Zobrazeno 1 - 10
of 12
pro vyhledávání: '"Vedrana Milinkovic"'
Autor:
Vedrana Milinkovic, Jasna Bankovic, Miodrag Rakic, Tijana Stankovic, Milica Skender-Gazibara, Sabera Ruzdijic, Nikola Tanic
Publikováno v:
PLoS ONE, Vol 8, Iss 12, p e82108 (2013)
Glioblastoma is the most frequent and malignant human brain tumor. High level of genomic instability detected in glioma cells implies that numerous genetic alterations accumulate during glioma pathogenesis. We investigated alterations in AP-PCR DNA p
Externí odkaz:
https://doaj.org/article/df98bcafab9540dd9bc2ce3df71194b3
Autor:
Saša N. Malkov, Milana Trifunovic, Biljana Filipović, Vedrana Milinkovic, Ana Simonović, Angelina Subotić, Slađana Jevremović
Publikováno v:
Plant Cell Tissue and Organ Culture
Arabinogalactan proteins (AGPs) are cell wall hydroxyproline-rich glycoproteins with diverse functions in plant growth and development. In this work the role of AGPs was comparatively studied in four developmental pathways that can be induced from ce
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::182f3104ede0f1162148d8166fd9918e
https://doi.org/10.1007/s11240-015-0741-3
https://doi.org/10.1007/s11240-015-0741-3
Autor:
Tijana Stanković, Ljiljana Denic-Markovic, Jasna Bankovic, Nikola Tanic, Zorica Milosevic, Miodrag Dragoj, Zorka Milovanovic, Milica Pešić, Jelena Stojsic, Sonja Stojkovic Buric, Vedrana Milinkovic, Vesna Skodric-Trifunovic
Publikováno v:
Journal of Molecular and Clinical Medicine. 2:129
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::d2a9c268535b43f7af9440981aeedb97
https://doi.org/10.31083/j.jmcm.2019.04.3241
https://doi.org/10.31083/j.jmcm.2019.04.3241
Autor:
Tijana Stanković, Milica Nedeljković, Nasta Tanić, Tatjana Dramićanin, Zorka Milovanovic, Verica Milošević, Nikola Tanic, Branka Šošić-Jurjević, Radan Džodić, Snežana Šušnjar, Vedrana Milinkovic
Publikováno v:
Journal of Medical Biochemistry. 32:1-8
Summary Background: Breast cancer is the most common form of cancer in women. It arises from multiple genetic changes in oncogenes and tumor suppressor genes. Among so far studied oncogenes relatively few, including epdermal growth factor receptor (E
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::b2ebb847282fc36ef9ce4767e7254eb1
https://doi.org/10.2478/jomb-2014-0005
https://doi.org/10.2478/jomb-2014-0005
Autor:
Vedrana Milinkovic, Sabera Ruzdijic, Nikola Tanic, Tijana Andjelkovic, Ana Podolski-Renić, Jasna Bankovic, Jelena Stojsic
Publikováno v:
Translational Research. 157:19-28
The inactivation of p53 and PTEN tumor suppressor genes is a common genetic event in lung cancer. However, data on the effect of the joint inactivation of tumor-suppressor genes in non-small cell lung carcinoma (NSCLC) are lacking. The purpose of thi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bda05a59b96cae326d5fbe4ca9981f5b
https://doi.org/10.1016/j.trsl.2010.09.004
https://doi.org/10.1016/j.trsl.2010.09.004
Autor:
Tijana Andjelkovic, Vedrana Milinkovic, Nikola Tanic, Jasna Bankovic, Jelena Stojsic, Sabera Ruzdijic, Dragana Jovanovic
Publikováno v:
Lung Cancer. 67:151-159
Lung cancer is the most common cause of neoplasia-related death worldwide. One of the crucial early events in carcinogenesis is the induction of genomic instability and mutator phenotype. We investigated genomic instability in 30 patients with non-sm
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b1e77a226728800c868252f25313800b
https://doi.org/10.1016/j.lungcan.2009.04.010
https://doi.org/10.1016/j.lungcan.2009.04.010
Autor:
Vedrana Milinkovic, Tijana Stanković, Zorica Milosevic, Jelena Dinić, Jelena Stojsic, Zorka Milovanovic, Sonja Stojković, Nikola Tanic, Jasna Bankovic
Publikováno v:
Experimental and Molecular Pathology
Lung cancer is the most common cause of neoplasia-related death worldwide. Accounting for approximately 80\% of all lung carcinomas, the non-small cell lung carcinoma (NSCLC) is the most common clinical form with its two predominant histological type
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::95096a50bf8b0ce20f67867d673bba1c
https://pubmed.ncbi.nlm.nih.gov/25449334
https://pubmed.ncbi.nlm.nih.gov/25449334
Autor:
Tatjana Gazibara, Emilija Manojlovic Gacic, Milica Skender Gazibara, Vedrana Milinkovic, Nikola Tanic
Publikováno v:
Experimental and molecular pathology. 97(2)
Cerebellar glioblastoma (cGBM) is a rare, inadequately characterized disease, without detailed information on its molecular basis. This is the first report analyzing both TP53 and RAS alterations in cGBM. TP53 mutations were detected in more than hal
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::010df180a587a233ae175164a99743d6
https://pubmed.ncbi.nlm.nih.gov/25036404
https://pubmed.ncbi.nlm.nih.gov/25036404
Autor:
Zorica Milosevic, Jasna Bankovic, Jelena Stojsic, Milica Pešić, Sabera Ruzdijic, Nikola Tanic, Tijana Andjelkovic, Vedrana Milinkovic
Publikováno v:
Cancer biomarkers : section A of Disease markers. 10(6)
p16 and PTEN are tumor suppressors that are commonly inactivated in human cancers. Loss of each of these molecules is widely studied in lung cancer, including non-small cell lung carcinoma (NSCLC), its most common clinical form. However, the importan
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d48754a0280b820f9975562117164b43
https://pubmed.ncbi.nlm.nih.gov/22820083
https://pubmed.ncbi.nlm.nih.gov/22820083
Autor:
Maria Chiourea, Jasna Bankovic, Vedrana Milinkovic, Tijana Andjelkovic, Sarantis Gagos, Milica Pešić, Sabera Ruzdijic, Nikola Tanic, Ana Podolski-Renić
Publikováno v:
Clinical Cancer Research. 16:B42-B42
Multidrug resistance (MDR) is the major cause of failure to a successful cancer treatment. Cancer cells manifesting MDR phenotype are cross-resistant to many structurally and functionally unrelated drugs. The purpose of this study was to establish in
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::cfc12d5f8651bee461117b78e9369e43
https://doi.org/10.1158/1078-0432.tcmusa10-b42
https://doi.org/10.1158/1078-0432.tcmusa10-b42