Zobrazeno 1 - 10
of 34
pro vyhledávání: '"Urpo Lappalainen"'
Publikováno v:
Acta Paediatrica. 100:1206-1212
Aim: To assess whether the levels of inflammatory and anti-inflammatory proteins in gastric fluid of premature infants shortly after birth are associated with the development of bronchopulmonary dysplasia (BPD). Methods: Gastric fluid retrieved withi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::2f88468691e3589cf766bcbaf26c6302
https://doi.org/10.1111/j.1651-2227.2011.02286.x
https://doi.org/10.1111/j.1651-2227.2011.02286.x
Publikováno v:
Pediatric Research. 69:312-318
Bronchopulmonary dysplasia (BPD) is a common inflammatory lung disease in premature infants. To study the hypothesis that the sensitivity of the lung to inflammatory injury depends on the developmental stage, we studied postnatal lung development in
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d499c6c9e18286b5d25c4485baf0cff5
https://doi.org/10.1203/pdr.0b013e31820bcb2a
https://doi.org/10.1203/pdr.0b013e31820bcb2a
Publikováno v:
American Journal of Respiratory Cell and Molecular Biology. 42:149-160
Little is known about the influence of maternal inflammation on neonatal outcome. Production of IL-1beta in the lungs of newborn infants is associated with bronchopulmonary dysplasia. Using bitransgenic (bi-TG) mice in which human (h) IL-1beta is exp
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::99556518f6d2d6ea51b15d8f58ca8dff
https://doi.org/10.1165/rcmb.2008-0287oc
https://doi.org/10.1165/rcmb.2008-0287oc
Publikováno v:
American Journal of Respiratory Cell and Molecular Biology. 36:32-42
Pulmonary inflammation and increased production of the inflammatory cytokine IL-1beta are associated with the development of bronchopulmonary dysplasia (BPD) in premature infants. To study the actions of IL-1beta in the fetal and newborn lung in vivo
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c0ed161d3158421a32d82355f9296e42
https://doi.org/10.1165/rcmb.2006-0116oc
https://doi.org/10.1165/rcmb.2006-0116oc
Autor:
R. Michael Rodriguez, Elizabeth A. Perkett, Gary Bienvenu, Jeffrey T. Rogers, Richard W. Light, D.K.-W. Cheng, Urpo Lappalainen
Publikováno v:
Chest. 116:760-765
The purpose of this study was to analyze the relationship of the pleural fluid vascular endothelial growth factor (VEGF) level with the diagnostic category and with the pleural fluid characteristics in a group of 70 patients.The VEGF levels of consec
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c40c5460e24b4c41d046ecabb2ef14f9
https://doi.org/10.1378/chest.116.3.760
https://doi.org/10.1378/chest.116.3.760
Publikováno v:
Pediatric Research. 45:247-254
Inhaled nitric oxide (iNO) is a specific pulmonary vasodilator. By serving as a pro-oxidant or antioxidant, iNO may influence other pulmonary functions as well. This study was designed to test the hypothesis that iNO affects the alveolar lining after
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::334057f367fae70e73f618ef003eef3a
https://doi.org/10.1203/00006450-199902000-00016
https://doi.org/10.1203/00006450-199902000-00016
Publikováno v:
Pediatric Research. 41:105-109
Granulocyte-macrophage colony-stimulating factor (GM-CSF), a cytokine that promotes white cell maturation, participates in the metabolism of pulmonary surfactant. Little is known on the production of GM-CSF during pregnancy or the neonatal period. We
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ebed7a78a6cc28ca15595c61390f5b0b
https://doi.org/10.1203/00006450-199701000-00016
https://doi.org/10.1203/00006450-199701000-00016
Publikováno v:
Journal of Applied Physiology. 80:2035-2043
Inhaled nitric oxide (NO) may modify surfactant either by interacting with the surfactant complex or by changing the capacity of the proteins of the epithelial lining fluid to inhibit the surface activity. Natural surfactant was exposed to NO (80 par
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::64b1beb5f56e2c7a2708469edb99e3c7
https://doi.org/10.1152/jappl.1996.80.6.2035
https://doi.org/10.1152/jappl.1996.80.6.2035
Publikováno v:
Seminars in Perinatology. 20:194-205
The production of pulmonary surfactant, a complex of lipids and proteins that reduces surface tension at the alveolar air-liquid interface, is developmentally regulated. Several hormones, most notably glucocorticoids, are known to accelerate maturati
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f4dd24a716c28b156e233ca767fc4c8d
https://doi.org/10.1016/s0146-0005(96)80048-6
https://doi.org/10.1016/s0146-0005(96)80048-6
Publikováno v:
American journal of respiratory cell and molecular biology. 47(6)
The contribution of neutrophils and CXC chemokines to the pathogenesis of bronchopulmonary dysplasia is not well defined. The transgenic expression of IL-1β in the pulmonary epithelium causes lung inflammation and disrupts alveolar development in in
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::072b1b2c57a51bcf9ae1f5e297f3c622
https://pubmed.ncbi.nlm.nih.gov/22865624
https://pubmed.ncbi.nlm.nih.gov/22865624