Runs of homozygosity in Estonian horse breeds

Autor: Miglior, F., author, Pinotti, L., author, Boyle, L., author, Kenny, D., author, Lee, M., author, De Marchi, M., author, Cadavez, V.A.P., author, Millet, S., author, Evans, R., author, Veldkamp, T., author, Pastell, M., author, Pollott, G., author, Spoolder, H., author, Sild, E., author, Värv, S., author, Kaart, T., author, Viinalass, H., author

Publikováno v: Book of Abstracts of the 73rd Annual Meeting of the European Federation of Animal Science. :381-381

Maternal and paternal genetic variation in Estonian local horse breeds in the context of geographically adjacent and distant Eurasian breeds.

Autor: Sild, E.¹ (AUTHOR) erkki.sild@emu.ee, Värv, S.¹ (AUTHOR), Kaart, T.¹ (AUTHOR), Kantanen, J.² (AUTHOR), Popov, R.³ (AUTHOR), Viinalass, H.¹ (AUTHOR)

Publikováno v: Animal Genetics. Dec2019, Vol. 50 Issue 6, p757-760. 4p.

Composite β-κ-casein genotypes and their effect on composition and coagulation of milk from Estonian Holstein cows

Autor: Vallas, M. ^{*, †}, Kaart, T. ^{*, †}, Värv, S. ^{*, †}, Pärna, K. ^‡, Jõudu, I. ^{*, †}, Viinalass, H. ^{*, †}, Pärna, E. ^{*, †}

Publikováno v: In Journal of Dairy Science November 2012 95(11):6760-6769

Transcriptional regulator Taf14 binds DNA and is required for the function of transcription factor TFIID in the absence of histone H2A.Z.

Autor: Peil K; Institute of Molecular and Cell Biology, University of Tartu, Tartu, Estonia., Värv S; Institute of Molecular and Cell Biology, University of Tartu, Tartu, Estonia., Ilves I; Institute of Technology, University of Tartu, Tartu, Estonia., Kristjuhan K; Institute of Molecular and Cell Biology, University of Tartu, Tartu, Estonia., Jürgens H; Institute of Molecular and Cell Biology, University of Tartu, Tartu, Estonia., Kristjuhan A; Institute of Molecular and Cell Biology, University of Tartu, Tartu, Estonia. Electronic address: arnoldk@ut.ee.

Publikováno v: The Journal of biological chemistry [J Biol Chem] 2022 Sep; Vol. 298 (9), pp. 102369. Date of Electronic Publication: 2022 Aug 13.

Rpb9-deficient cells are defective in DNA damage response and require histone H3 acetylation for survival.

Autor: Sein H; Department of Cell Biology, Institute of Molecular and Cell Biology, University of Tartu, Riia 23, 51010, Tartu, Estonia., Reinmets K; Department of Cell Biology, Institute of Molecular and Cell Biology, University of Tartu, Riia 23, 51010, Tartu, Estonia., Peil K; Department of Cell Biology, Institute of Molecular and Cell Biology, University of Tartu, Riia 23, 51010, Tartu, Estonia., Kristjuhan K; Department of Cell Biology, Institute of Molecular and Cell Biology, University of Tartu, Riia 23, 51010, Tartu, Estonia., Värv S; Department of Cell Biology, Institute of Molecular and Cell Biology, University of Tartu, Riia 23, 51010, Tartu, Estonia.; Department of Biosciences, Section for Biochemistry and Molecular Biology, University of Oslo, Blindernveien 31, 0371, Oslo, Norway., Kristjuhan A; Department of Cell Biology, Institute of Molecular and Cell Biology, University of Tartu, Riia 23, 51010, Tartu, Estonia. arnoldk@ut.ee.

Publikováno v: Scientific reports [Sci Rep] 2018 Feb 13; Vol. 8 (1), pp. 2949. Date of Electronic Publication: 2018 Feb 13.