Zobrazeno 1 - 10
of 61
pro vyhledávání: '"V. V. Bulygina"'
Autor:
T. M. Chernova, V. N. Timchenko, E. V. Barakina, A. A. Zherebtsova, N. S. Gusarova, Yu. S. Khabarova, V. V. Bulygina, D. E. Shakhrai, A. V. Zhiglova, E. A. Ttsvetkova
Publikováno v:
Журнал инфектологии, Vol 14, Iss 2, Pp 96-106 (2022)
At present, there are isolated data on the clinical outcomes of COVID-19 in children, especially those who have experienced asymptomatic and mild forms of infection.Purpose: to analyze the nature and frequency of postCOVID symptoms in children during
Externí odkaz:
https://doaj.org/article/d068224a49ae45cc84e2903b09e00b7f
Autor:
D. A. Lanshakov, E. V. Sukhareva, V. V. Bulygina, A. V. Bannova, E. V. Shaburova, T. S. Kalinina
Publikováno v:
Scientific Reports, Vol 11, Iss 1, Pp 1-13 (2021)
Abstract Elevated glucocorticoid level in the early postnatal period is associated with glucocorticoid therapy prescribed at preterm delivery most often has severe long-lasting neurodevelopmental and behavioural effects. Detailed molecular mechanisms
Externí odkaz:
https://doaj.org/article/a280503cb5e74d659f110148f10597e3
Publikováno v:
Вавиловский журнал генетики и селекции, Vol 20, Iss 2, Pp 212-219 (2016)
Early life stress events can produce long-lasting changes in neurochemistry and behaviors related to monoamine systems, with increased risks of cardiovascular, metabolic, neuroendocrine, psychiatric disorders, generalized anxiety and depression in ad
Externí odkaz:
https://doaj.org/article/88807e3ed3ff49d5b064a18b5fd1f957
Autor:
Timofey A. Lagunov, E. V. Sukhareva, Dmitriy A. Lanshakov, Tatyana S. Kalinina, V. V. Bulygina
Publikováno v:
Интегративная физиология, Vol 2, Iss 1 (2021)
Glucocorticoids (GC) are crucial regulators of homeostasis and function. Despite its negative side effects, glucocorticoid therapy in neonates is widely used antenatally for accelerating fetal lung maturation in cases of preterm birth. GC action is m
Publikováno v:
Neurochemical Journal. 13:349-354
—Elevated levels of glucocorticoids in the perinatal period of ontogeny may provoke further development of neuropathology, whose mechanisms can involve apoptosis of brain cells caused by impaired expression of neurotrophins, including the practical
Publikováno v:
Russian Journal of Genetics: Applied Research. 7:226-234
Adverse factors of early development can produce long-lasting alterations of the brain neurochemical systems, the physiological functions and behavior. Tyrosine hydroxylase (TH), the key enzyme of catecholamine biosynthesis, determines the activity o
Publikováno v:
European Neuropsychopharmacology. 40:S55-S56
Autor:
K. Egorova, V. V. Bulygina, Nikolay N. Dygalo, E. V. Sukhareva, Tatyana S. Kalinina, D. A. Lanshakov
Publikováno v:
European Neuropsychopharmacology. 29:S166-S167
Publikováno v:
Neurochemical Journal. 12:152-154
The mechanisms of development of stress-induced depression symptoms may include interaction between the elements of the serotonergic (5-HT) system and brain-derived neurotrophic factor (BDNF) at the level of the cell bodies of 5-HT neurons. In order
Publikováno v:
Russian Journal of Genetics: Applied Research. 5:577-581
Stressful events induce changes in the expression of numerous genes and their protein products in the brain. However, the mechanisms mediating these changes, as well as their significance for the development of stress-induced depression or for coping