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Autor:
P. Blyszczuk, G. Kania, E. Pachera, F. Rolski, A. Hukara, V. Tela, M. Mayo, V. Dixit, B. Yang, J. Gollob, N. Mainolfi, A. Slavin, C. Hubeau, O. Distler
Publikováno v:
Annals of the Rheumatic Diseases. 81:494-495
BackgroundThe ubiquitin-proteasome system (UPS) is the endogenous intracellular mechanism for maintaining protein homeostasis through protein degradation and turnover. Heterobifunctional small molecules are a new class of compounds that form a ternar
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::dcc4a7b7f2f55718eaa786be77a74d57
https://doi.org/10.1136/annrheumdis-2022-eular.2573
https://doi.org/10.1136/annrheumdis-2022-eular.2573
