Zobrazeno 1 - 10
of 31
pro vyhledávání: '"Ulrike Harre"'
Autor:
Cecilia Engdahl, Albert Bondt, Ulrike Harre, Jasmin Raufer, René Pfeifle, Alessandro Camponeschi, Manfred Wuhrer, Michaela Seeling, Inga-Lill Mårtensson, Falk Nimmerjahn, Gerhard Krönke, Hans U. Scherer, Helena Forsblad-d’Elia, Georg Schett
Publikováno v:
Arthritis Research & Therapy, Vol 20, Iss 1, Pp 1-11 (2018)
Abstract Background Rheumatoid arthritis (RA) preferentially affects women, with the peak incidence coinciding with estrogen decrease in menopause. Estrogen (E2) may therefore have intrinsic immune-regulatory properties that vanish with menopause. Fc
Externí odkaz:
https://doaj.org/article/e440625e94dc4c1c8ab28633f770c1aa
Autor:
Anja Derer, Bettina Groetsch, Ulrike Harre, Christina Böhm, Jennifer Towne, Georg Schett, Silke Frey, Axel J Hueber
Publikováno v:
PLoS ONE, Vol 9, Iss 8, p e101954 (2014)
INTRODUCTION: Interleukin (IL)-36α is a newly described member of the IL-1 cytokine family with a known inflammatory and pathogenic function in psoriasis. Recently, we could demonstrate that the receptor (IL-36R), its ligand IL-36α and its antagoni
Externí odkaz:
https://doaj.org/article/0f4e842f23ba451d8eec20a2a2e2cc17
Autor:
Georg Schett, Ulrike Harre
Publikováno v:
Oxford Textbook of Rheumatoid Arthritis
Structural damage of cartilage and bone tissue is a hallmark of rheumatoid arthritis (RA). The resulting joint destruction constitutes one of the major disease consequences for patients and creates a significant burden for the society. The main cells
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::62b8862cc0984fa9d50ae076175d3845
https://doi.org/10.1093/med/9780198831433.003.0009
https://doi.org/10.1093/med/9780198831433.003.0009
Autor:
Ulrike Harre, Barbara Hauser
Publikováno v:
Calcified Tissue International. 102:522-532
Many autoimmune diseases are associated with deranged bone metabolism. The resulting localized or systemic bone loss can compromise the quality of life of patients by causing local bone deformities or fragility fractures. There is emerging evidence t
Autor:
Ulrike Harre, Georg Schett, Anja Derer, Aline Bozec, Dominic Simon, Martin Herrmann, Fabian T. Andes, Patrick Lezuo
Publikováno v:
Bone. 105:35-41
Bone tissue undergoes permanent and lifelong remodeling with a concerted action of bone-building osteoblasts and bone-resorbing osteoclasts. A precise cooperation between those two cell types is critical in the complex process of bone renewal. Galect
Autor:
Christiane Maier, Jingang Huang, Ulrike Harre, Jörg H W Distler, Clara Dees, Alina Soare, Christian Beyer, Georg Schett, Lutz Wollin, Oliver Distler, Yun Zhang, Chih-Wei Chen
Publikováno v:
Annals of the Rheumatic Diseases. 76:1941-1948
BackgroundNintedanib is an inhibitor targeting platelet-derived growth factor receptor, fibroblast growth factor receptor and vascular endothelial growth factor receptor tyrosine kinases that has recently been approved for the treatment of idiopathic
Autor:
Georg Schett, Ulrike Harre
Publikováno v:
Seminars in Immunopathology. 39:355-363
Structural damage of cartilage and bone tissue is a hallmark of rheumatoid arthritis (RA). The resulting joint destruction constitutes one of the major disease consequences for patients and creates a significant burden for the society. The main cells
Publikováno v:
Journal of Bone and Mineral Research. 32:1681-1691
Autor:
Georg Schett, Dirk Mielenz, Wolfgang Baum, Jonas Hahn, Deborah Kienhöfer, Martin Herrmann, Katharina Dietel, Ulrike Harre, Pavitra Purohit, Stefanie C. Lang
Publikováno v:
The Journal of Immunology. 198:2394-2402
The prevalence of neurodegenerative disease and arthritis increases with age. Despite both processes being associated with immune activation and inflammation, little is known about the mechanistic interactions between neurodegenerative disease and ar
Autor:
Stefan Rehart, Elena Neumann, Ulrike Harre, Silvia Capellino, Ulf Müller-Ladner, L. Salinas Tejedor, J. Steinmeyer, Georg Schett, Markus Rickert
Publikováno v:
Brain, Behavior, and Immunity. 76:e17-e18
Background Dysfunction of either osteoclastic bone resorption or osteoblastic bone formation leads to bone disorders such in rheumatoid arthritis (RA). Several therapies target osteoclasts to reduce bone degradation, but treatments able to induce bon