Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Uwe Rückschloß"'
Autor:
Berin Upcin, Erik Henke, Florian Kleefeldt, Helene Hoffmann, Andreas Rosenwald, Ster Irmak-Sav, Huseyin Bertal Aktas, Uwe Rückschloß, Süleyman Ergün
Publikováno v:
Cells, Vol 10, Iss 7, p 1719 (2021)
Blocking tumor vascularization has not yet come to fruition to the extent it was hoped for, as angiogenesis inhibitors have shown only partial success in the clinic. We hypothesized that under-appreciated vascular wall-resident stem and progenitor ce
Externí odkaz:
https://doaj.org/article/d537661bd9ee4c279af165a8476f4bd5
Autor:
Berin, Upcin, Erik, Henke, Florian, Kleefeldt, Helene, Hoffmann, Andreas, Rosenwald, Ster, Irmak-Sav, Huseyin Bertal, Aktas, Uwe, Rückschloß, Süleyman, Ergün
Publikováno v:
Cells, Vol 10, Iss 1719, p 1719 (2021)
Cells
Volume 10
Issue 7
Cells
Volume 10
Issue 7
Blocking tumor vascularization has not yet come to fruition to the extent it was hoped for, as angiogenesis inhibitors have shown only partial success in the clinic. We hypothesized that under-appreciated vascular wall-resident stem and progenitor ce
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid_dedup__::612ae08d9fa6dcae731981cd9afb335f
https://opus.bibliothek.uni-wuerzburg.de/files/24257/cells-10-01719.pdf
https://opus.bibliothek.uni-wuerzburg.de/files/24257/cells-10-01719.pdf
Autor:
Michael Gekle, Gerald Schwerdt, Uwe Rückschloss, Hans-Ulrich Humpf, Christopher Röhl, Ines Ferse, Ulrike Rottkord, Marie-Christin Schulz
Publikováno v:
Archives of Toxicology. 91:1461-1471
The enigma why the mycotoxin ochratoxin A (OTA) impairs cell and organ function is still not solved. However, an interaction with target molecules is a prerequisite for any observed adverse effect. This interaction depends on characteristics of the t
Publikováno v:
Arteriosclerosis, Thrombosis, and Vascular Biology. 20:61-69
Abstract —Three 3-hydroxy-3-methylglutaryl coenzyme A (HMG-CoA) reductase inhibitors (HCRIs), atorvastatin, pravastatin, and cerivastatin, inhibited phorbol ester–stimulated superoxide anion (O 2 − ) formation in endothelium-intact segments of