Výsledky vyhledávání - "V A, Shatskaia"

[Role of phosphatidylinositol signalling path in developing hormonal resistance in tumor cells]

Autor: M A, Krasil'nikov, V A, Shatskaia

Publikováno v: Voprosy onkologii. 47(2)

Phosphatidylinositol 3-kinase (PI3K) is a key regulatory protein which is responsible for anti-apoptotic signal transduction regulating cell survival during exposure to damaging factors. The report deals with the role of the PI3K signaling pathway in

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::29ba7240eb3d3819240eb329d610e33d
https://pubmed.ncbi.nlm.nih.gov/11383461

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[Determination of steroid hormone receptors in different tissues and neoplasms]

Autor: Z V, Kuz'mina, V A, Shatskaia, Z S, Smirnova

Publikováno v: Biulleten' eksperimental'noi biologii i meditsiny. 125(5)

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::d21eb7e7f56008e3db154edbaa3ad7e1
https://pubmed.ncbi.nlm.nih.gov/9644559

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[Effect of specific activation of phosphatidylcholine metabolism in hamster fibroblasts transformed by Rous sarcoma virus]

Autor: M A, Krasil'nikov, V A, Shatskaia, O A, Mizenina, A G, Tatosian

Publikováno v: Biokhimiia (Moscow, Russia). 61(3)

Transformation of embryonic hamster fibroblasts by the Rous sarcoma virus results in sharp increase of the turnover rate of one of cellular phospholipids-phosphatidylcholine. The decrease in the rate of virus-transformed cells (HETSR strain) during t

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::4962fd6e5359227be8d6c021e7ceae3a
https://pubmed.ncbi.nlm.nih.gov/8724608

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[Evaluation of the effectiveness of tamoxifen in malignant breast tumors: new approaches]

Autor: M A, Krasil'nikov, V A, Shatskaia, V D, Ermilova, V P, Letiagin

Publikováno v: Vestnik Rossiiskoi akademii meditsinskikh nauk. (4)

It has been shown that 17 beta-estradiol stimulates the cycle of phospholipid conversion in the breast tumor cells. The action of tamoxifen on antiestrogen cells is not limited by weakening the stimulating effect of 17 beta-estradiol on the exchange

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::16024f1670cfe5d7792ce1d4e9e75d2a
https://pubmed.ncbi.nlm.nih.gov/7780335

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[Regulation of the cycle of phospholipid turnover in hamster fibroblasts transformed by v-src and N-ras oncogenes]

Autor: M A, Krasil'nikov, V A, Shatskaia, M S, Shtutman

Publikováno v: Biokhimiia (Moscow, Russia). 59(11)

The phospholipid turnover has been studied in two lines of golden hamster cells: in cells transformed by the Rous sarcoma virus (line HET-SR) and in cells additionally transfected with the activated oncogene N-ras (line HET-SR-N-ras, clone 6). It has

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::604c059d60c075b02c273878c1499f57
https://pubmed.ncbi.nlm.nih.gov/7873683

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[The effect of progesterone and tamoxifen on EGF-dependent activation of phospholipid turnover in uterine and breast tumor cells]

Autor: M A, Krasil'nikov, V A, Shatskaia, L S, Bassalyk

Publikováno v: Biokhimiia (Moscow, Russia). 58(6)

The in vitro effects of the epidermal growth factor (EGF) and progesterone on phospholipid turnover in cells of 19 human adenocarcinomas (postsurgical material) have been studied. In 58% of tumours EGF increased the 32P incorporation into two basic c

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::93923b43f1ee52d3a70eeea302025c6f
https://pubmed.ncbi.nlm.nih.gov/8364119

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[The effect of steroid hormones and tamoxifen on the rate of phospholipid turnover in the cells of uterine and breast tumors]

Autor: M A, Krasil'nikov, V A, Shatskaia, Z V, Kuz'mina, V V, Barinov, V P, Letiagin, L S, Bassalyk

Publikováno v: Vestnik Rossiiskoi akademii meditsinskikh nauk. (3)

The cycle of phospholipid turnover has been found to be under the negative control of hormonal cytostatics (progesterone) and under the positive control of proliferation stimulants (17 beta-estradiol, epidermal growth factor). Specific changes in the

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::f9d99a9d56c6f216e6eaaafdd69911e9
https://pubmed.ncbi.nlm.nih.gov/7687911

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[Regulation of the phospholipid turnover rate and protein kinase C activity as a necessary stage in the realization of the growth-inhibiting effect of dexamethasone on hepatoma 22 cells]

Autor: M A, Krasil'nikov, V M, Bezrukov, V A, Shatskaia

Publikováno v: Biokhimiia (Moscow, Russia). 57(4)

The role of protein kinase C and phospholipid turnover in the realization of the cytostatic effect of dexamethasone on hormone-sensitive cells of mouse hepatoma 22 has been studied. It was found that dexamethasone added to hepatoma cells induces a ra

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::2e01a8165d861a06e39cbd55b2b839c5
https://pubmed.ncbi.nlm.nih.gov/1637923

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[Growth inhibiting effect of glucocorticoids and progestins on tumor cells in vitro: effect on the rate of phospholipid reversal]

Autor: M A, Krasil'nikov, V A, Shatskaia

Publikováno v: Biokhimiia (Moscow, Russia). 56(7)

The growth-inhibiting effect of dexamethasone was estimated by the ability of the hormone to inhibit the proliferative activity of in vitro cultured hepatoma 22 cells. The effect of another steroid proliferation inhibitor, progesterone, was studied i

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::16dffcefd2d4ed532806e5afbe99e460
https://pubmed.ncbi.nlm.nih.gov/1932351

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