Zobrazeno 1 - 10
of 10
pro vyhledávání: '"Ulrich Popper"'
Autor:
Helmut Schaider, Helmut Bergler, Meenhard Herlyn, Christine Hafner, Melitta Kitzwoegerer, Heimo Breiteneder, Dinoop Ravindran Menon, Ulrich Popper, Christian Wels, Heinz Hammerlindl, Ehsan Bonyadi Rad
Supplemental Figure Legends for Supplemental Figures 1 to 5
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0868ca7dbb4395e45d6dd8aa0602f57b
https://doi.org/10.1158/0008-5472.22410959
https://doi.org/10.1158/0008-5472.22410959
Autor:
Helmut Schaider, Helmut Bergler, Meenhard Herlyn, Christine Hafner, Melitta Kitzwoegerer, Heimo Breiteneder, Dinoop Ravindran Menon, Ulrich Popper, Christian Wels, Heinz Hammerlindl, Ehsan Bonyadi Rad
SF1: Phenotypic and functional changes of 451Lu, WM9 and WM164 following N4ICD overexpression SF2: Immunoblots and immunohistochemistry of EMT markers and Notch4 SF3: Details of CSL binding to Snail2 and Twist1 promoter regions SF4: Immunoblots of ov
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::28fe04977c60eb4c3f781526c094f64c
https://doi.org/10.1158/0008-5472.22410956
https://doi.org/10.1158/0008-5472.22410956
Autor:
Helmut Schaider, Helmut Bergler, Meenhard Herlyn, Christine Hafner, Melitta Kitzwoegerer, Heimo Breiteneder, Dinoop Ravindran Menon, Ulrich Popper, Christian Wels, Heinz Hammerlindl, Ehsan Bonyadi Rad
Supplemental Materials and Methods used to create Supplemental Figures only in addition to Materials and Methods listed in the main manuscript
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::84199b9bba13cd2b02b63ead794c7f08
https://doi.org/10.1158/0008-5472.22410953
https://doi.org/10.1158/0008-5472.22410953
Autor:
Helmut Schaider, Helmut Bergler, Meenhard Herlyn, Christine Hafner, Melitta Kitzwoegerer, Heimo Breiteneder, Dinoop Ravindran Menon, Ulrich Popper, Christian Wels, Heinz Hammerlindl, Ehsan Bonyadi Rad
The effects of Notch signaling are context-dependent and both oncogenic and tumor-suppressive functions have been described. Notch signaling in melanoma is considered oncogenic, but clinical trials testing Notch inhibition in this malignancy have not
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::04a20c9b2f91ef09810426dfbf0248e1
https://doi.org/10.1158/0008-5472.c.6508118.v1
https://doi.org/10.1158/0008-5472.c.6508118.v1
Autor:
Holger Rumpold, Ulrich Popper
Publikováno v:
memo - Magazine of European Medical Oncology. 14:180-183
During this year’s virtual congress of the European Society of Oncology (ESMO) some practice-changing abstracts were presented. Especially immunotherapy (IO) has found its way into the treatment of esophageal (EC) and gastric cancer (GC) in both th
Autor:
Ulrich Popper, Bernhard Doleschal
Publikováno v:
memo - Magazine of European Medical Oncology. 14:115-118
Recent advances in molecular diagnostics classify Langerhans cell histiocytosis as an orphan disease strongly dependent on alterations in MAP kinase pathway, most prominently BRAF V600E mutation. Molecular targeted therapy with vemurafenib often lead
Publikováno v:
Wiener klinisches Magazin. 19:96-106
Autor:
Ulrich Popper, Christine Hafner, Heinz Hammerlindl, Melitta Kitzwoegerer, Dinoop Ravindran Menon, Meenhard Herlyn, Christian Wels, Helmut Bergler, Ehsan Bonyadi Rad, Helmut Schaider, Heimo Breiteneder
Publikováno v:
Cancer Research. 76:1690-1697
The effects of Notch signaling are context-dependent and both oncogenic and tumor-suppressive functions have been described. Notch signaling in melanoma is considered oncogenic, but clinical trials testing Notch inhibition in this malignancy have not
Publikováno v:
memo - Magazine of European Medical Oncology. 7:102-106
Follow-up after curative treatment of patients with gastrointestinal cancers aims to detect recurrent disease and to provide curative treatment to prolong overall survival if possible. This, however, has only been shown for colorectal cancers. Regula
Autor:
Holger Rumpold, Ulrich Popper
Publikováno v:
Journal of Clinical Oncology. 34:590-590
590 Background: Carcinogenesis is considered to be an evolutionary process by natural selection of cell clones which have acquired advantageous heritable characteristics. This adaption process, which resembles Darwin’s evolutionary theory, has also