Výsledky vyhledávání - "V E, Laubach"

Characterization of a human α1-antitrypsin null allele involving aberrant mRNA splicing

Publikováno v: Human Molecular Genetics. 2:1001-1005

alpha 1-Antitrypsin (alpha 1AT) is a major protease inhibitor present in high concentrations in the plasma. Inheritance of alpha 1AT deficiency or null alleles (alleles associated with no detectable serum alpha 1AT) is associated with an increased ri

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ad201c649577dd9d0048578830c526b8
https://doi.org/10.1093/hmg/2.7.1001

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Indomethacin promotes nitric oxide function in the ductus arteriosus in the mouse

Autor: D, Sodini, B, Baragatti, S, Barogi, V E, Laubach, F, Coceani

Publikováno v: British journal of pharmacology. 153(8)

Prenatal patency of ductus arteriosus is maintained by prostaglandin (PG) E(2) in concert with nitric oxide (NO) and carbon monoxide (CO). Accordingly, we have previously found that NO activity increases upon deletion of either COX. Here, we have exa

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::711ccc8db21fe690c780a50ed66a281c
https://pubmed.ncbi.nlm.nih.gov/18297107

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Interactions between NO, CO and an endothelium-derived hyperpolarizing factor (EDHF) in maintaining patency of the ductus arteriosus in the mouse

Autor: B, Baragatti, F, Brizzi, S, Barogi, V E, Laubach, D, Sodini, E G, Shesely, R F, Regan, F, Coceani

Publikováno v: British journal of pharmacology. 151(1)

Prenatal patency of ductus arteriosus is maintained by prostaglandin (PG) E(2), possibly along with nitric oxide (NO) and carbon monoxide (CO), and cyclooxygenase (COX) deletion upregulates NO. Here, we have examined enzyme source and action of NO fo

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::367f2e6f63e142745dc30e1660d749a2
https://pubmed.ncbi.nlm.nih.gov/17351656

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Inducible nitric oxide synthase (iNOS) gene deficiency increases the mortality of sepsis in mice

Autor: J P, Cobb, R S, Hotchkiss, P E, Swanson, K, Chang, Y, Qiu, V E, Laubach, I E, Karl, T G, Buchman

Publikováno v: Surgery. 126(2)

Nitric oxide (NO) produced by the inducible isoform of NO synthase (iNOS or NOS2) has been implicated in the hypotension, organ failure, and death that complicate sepsis. To avoid the confounding effects and limitations of iNOS inhibitors, we used iN

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::0535884707da290612e7524c6dc846e9
https://pubmed.ncbi.nlm.nih.gov/10455918

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