Zobrazeno 1 - 10
of 57
pro vyhledávání: '"Valerie Urbach"'
Autor:
Mickael Shum, Charlie M. London, Maelle Briottet, Khadeeja Adam Sy, Vincent Baillif, Reginald Philippe, Abdolhossein Zare, Sadegh Ghorbani-Dalini, Natacha Remus, Agathe Tarze, Virginie Escabasse, Ralph Epaud, Marc Dubourdeau, Valerie Urbach
Publikováno v:
Frontiers in Immunology, Vol 13 (2022)
Specialized pro-resolving lipid mediators (SPMs) as lipoxins (LX), resolvins (Rv), protectins (PD) and maresins (MaR) promote the resolution of inflammation. We and others previously reported reduced levels of LXA4 in bronchoalveolar lavages from cys
Externí odkaz:
https://doaj.org/article/ce8e81c55de74fc78b83a24bf45eb052
Publikováno v:
Frontiers in Pharmacology, Vol 11 (2020)
Cystic Fibrosis (CF) is a recessive genetic disease due to mutations of the Cystic Fibrosis Transmembrane Conductance Regulator (CFTR) gene encoding the CFTR chloride channel. The ion transport abnormalities related to CFTR mutation generate a dehydr
Externí odkaz:
https://doaj.org/article/257a5614095b451b9fc285f78e672ce5
Autor:
Iwona M. Pranke, Aurélie Hatton, Juliette Simonin, Jean Philippe Jais, Françoise Le Pimpec-Barthes, Ania Carsin, Pierre Bonnette, Michael Fayon, Nathalie Stremler-Le Bel, Dominique Grenet, Matthieu Thumerel, Julie Mazenq, Valerie Urbach, Myriam Mesbahi, Emanuelle Girodon-Boulandet, Alexandre Hinzpeter, Aleksander Edelman, Isabelle Sermet-Gaudelus
Publikováno v:
Scientific Reports, Vol 7, Iss 1, Pp 1-11 (2017)
Abstract Clinical studies with modulators of the Cystic Fibrosis Transmembrane conductance Regulator (CFTR) protein have demonstrated that functional restoration of the mutated CFTR can lead to substantial clinical benefit. However, studies have show
Externí odkaz:
https://doaj.org/article/71753db8957441c5881e8e0b86b29dbf
Autor:
Réginald Philippe, Valerie Urbach
Publikováno v:
International Journal of Molecular Sciences, Vol 19, Iss 10, p 2865 (2018)
In cystic fibrosis (CF), impaired airway surface hydration (ASL) and mucociliary clearance that promote chronic bacterial colonization, persistent inflammation, and progressive structural damage to the airway wall architecture are typically explained
Externí odkaz:
https://doaj.org/article/268fa98c31d644bba9c944e23ac8bc11
Autor:
Gerard Higgins, Isabelle Sermet-Gaudelus, Paul McNally, Monika Hollenhorst, Ahmad Moukachar, Aurélie Hatton, Coral Fustero-Torre, Valerie Urbach, Fiona C. Ringholz, Brian J. Harvey, Ali Sassi
Publikováno v:
Journal of Cystic Fibrosis
Journal of Cystic Fibrosis, Elsevier, 2018, 17 (5), pp.607-615. ⟨10.1016/j.jcf.2017.11.017⟩
Journal of Cystic Fibrosis, Elsevier, 2018, 17 (5), pp.607-615. ⟨10.1016/j.jcf.2017.11.017⟩
Background Cystic Fibrosis (CF) lung disease is characterised by dysregulated ion transport that promotes chronic bacterial infection and inflammation. The impact of the specialised pro-resolution mediator resolvin D1 (RvD1) on airway surface liquid
Autor:
Thomas Flament, A. Masson, Aurélie Hatton, Aleksander Edelman, F. Chedevergne, Valerie Urbach, Muriel Le Bourgeois, Alexandre Hinzpeter, C. Bailly, Iwona Pranke, Isabelle Sermet-Gaudelus
Publikováno v:
American Journal of Respiratory and Critical Care Medicine
American Journal of Respiratory and Critical Care Medicine, American Thoracic Society, 2019, 199 (1), pp.123-126. ⟨10.1164/rccm.201808-1436LE⟩
American Journal of Respiratory and Critical Care Medicine, American Thoracic Society, 2019, 199 (1), pp.123-126. ⟨10.1164/rccm.201808-1436LE⟩
International audience
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7129003ca6e4de8b8a624ceef5e13b11
https://hal.archives-ouvertes.fr/hal-03110513
https://hal.archives-ouvertes.fr/hal-03110513
Autor:
Gerard Higgins, Valerie Urbach, Coral Fustero Torre, Jean Tyrrell, Paul McNally, Brian J. Harvey
Publikováno v:
American Journal of Physiology-Lung Cellular and Molecular Physiology
American Journal of Physiology-Lung Cellular and Molecular Physiology, American Physiological Society, 2016, 310 (11), pp.L1053-L1061. ⟨10.1152/ajplung.00368.2015⟩
American Journal of Physiology-Lung Cellular and Molecular Physiology, American Physiological Society, 2016, 310 (11), pp.L1053-L1061. ⟨10.1152/ajplung.00368.2015⟩
The specialized proresolution lipid mediator lipoxin A4(LXA4) is abnormally produced in cystic fibrosis (CF) airways. LXA4increases the CF airway surface liquid height and stimulates airway epithelial repair and tight junction formation. We report he
Autor:
Fiona O'Mahony, Rodrigo Alzamora, Francisco V. Sepúlveda, Raphael Rapetti-Mauss, Brian J. Harvey, Valerie Urbach, Viviana Bustos, L. Pablo Cid
Publikováno v:
The Journal of Physiology. 589:5091-5107
The KCNQ1 potassium channel associates with various KCNE ancillary subunits that drastically affect channel gating and pharmacology. Co-assembly with KCNE3 produces a current with nearly instantaneous activation, some time-dependent activation at ver
Publikováno v:
BioMed Research International, Vol 2015 (2015)
BioMed Research International
BioMed Research International, Hindawi Publishing Corporation, 2015, 2015, pp.1-10. ⟨10.1155/2015/781087⟩
BioMed Research International
BioMed Research International, Hindawi Publishing Corporation, 2015, 2015, pp.1-10. ⟨10.1155/2015/781087⟩
Lipoxin A4has been described as a major signal for the resolution of inflammation and is abnormally produced in the lungs of patients with cystic fibrosis (CF). In CF, the loss of chloride transport caused by the mutation in the cystic fibrosis trans
Autor:
Sara Bitam, Danielle Tondelier, M. I Hollenhorst, Valerie Urbach, Iwona Pranke, Aleksander Edelman, Nathalie Servel, Alexandre Hinzpeter, Aurélie Hatton, Isabelle Sermet-Gaudelus, Christelle Moquereau
Publikováno v:
F1000Research
F1000Research, Faculty of 1000, 2015, 4, pp.218. ⟨10.12688/f1000research.6683.2⟩
F1000Research, Faculty of 1000, 2015, 4, pp.218. ⟨10.12688/f1000research.6683.2⟩
Cystic fibrosis (CF) is a multifactorial disease caused by mutations in the cystic fibrosis transmembrane conductance regulator gene (CFTR),which encodes a cAMP-dependent Cl-channel. The most frequent mutation, F508del, leads to the synthesis of a pr
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d36a54aff446e3078ab87ec0749d5a94
https://hal.archives-ouvertes.fr/hal-03110527
https://hal.archives-ouvertes.fr/hal-03110527