Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Umeer Ashraf"'
Autor:
Manpreet K. Chadha, Annette Sunga, Mary Nesline, Wei Tan, Lili Tian, C. Silliman, Umeer Ashraf, Bruce W. Hollis, Candace S. Johnson, Donald L. Trump, Marwan Fakih
Publikováno v:
BJU International. 104:909-914
OBJECTIVE To assess the frequency of vitamin D deficiency among men with prostate cancer, as considerable epidemiological, in vitro, in vivo and clinical data support an association between vitamin D deficiency and prostate cancer outcome. PATIENTS,
Autor:
C. Silliman, Donald L. Trump, Paul Escott, Valencia Payne, David Lawrence, Ellis G. Levine, Lili Tian, Manpreet K. Chadha, Umeer Ashraf
Publikováno v:
The Prostate. 68:1461-1466
BACKGROUND Preclinical evidence supports the role of estrogen receptor signaling in prostate cancer. In this trial we investigated the tolerability and efficacy of fulvestrant, a pure estrogen receptor antagonist, in the treatment of castration resis
Autor:
Umeer Ashraf, Myron S. Czuczman, Francisco J. Hernandez-Ilizaliturri, John F. Gibbs, Cory Marvis
Publikováno v:
Blood. 114:3727-3727
Abstract 3727 Poster Board III-663 Deacetylases (DACs) are enzymes that remove the acetyl groups from target proteins, leading to regulation of gene transcription and other cellular processes. Entinostat (SNDX-275) is a novel and potent DAC inhibitor
Autor:
Zale P. Bernstein, Philip L. McCarthy, Francisco J. Hernandez-Ilizaliturri, Shannon Smiley, Myron S. Czuczman, Minoo Battiwala, Ritika Mahajan, Umeer Ashraf, Theresa Hahn, Asher A. Chanan-Khan
Publikováno v:
Blood. 112:1138-1138
Despite the improved outcome in patients with DLBCL treated with rituximab (R) in combination with systemic chemotherapy (R + chemotherapy), a significant number of patients either relapse or fail to respond as a consequence of resistant disease. HDC
Autor:
Anjana Elefante, Umeer Ashraf, Raymond Cruz, Myron S. Czuczman, Paul K. Wallace, Francisco J. Hernandez-llizaliturri
Publikováno v:
Blood. 110:2054-2054
Cancer cells, including B-cell lymphoproliferative disorders, sculpt their phenotype in an attempt to escape not only immune-surveillance but also evade the anti-tumor activity of biological agents and/or chemotherapy drugs including rituximab. In an