Zobrazeno 1 - 10
of 20
pro vyhledávání: '"Vânia L. Batalha"'
Autor:
Joana E. Coelho, Tiago F. Outeiro, Mariana Temido-Ferreira, Inga Zerr, Jeong-Seop Rhee, Matthias Schmitz, Éva M. Szegö, Sandra H. Vaz, Diana G. Ferreira, Vânia L. Batalha, Hugo Vicente Miranda, Luísa V. Lopes, Inês Marques-Morgado
Publikováno v:
Nature reviews / Neuroscience 20(11), 1569-1579 (2017). doi:10.1038/nn.4648
Nature Neuroscience
Nature Neuroscience
© 2017 Nature America, Inc., part of Springer Nature. All rights reserved.
Synucleinopathies, such as Parkinson's disease and dementia with Lewy bodies, are neurodegenerative disorders that are characterized by the accumulation of α-synuclein
Synucleinopathies, such as Parkinson's disease and dementia with Lewy bodies, are neurodegenerative disorders that are characterized by the accumulation of α-synuclein
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::100a137a74503d7ee4176a5166446ddb
https://doi.org/10.1038/nn.4648
https://doi.org/10.1038/nn.4648
Autor:
Francisco M. Mouro, Luísa V. Lopes, Ana M. Sebastião, Joana E. Coelho, Younis Baqi, Christa E. Müller, Vânia L. Batalha, Joaquim A. Ribeiro, Diana G. Ferreira
Publikováno v:
Neuropharmacology
© 2017 Elsevier Ltd. All rights reserved.
Cannabinoid-mediated memory impairment is a concern in cannabinoid-based therapies. Caffeine exacerbates cannabinoid CB1 receptor (CB1R)-induced memory deficits through an adenosine A1 receptor-mediated
Cannabinoid-mediated memory impairment is a concern in cannabinoid-based therapies. Caffeine exacerbates cannabinoid CB1 receptor (CB1R)-induced memory deficits through an adenosine A1 receptor-mediated
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::564e89b62aa58349c28b21a1fb2f29e5
https://doi.org/10.1016/j.neuropharm.2017.02.021
https://doi.org/10.1016/j.neuropharm.2017.02.021
Autor:
Andreia Pinto, José Pimentel, Michael Bader, Rodrigo A. Cunha, Luísa V. Lopes, Rui Gomes, Christa E. Müller, Inês Marques-Morgado, Vânia L. Batalha, Joana E. Coelho, Pedro M. Pereira, Laetitia Cuvelier, Tiago F. Outeiro, Diana G. Ferreira, Younis Baqi, Paula A. Pousinha, David Blum, Emilie Faivre, Serge N. Schiffmann, Luc Buée, Hélène Marie, Sara Carvalho, Mariana Temido-Ferreira, Valérie Buée-Scherrer, Paula M. Canas
Publikováno v:
Molecular Psychiatry
Molecular Psychiatry, Nature Publishing Group, 2018, ⟨10.1038/s41380-018-0110-9⟩
Molecular Psychiatry, 2018, ⟨10.1038/s41380-018-0110-9⟩
Molecular psychiatry, 25 (8
Molecular Psychiatry, Nature Publishing Group, 2018, ⟨10.1038/s41380-018-0110-9⟩
Molecular Psychiatry, 2018, ⟨10.1038/s41380-018-0110-9⟩
Molecular psychiatry, 25 (8
© The Author(s) 2018. This article is published with open access. Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any mediu
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e5b10a7b64b9f6f27e03756e908d6ecd
https://hdl.handle.net/10451/55389
https://hdl.handle.net/10451/55389
Autor:
Alexandre de Mendonça, Raquel B. Dias, Ana M. Sebastião, Ruben Vale, Ana Rita Costenla, Diogo M. Rombo, Maria José Diógenes, Vânia L. Batalha, Júlia Pinho
Publikováno v:
Neuropharmacology
© 2016 Published by Elsevier Ltd.
Age-dependent memory deterioration has been well documented and yet an increase in rat hippocampal LTP upon aging has been reported. This poses the question of whether the enhanced LTP is a cause or an attempt
Age-dependent memory deterioration has been well documented and yet an increase in rat hippocampal LTP upon aging has been reported. This poses the question of whether the enhanced LTP is a cause or an attempt
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::599cfe6de9041f3f3745a0aa194851fc
Autor:
Luísa V. Lopes, Ana M. Sebastião, Vânia L. Batalha, Younis Baqi, André Jerónimo-Santos, Christa E. Müller, Maria José Diógenes
Publikováno v:
Neuropharmacology
© 2014 Elsevier Ltd. All rights reserved
Brain-derived neurotrophic factor (BDNF) through the activation of its receptor (TrkB-FL) exert well-described neuroprotective effects playing a major role in hippocampal synaptic transmission and plasti
Brain-derived neurotrophic factor (BDNF) through the activation of its receptor (TrkB-FL) exert well-described neuroprotective effects playing a major role in hippocampal synaptic transmission and plasti
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0d73d16103e1e75e111650d0cfba40b8
https://doi.org/10.1016/j.neuropharm.2014.04.006
https://doi.org/10.1016/j.neuropharm.2014.04.006
Autor:
Diana G, Ferreira, Mariana, Temido-Ferreira, Hugo, Vicente Miranda, Vânia L, Batalha, Joana E, Coelho, Éva M, Szegö, Inês, Marques-Morgado, Sandra H, Vaz, Jeong Seop, Rhee, Matthias, Schmitz, Inga, Zerr, Luísa V, Lopes, Tiago F, Outeiro
Publikováno v:
Nature neuroscience. 20(11)
Synucleinopathies, such as Parkinson's disease and dementia with Lewy bodies, are neurodegenerative disorders that are characterized by the accumulation of α-synuclein (aSyn) in intracellular inclusions known as Lewy bodies. Prefibrillar soluble aSy
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::5e0cdb09fbd03b3cf342bfa86de7c85b
https://pubmed.ncbi.nlm.nih.gov/28945221
https://pubmed.ncbi.nlm.nih.gov/28945221
Autor:
Detlev Boison, Long Liu, Abbie Jones, Yuqing Huo, Shayla Q. Coffman, Bounmy Saleumvong, Catarina Miranda-Lourenço, Mariana Colino-Oliveira, Ana M. Sebastião, Maria José Diógenes, Cátia Palminha, Yiming Xu, Vânia L. Batalha, Ursula S. Sandau
Publikováno v:
The Journal of neuroscience : the official journal of the Society for Neuroscience. 36(48)
Adenosine kinase (ADK) deficiency in human patients (OMIM:614300) disrupts the methionine cycle and triggers hypermethioninemia, hepatic encephalopathy, cognitive impairment, and seizures. To identify whether this neurological phenotype is intrinsica
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::af49d8e067119cfc945cab616ee94a4d
https://pubmed.ncbi.nlm.nih.gov/27903722
https://pubmed.ncbi.nlm.nih.gov/27903722
Autor:
Luísa V. Lopes, Vânia L. Batalha, Ana M. Sebastião, Joaquim A. Ribeiro, Joana Vital, Vasco C. Sousa, Ana Rita Costenla
Publikováno v:
Repositório Científico de Acesso Aberto de Portugal
Repositório Científico de Acesso Aberto de Portugal (RCAAP)
instacron:RCAAP
Neurobiology of Aging
Repositório Científico de Acesso Aberto de Portugal (RCAAP)
instacron:RCAAP
Neurobiology of Aging
© 2014 Elsevier Inc. All rights reserved.
Exposure to chronic stress during the neonatal period is known to induce permanent long-term changes in the central nervous system and hipothalamic-pituitary-adrenal axis reactivity that are associated
Exposure to chronic stress during the neonatal period is known to induce permanent long-term changes in the central nervous system and hipothalamic-pituitary-adrenal axis reactivity that are associated
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f3a420921e9e0f9ef8b5758d274dedef
https://hdl.handle.net/10451/51221
https://hdl.handle.net/10451/51221
Autor:
João Data-Franca, Jorge S. Valadas, Joaquim A. Ribeiro, Frederico Simões do Couto, Luísa V. Lopes, Vânia L. Batalha
Publikováno v:
European Journal of Pharmacology. 695:71-75
Maternal separation (MS) induces depressive-like behavior and long-term changes in cognition in rats. Escitalopram is an antidepressant drug shown to reverse the depressive-like features caused by this stress model. However, it is not known if it can
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::dd104dba65ea28767d95d78eb388d2d3
https://doi.org/10.1016/j.ejphar.2012.08.020
https://doi.org/10.1016/j.ejphar.2012.08.020
Autor:
Luísa V. Lopes, Rui Gomes, Tiago F. Outeiro, Vânia L. Batalha, José Pedro Rino, António Albino-Teixeira, Joana E. Coelho, Hugo Vicente Miranda, Rodrigo A. Cunha, Francisco Q. Gonçalves, Joana I. Real, Diana G. Ferreira
Publikováno v:
Cerebral Cortex
© The Author 2015. Published by Oxford University Press. All rights reserved.
Abnormal accumulation of aggregated α-synuclein (aSyn) is a hallmark of sporadic and familial Parkinson's disease (PD) and related synucleinopathies. Recent studies
Abnormal accumulation of aggregated α-synuclein (aSyn) is a hallmark of sporadic and familial Parkinson's disease (PD) and related synucleinopathies. Recent studies
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ee0d8e9d8b33491d562a437b79f83ddc
https://hdl.handle.net/10451/51438
https://hdl.handle.net/10451/51438