Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Vasiliki Vana"'
Autor:
Camilla Lund, Pablo Ranea-Robles, Sarah Falk, Dylan M. Rausch, Grethe Skovbjerg, Victoria Kamma Vibe-Petersen, Nathalie Krauth, Jacob Lercke Skytte, Vasiliki Vana, Urmas Roostalu, Tune H. Pers, Jens Lund, Christoffer Clemmensen
Publikováno v:
Nature Communications, Vol 15, Iss 1, Pp 1-13 (2024)
Abstract Overfeeding triggers homeostatic compensatory mechanisms that counteract weight gain. Here, we show that both lean and diet-induced obese (DIO) male mice exhibit a potent and prolonged inhibition of voluntary food intake following overfeedin
Externí odkaz:
https://doaj.org/article/149adc83ffa04fb0a50fbe581eab9297
Autor:
Myrtha Arnold, Harald S. Hansen, Jens F. Rehfeld, Michelle K. Lærke, Thue W. Schwartz, Oksana Dmytriyeva, Wolfgang Langhans, Vasiliki Vana
Publikováno v:
Vana, V, Lærke, M K, Rehfeld, J F, Arnold, M, Dmytriyeva, O, Langhans, W, Schwartz, T W & Hansen, H S 2022, ' Vagal afferent cholecystokinin receptor activation is required for glucagon-like peptide-1–induced satiation ', Diabetes, Obesity and Metabolism, vol. 24, no. 2, pp. 268-280 . https://doi.org/10.1111/dom.14575
Peripheral glucagon-like peptide-1 (GLP-1) and cholecystokinin (CCK) are secreted from enteroendocrine cells, and their plasma concentrations increase in response to eating. While the satiating effect of gut-derived CCK on food intake control is well
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6f7632de8f0f6fc53a94a4341c3b0785
https://doi.org/10.1111/dom.14575
https://doi.org/10.1111/dom.14575
Autor:
Jens F. Rehfeld, Vasiliki Vana, Myrtha Arnold, Thue W. Schwartz, Harald S. Hansen, Wolfgang Langhans, Michelle K. Lærke, Oksana Dmytriyeva
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::da7985af0b947d9513d31180adc39744
https://doi.org/10.1111/dom.14575/v2/response1
https://doi.org/10.1111/dom.14575/v2/response1
Autor:
Jeppe H. Ekberg, Birgit L. Lindberg, Thue W. Schwartz, Karen Kleberg, Vasiliki Vana, Piotr A. Mroz, Michelle K. Lærke, Harald S. Hansen, Jens F. Rehfeld
Publikováno v:
Physiologybehavior. 234
Triacylglycerol is the most abundant dietary lipid, and a strong stimulator of satiation. Absorption of triacylglycerol in the small intestine occurs in the form of free fatty acids and 2-monoacylglycerol, a process known to trigger not only the rele
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0644860fd5fb33009290dab41129620e
https://pubmed.ncbi.nlm.nih.gov/33460676
https://pubmed.ncbi.nlm.nih.gov/33460676
Autor:
Harald S. Hansen, Vasiliki Vana
Publikováno v:
British Journal of Pharmacology. 176:1443-1454
This review focuses on recent findings of the physiological and pharmacological role of non-endocannabinoid N-acylethanolamines (NAEs) and 2-monoacylglycerols (2-MAGs) in the intestine and their involvement in the gut-brain signalling. Dietary fat su
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::64998c9fe786f5d6c6691b1e8de7e045
https://doi.org/10.1111/bph.14175
https://doi.org/10.1111/bph.14175
Publikováno v:
European Journal of Hospital Pharmacy. 21:8-12
The use of fixed-dose combinations (FDCs) in the daily practice of pharmacotherapy is increasing after years in the shadows. The main reasons for this renewed popularity are the increasing number of drugs prescribed per patient and the complexity of
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::2e58416eb15e47d25054c1c19a85c9a4
https://doi.org/10.1136/ejhpharm-2013-000390
https://doi.org/10.1136/ejhpharm-2013-000390
Publikováno v:
Journal of pharmaceutical sciences. 105(9)
An important excipient used to overcome poor solubility is cyclodextrin. However, data in the literature suggest that excessive overdosing of cyclodextrins can decrease the absorption of compounds administered with cyclodextrins, due to their lack of
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2c97dc56dcd41927e26dd1961f7ddc1b
https://pubmed.ncbi.nlm.nih.gov/26886315
https://pubmed.ncbi.nlm.nih.gov/26886315