Zobrazeno 1 - 10
of 16
pro vyhledávání: '"Venkata Garlapati"'
Autor:
Venkata Garlapati, Michael Molitor, Thomas Michna, Gregory S. Harms, Stefanie Finger, Rebecca Jung, Jeremy Lagrange, Panagiotis Efentakis, Johannes Wild, Maike Knorr, Susanne Karbach, Sabine Wild, Ksenija Vujacic-Mirski, Thomas Münzel, Andreas Daiber, Moritz Brandt, Tommaso Gori, Hendrik Milting, Stefan Tenzer, Wolfram Ruf, Philip Wenzel
Publikováno v:
The Journal of Clinical Investigation, Vol 133, Iss 4 (2023)
Despite major advances in acute interventions for myocardial infarction (MI), adverse cardiac remodeling and excess fibrosis after MI causing ischemic heart failure (IHF) remain a leading cause of death worldwide. Here we identify a profibrotic coagu
Externí odkaz:
https://doaj.org/article/7166825a634c4b23b74216fd69860df6
Autor:
Jeremy Lagrange, Stefanie Finger, Sabine Kossmann, Venkata Garlapati, Wolfram Ruf, Philip Wenzel
Publikováno v:
Artery Research, Vol 26, Iss Supplement 1 (2020)
Background/Objectives: Myeloid cells are crucial for the development of vascular inflammation. Low-density lipoprotein receptor-related protein 8 (LRP8) or Apolipoprotein E receptor 2 (ApoER2), is expressed by macrophages, endothelial cells and plate
Externí odkaz:
https://doaj.org/article/f67da4c233f04b0fbd104f70338f010e
Autor:
Wolf-Stephan Rudi, Michael Molitor, Venkata Garlapati, Stefanie Finger, Johannes Wild, Thomas Münzel, Susanne H. Karbach, Philip Wenzel
Publikováno v:
Antioxidants, Vol 10, Iss 3, p 396 (2021)
Aims: Angiotensin-converting-enzyme inhibitors (ACE inhibitors) are a cornerstone of drug therapy after myocardial infarction (MI) and improve left ventricular function and survival. We aimed to elucidate the impact of early treatment with the ACE in
Externí odkaz:
https://doaj.org/article/be4a671943a14cb6aad076c20cc2e89a
Autor:
Moritz Brandt, Hendrik Dörschmann, Sana’a Khraisat, Tanja Knopp, Julia Ringen, Sanela Kalinovic, Venkata Garlapati, Svenja Siemer, Michael Molitor, Sebastian Göbel, Roland Stauber, Susanne Helena Karbach, Thomas Münzel, Andreas Daiber, Philip Wenzel
Publikováno v:
Hypertension (Dallas, Tex. : 1979). 79(10)
Background: Heart failure (HF) coincides with cardiomyocyte telomere shortening. Arterial hypertension is the most prominent risk factor for HF. Both HF and arterial hypertension are associated with dysregulation of the neurohormonal axis. How neuroh
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4776272e0078a17c4c359ea863d30cdb
https://pubmed.ncbi.nlm.nih.gov/36074986
https://pubmed.ncbi.nlm.nih.gov/36074986
Autor:
Stefanie Finger, Philip Wenzel, Tanja Knopp, Johannes Wild, Rebecca Schüler, Venkata Garlapati, Michael Molitor, Susanne Karbach, Wolf-Stephan Rudi, Wolfram Ruf, Maike Knorr, Thomas Münzel, Thanh Son Nguyen, Jeremy Lagrange, Sabine Kossmann
Publikováno v:
Cardiovascular Research. 117:162-177
Aims Heart failure (HF) ensuing myocardial infarction (MI) is characterized by the initiation of a systemic inflammatory response. We aimed to elucidate the impact of myelomonocytic cells and their activation by angiotensin II on vascular endothelial
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d3f4071a11ed7834b1f2a50816f8e035
https://doi.org/10.1093/cvr/cvaa042
https://doi.org/10.1093/cvr/cvaa042
Autor:
Venkata Garlapati, Joanna Wegner, Jens Titze, Lajos Markó, Dimitra Benaki, Tanja Knopp, Philip Wenzel, Johannes Wild, Emmanuel Mikros, Panagiotis Efentakis, Ari Waisman, Natalia Rakova, Thomas Münzel, Susanne Karbach, Friedrich C. Luft, Adriana Marton, Manfred Rauh, Sabine Kossmann, Kento Kitada, Alexandra Grill, Rebecca Jung, Daisuke Nakano, Michael Molitor
Aim Recent evidence suggests that arterial hypertension could be alternatively explained as a physiological adaptation response to water shortage, termed aestivation, which relies on complex multi-organ metabolic adjustments to prevent dehydration. H
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::da018d6c8e256427c316fc649fe064ca
http://edoc.mdc-berlin.de/20011/1/20011oa.pdf
http://edoc.mdc-berlin.de/20011/1/20011oa.pdf
Autor:
Jeremy Lagrange, Philip Wenzel, Michael Molitor, Wolfram Ruf, T Gori, Hendrik Milting, Johannes Wild, Sabine Kossmann, Ksenija Vujacic-Mirski, Thomas Muenzel, Venkata Garlapati, Susanne Karbach, Panagiotis Efentakis, Moritz Brandt, Andreas Daiber
Publikováno v:
European Heart Journal. 41
Background In the setting of myocardial infarction (MI), patients with coronary no-reflow and/or delayed presentation after onset of symptoms (sub-acute MI) are inflicted by severe thrombo-inflammation and are marked by worse clinical outcome. Howeve
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::4b4f63b6cb64acacfd6fe99bc79ba21e
https://doi.org/10.1093/ehjci/ehaa946.3635
https://doi.org/10.1093/ehjci/ehaa946.3635
Autor:
Johannes Wild, Thomas Muenzel, Tanja Knopp, Susanne Karbach, Wolfram Ruf, Stefanie Finger, Venkata Garlapati, Wolf-Stephan Rudi, Michael Molitor, Philip Wenzel
Publikováno v:
European Heart Journal. 41
Background Ischemic heart failure (HF) ensuing myocardial infarction (MI) leads to impaired left ventricular function, reduced cardiac output and counterregulatory activation of angiotensin II (AngII) levels. Furthermore, it is characterized by the i
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::42dfede26465ea7d55d3f952f4d6edf5
https://doi.org/10.1093/ehjci/ehaa946.3751
https://doi.org/10.1093/ehjci/ehaa946.3751
Autor:
Jeremy Lagrange, Stefanie Finger, Sabine Kossmann, Venkata Garlapati, Wolfram Ruf, Philip Wenzel
Publikováno v:
International Journal of Molecular Sciences
International Journal of Molecular Sciences, 2020, 21 (14), pp.4916. ⟨10.3390/ijms21144916⟩
Volume 21
Issue 14
International Journal of Molecular Sciences, Vol 21, Iss 4916, p 4916 (2020)
International Journal of Molecular Sciences, 2020, 21 (14), pp.4916. ⟨10.3390/ijms21144916⟩
Volume 21
Issue 14
International Journal of Molecular Sciences, Vol 21, Iss 4916, p 4916 (2020)
Myeloid cells are crucial for the development of vascular inflammation. Low-density lipoprotein receptor-related protein 8 (LRP8) or Apolipoprotein E receptor 2 (ApoER2), is expressed by macrophages, endothelial cells and platelets and has been impli
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fb2b2f67e925c3d63afaa1184d0dec7f
https://pubmed.ncbi.nlm.nih.gov/32664652
https://pubmed.ncbi.nlm.nih.gov/32664652
Autor:
Wolfram Ruf, Venkata Garlapati, Jeremy Lagrange, Stefanie Finger, Sabine Kossmann, Philip Wenzel
Publikováno v:
Artery Research, Vol 26, Iss Supplement 1 (2020)
Background/Objectives: Myeloid cells are crucial for the development of vascular inflammation. Low-density lipoprotein receptor-related protein 8 (LRP8) or Apolipoprotein E receptor 2 (ApoER2), is expressed by macrophages, endothelial cells and plate
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::abb99931d0da39e3a906155bc140c634
https://doi.org/10.2991/artres.k.201209.043
https://doi.org/10.2991/artres.k.201209.043