of cardiac alterations in obese insulin-resistant mice
Autor: Dominique Guerrot, Isabelle Remy-Jouet, Antoine Ouvrard-Pascaud, Jeremy Bellien, Paul Mulder, Anne Marie Madec, Lionel Nicol, David Coquerel, Emmanuelle Loizon, Roméo Cassel, Najah Harouki, Vincent Richard, Christophe Morisseau, Clothilde Roche, Marie Besnier
Publikováno v: Am J Physiol Heart Circ Physiol
Am J Physiol Heart Circ Physiol, 2015, 308 (9), pp.1020-1029. ⟨10.1152/ajpheart.00465.2014⟩
AJP-Heart and Circulatory Physiology
AJP-Heart and Circulatory Physiology, American Physiological Society, 2015, 308 (9), pp.1020-1029. ⟨10.1152/ajpheart.00465.2014⟩
AJP-Heart and Circulatory Physiology, 2015, 308 (9), pp.1020-1029. ⟨10.1152/ajpheart.00465.2014⟩
AJP-Heart and Circulatory Physiology, American Physiological Society, 2015, 308 (9), pp.H1020-9. ⟨10.1152/ajpheart.00465.2014⟩
International audience; This study addressed the hypothesis that inhibiting the soluble epoxide hydrolase (sEH)-mediated degradation of epoxy-fatty acids, notably epoxyeicosatrienoic acids, has an additional impact against cardiovascular damage in in
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::baa58d9216193c4d5b3ec0efd1c602ce
https://www.hal.inserm.fr/inserm-02296633
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6
Prolonged effects of B-type natriuretic peptide infusion on cardiac remodeling after sustained myocardial injury
Autor: Jie Wang, Geng-Hua Yi, Andrew A. Protter, Mehmet C. Oz, Ed X. Wu, Isaac George, Kai Xu, Brad T. Morrow, Jeffrey W. Holmes, Zhihe Li
B-type natriuretic peptide (BNP) is an established first-line therapy for acute decompensated heart failure (HF), but its efficacy in preventing left ventricular (LV) remodeling after myocardial injury is unknown. The goal of this study was to evalua
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3e34936473574b7eb36a423489070172
http://hdl.handle.net/10722/129216
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7
Randomised comparison of growth hormone versus IGF-1 on early post-myocardial infarction ventricular remodelling in rats
Autor: Vassiliki Malamou-Mitsi, Theofilos M. Kolettis, Andreas Fotopoulos, Maria G. Agelaki, A. Skyrlas, A.C. Mitsi, C. Zachariou, E. Kapatou, Konstantinos E. Hatzistergos
OBJECTIVE: Growth hormone and insulin-like growth factor-1 participate in post-myocardial infarction healing, but their relative importance is unclear. We compared the treatment effects of these agents on left ventricular remodelling. DESIGN: Wistar
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::213d52cfefefb633ea23933eb8e178e8
http://olympias.lib.uoi.gr/jspui/handle/123456789/18810
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9
Early, intracoronary growth hormone administration attenuates ventricular remodeling in a porcine model of myocardial infarction
Autor: Mitsi, A. C., Hatzistergos, K. E., Niokou, D., Pappa, L., Baltogiannis, G. G., Tsalikakis, D. G., Papalois, A., Kyriakides, Z. S., Malamou-Mitsi, V., Kolettis, T. M.
OBJECTIVE: Ventricular remodeling is a common corollary of myocardial infarction. We hypothesized that this process may be attenuated by growth hormone, administered as a single high-dose, selectively in the infarct zone, early postmyocardial infarct
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=od_____10561::450bac181b5ead44f4035d5c6d73bd4f
http://olympias.lib.uoi.gr/jspui/handle/123456789/22277
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10
A new, potent poly(ADP-ribose) polymerase inhibitor improves cardiac and vascular dysfunction associated with advanced aging
Autor: Jon G. Mabley, Pal Pacher, Csaba Szabó, Anita Marton, Anne Vaslin, Sandor Batkai, Rita Benko, Lucas Liaudet, György Haskó, Márk Kollai
Publikováno v: Journal of Pharmacology and Experimental Therapeutics, vol. 311, no. 2, pp. 485-91
University of Brighton
Increased production of reactive oxygen and nitrogen species has recently been implicated in the pathogenesis of cardiac and endothelial dysfunction associated with atherosclerosis, hypertension, and aging. Oxidant-induced cell injury triggers the ac
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4d1271832d4169661255734a42f39c6a
https://serval.unil.ch/notice/serval:BIB_EDCEF6840427
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