Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Ulrike Schrameck"'
Autor:
Andrea Grund, Malgorzata Szaroszyk, Mortimer Korf‐Klingebiel, Mona Malek Mohammadi, Felix A Trogisch, Ulrike Schrameck, Anna Gigina, Christopher Tiedje, Matthias Gaestel, Theresia Kraft, Jan Hegermann, Sandor Batkai, Thomas Thum, Andreas Perrot, Cris dos Remedios, Eva Riechert, Mirko Völkers, Shirin Doroudgar, Andreas Jungmann, Ralf Bauer, Xiaoke Yin, Manuel Mayr, Kai C Wollert, Andreas Pich, Hua Xiao, Hugo A Katus, Johann Bauersachs, Oliver J Müller, Joerg Heineke
Publikováno v:
EMBO Molecular Medicine, Vol 11, Iss 10, Pp 1-20 (2019)
Abstract Pathological cardiac overload induces myocardial protein synthesis and hypertrophy, which predisposes to heart failure. To inhibit hypertrophy therapeutically, the identification of negative regulators of cardiomyocyte protein synthesis is n
Externí odkaz:
https://doaj.org/article/f9470b276260446599ccfd463aeb960b
Autor:
Mona Malek Mohammadi, Badder Kattih, Andrea Grund, Natali Froese, Mortimer Korf‐Klingebiel, Anna Gigina, Ulrike Schrameck, Carsten Rudat, Qiangrong Liang, Andreas Kispert, Kai C Wollert, Johann Bauersachs, Joerg Heineke
Publikováno v:
EMBO Molecular Medicine, Vol 9, Iss 2, Pp 265-279 (2017)
Abstract Heart failure is often the consequence of insufficient cardiac regeneration. Neonatal mice retain a certain capability of myocardial regeneration until postnatal day (P)7, although the underlying transcriptional mechanisms remain largely unk
Externí odkaz:
https://doaj.org/article/df2851935f7949dea1f62a64768c2d67
Autor:
Mona Malek Mohammadi, Badder Kattih, Andrea Grund, Natali Froese, Mortimer Korf‐Klingebiel, Anna Gigina, Ulrike Schrameck, Carsten Rudat, Qiangrong Liang, Andreas Kispert, Kai C Wollert, Johann Bauersachs, Joerg Heineke
Publikováno v:
EMBO Molecular Medicine, Vol 11, Iss 6, Pp n/a-n/a (2019)
Externí odkaz:
https://doaj.org/article/ba9511e0c2b645b48c20814daa8ebb5b
Autor:
G. William Wong, Hugo A. Katus, Oliver J. Müller, Daniela Fraccarollo, Andrea Grund, Astrid Breitbart, Joerg Heineke, Johann Bauersachs, Mona Malek Mohammadi, Kai C. Wollert, Florian Brandes, Mortimer Korf-Klingebiel, Mahesh Appari, Honghui Wang, Carolin Zwadlo, Natali Froese, Mona Nemer, Malgorzata Szaroszyk, Gesine M. Scharf, Ulrike Schrameck
Publikováno v:
Circulation Research. 120:66-77
Rationale: Myocardial endothelial cells promote cardiomyocyte hypertrophy, possibly through the release of growth factors. The identity of these factors, however, remains largely unknown, and we hypothesized here that the secreted CTRP9 (C1q-tumor ne
Autor:
Carsten Rudat, Johann Bauersachs, Anna Gigina, Joerg Heineke, Badder Kattih, Qiangrong Liang, Natali Froese, Mortimer Korf-Klingebiel, Andrea Grund, Ulrike Schrameck, Kai C. Wollert, Mona Malek Mohammadi, Andreas Kispert
Publikováno v:
EMBO Molecular Medicine
EMBO Molecular Medicine, Vol 11, Iss 6, Pp n/a-n/a (2019)
EMBO Molecular Medicine, Vol 11, Iss 6, Pp n/a-n/a (2019)
Heart failure is often the consequence of insufficient cardiac regeneration. Neonatal mice retain a certain capability of myocardial regeneration until postnatal day (P)7, although the underlying transcriptional mechanisms remain largely unknown. We
Autor:
Sandor Batkai, Ulrike Schrameck, Christopher Tiedje, Hugo A. Katus, Oliver J. Müller, Shirin Doroudgar, Anna Gigina, Jan Hegermann, Mirko Völkers, Cris dos Remedios, Theresia Kraft, Matthias Gaestel, Hua Xiao, Andrea Grund, Felix A. Trogisch, Eva Riechert, Thomas Thum, Mortimer Korf-Klingebiel, Malgorzata Szaroszyk, Xiaoke Yin, Joerg Heineke, Andreas Perrot, Ralf Bauer, Manuel Mayr, Andreas Jungmann, Mona Malek Mohammadi, Johann Bauersachs, Andreas Pich, Kai C. Wollert
Publikováno v:
EMBO Molecular Medicine
EMBO Molecular Medicine, Vol 11, Iss 10, Pp n/a-n/a (2019)
EMBO Molecular Medicine, Vol 11, Iss 10, Pp n/a-n/a (2019)
Pathological cardiac overload induces myocardial protein synthesis and hypertrophy, which predisposes to heart failure. To inhibit hypertrophy therapeutically, the identification of negative regulators of cardiomyocyte protein synthesis is needed. He
Autor:
Johann Bauersachs, Ulrike Schrameck, Kai C. Wollert, Mortimer Korf-Klingebiel, Natali Froese, Florian Brandes, Jörg Heineke, Astrid Breitbart, Oliver J. Müller
Publikováno v:
Circulation Research. 113
Background: CTRP9 (also called C1qtnf9) is a newly discovered secreted protein and a paralog of adiponectin. The biological functions of CTRP9, however, are still largely unknown. Results: Although previous data from a semi-quantitative real-time PCR