Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Urs Hasse"'
Autor:
D. Baumann, Huldrych F. Günthard, Barbara Hasse, Rainer Weber, Felix Grimm, Urs Hasse, Christine Schraner, Anja Faeh, Günther Burg, Alexander Mathis
Publikováno v:
Clinical Infectious Diseases. 40:e120-e124
We describe here a case of disseminated cutaneous leishmaniasis due to Leishmania major in a severely immunocompromised patient from Burkino Faso, Africa, who is infected with human immunodeficiency virus-1. The skin lesions failed to respond to full
Autor:
Andreas Tobler, Feyruz V. Rassool, Elisabeth Oppliger, Mario P. Tschan, Martin F. Fey, S. Tiong Ong, U. R. Peters, Urs Hasse, Bettina Borisch
Publikováno v:
Oncogene. 18:79-85
Aberrant FHIT mRNA transcripts are present in malignant and normal haematopoiesis, but absence of FHIT protein is restricted to leukaemia Alterations of the recently cloned fragile histidine triad (FHIT) gene at chromosome 3p14.2 are frequent in a va
Autor:
Vahid Djamei, Urs Hasse, Sergio Chimenti, Roger Kropf, H. Peter Soyer, Günter Burg, Claudio Cipolat
Publikováno v:
Dermatology (Basel, Switzerland). 210(2)
Telemedicine is the practice of healthcare using interactive audio, visual and data communication. This includes healthcare delivery, diagnoses, consultation and treatment as well as education and transfer of medical data. The transmission of multime
Autor:
Elisabeth Oppliger Leibundgut, Bettina Borisch, Martin F. Fey, Urs Hasse, Jean-François Cajot, Alex M. Garvin, Marianne Tinguely, Andreas Tobler
Publikováno v:
Hasse, Urs; Tinguely, Marianne; Oppliger Leibundgut, Elisabeth; Cajot, Jean-François; Garvin, Alex M.; Tobler, Andreas; Borisch, Bettina; Fey, Martin F. (1999). Clonal Loss of Heterozygosity in Microdissected Hodgkin and Reed-Sternberg Cells. Journal of the National Cancer Institute JNCI, 91(18), pp. 1581-1583. Oxford University Press 10.1093/jnci/91.18.1581
The typical Hodgkin and ReedSternberg (HRS) cells are thought to represent the malignant cellular elements of Hodgkin’s disease (HD). The detection of immunoglobulin gene rearrangements in HRS cells indicates that they are clonally derived from B c