Zobrazeno 1 - 10
of 640
pro vyhledávání: '"VASCULAR POROSITY"'
Akademický článek
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Autor:
Javaheri, B., Carriero, A., Staines, K.A., Chang, Y.-M., Houston, D.A., Oldknow, K.J., Millan, J.L., Kazeruni, Bassir N., Salmon, P., Shefelbine, S., Farquharson, C., Pitsillides, A.A.
Publikováno v:
In Bone December 2015 81:277-291
Autor:
Henry Mosey, Juan A. Núñez, Alice Goring, Claire E. Clarkin, Katherine A. Staines, Peter D. Lee, Andrew A. Pitsillides, Behzad Javaheri
Publikováno v:
Frontiers in Materials, Vol 4 (2017)
SCLEROSTIN (Sost) is expressed predominantly in osteocytes acting as a negative regulator of bone formation. In humans, mutations in the SOST gene lead to skeletal overgrowth and increased bone mineral density, suggesting that SCLEROSTIN is a key reg
Externí odkaz:
https://doaj.org/article/4c909af927814f928820d6d81255a2b3
Publikováno v:
In Bone September 2014 66:121-130
Autor:
Carriero, A., Doube, M., Vogt, M., Busse, B., Zustin, J., Levchuk, A., Schneider, P., Müller, R., Shefelbine, S.J.
Publikováno v:
In Bone April 2014 61:116-124
Akademický článek
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Autor:
Goulet, Grant C., Hamilton, Nicolas, Cooper, David, Coombe, Dennis, Tran, David, Martinuzzi, Robert, Zernicke, Ronald F.
Publikováno v:
In Journal of Biomechanics 2008 41(10):2169-2175
Autor:
Karla J. Oldknow, Andrew A. Pitsillides, Behzad Javaheri, Bassir N. Kazeruni, Sandra J. Shefelbine, Katherine Staines, Dean Houston, Phil Salmon, Alessandra Carriero, Colin Farquharson, Yu-Mei Chang, José Luis Millán
Publikováno v:
Bone
BONE
Javaheri, B, Carriero, A, Staines, K A, Chang, Y-M, Houston, D A, Suchacki, K, Millan, J L, Kazeruni, B N, Salmon, P, Shefelbine, S, Farquharson, C & Pitsillides, A A 2015, ' Phospho1 deficiency transiently modifies bone architecture yet produces consistent modification in osteocyte differentiation and vascular porosity with ageing ', Bone, vol. 81, pp. 277-291 . https://doi.org/10.1016/j.bone.2015.07.035
BONE
Javaheri, B, Carriero, A, Staines, K A, Chang, Y-M, Houston, D A, Suchacki, K, Millan, J L, Kazeruni, B N, Salmon, P, Shefelbine, S, Farquharson, C & Pitsillides, A A 2015, ' Phospho1 deficiency transiently modifies bone architecture yet produces consistent modification in osteocyte differentiation and vascular porosity with ageing ', Bone, vol. 81, pp. 277-291 . https://doi.org/10.1016/j.bone.2015.07.035
PHOSPHO1 is one of principal proteins involved in initiating bone matrix mineralisation. Recent studies have found that Phospho1 KO mice (Phospho1-R74X) display multiple skeletal abnormalities with spontaneous fractures, bowed long bones, osteomalaci
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3593eaccc3e6a21e2520aaeb1dcb939b
https://doi.org/10.1016/j.bone.2015.07.035
https://doi.org/10.1016/j.bone.2015.07.035
Autor:
Sandra Sefa, Jonathan Espiritu, Hanna Ćwieka, Imke Greving, Silja Flenner, Olga Will, Susanne Beuer, D.C Florian Wieland, Regine Willumeit-Römer, Berit Zeller-Plumhoff
Publikováno v:
Bioactive Materials, Vol 30, Iss , Pp 154-168 (2023)
The utilization of biodegradable magnesium (Mg)-based implants for restoration of bone function following trauma represents a transformative approach in orthopaedic application. One such alloy, magnesium-10 weight percent gadolinium (Mg-10Gd), has be
Externí odkaz:
https://doaj.org/article/6dbcf960371c4a00883d3f4255cb5e0d
Autor:
Stephanie Gohin, Timothy R. Arnett, Andrew A. Pitsillides, Chantal Chenu, Mark Hopkinson, Behzad Javaheri
Publikováno v:
Journal of applied physiology (Bethesda, Md. : 1985). 128(4)
Blood supply is essential for osteogenesis, yet its relationship to load-related increases in bone mass is poorly defined. Herein, we aim to investigate the link between load-induced osteogenesis and the blood supply (bone perfusion and vascular poro
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a35180eee20aa2a3b8f40bab713150b2
https://pubmed.ncbi.nlm.nih.gov/32163331
https://pubmed.ncbi.nlm.nih.gov/32163331