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pro vyhledávání: '"Ulrich A. Simanowski"'
Autor:
Ulrich A. Simanowski, Helmut K. Seitz
Publikováno v:
Alcohol and the Gastrointestinal Tract ISBN: 9781315149844
This chapter discusses gastrointestinal cell proliferation in association with carcinogenesis. Changes of cell turnover are especially described in the colon where an enormous body of literature has been recently accumulated linking proliferation wit
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::a814678a4090a25f4b1c735fa48e2c4b
https://doi.org/10.4324/9781315149844-15
https://doi.org/10.4324/9781315149844-15
Autor:
Nils Homann, Ulrich A. Simanowski, Helmut K. Seitz, Franz X. Bosch, M Knühl, Rüdiger Waldherr, Christian Conradt, L Arce
Publikováno v:
Gut. 49:418-422
BACKGROUND—Epidemiological data indicate an increased risk for rectal cancer following chronic alcohol consumption. As chronic ethanol ingestion leads to rectal hyperregeneration in experimental animals, indicating a state of increased susceptibili
Autor:
Gudrun Poeschl, Christian Conradt, Felix Stickel, Robert S. McCuskey, Renate Urbaschek, Detlef Schuppan, Ulrich A. Simanowski, Helmut K. Seitz, Christine Oesterling
Publikováno v:
Digestive Diseases and Sciences. 46:2025-2032
Hepatic fibrosis in alcoholic liver disease often heralds progression to cirrhosis and, therefore, noninvasive parameters are required for early diagnosis and follow-up. Collagens VI and XIV, procollagen-III-N-propeptide, hyaluronic acid, and active
The past 20 years have seen a surge of research into colorectal cancer, which is a reflection of the need to improve our methods of treating patients suffering from this increasingly common form of cancer. Greater knowledge of the basic mechanisms in
Autor:
Ulrich A. Simanowski, Rüdiger Waldherr, Luis Arce, Xavier Parés, Thomas Keil, Rolf Aderjan, Robert M. Russell, Helmut K. Seitz, Fritz Klee, Christian Conradt, Gerlinde Egerer, Carl Oneta, Felix Stickel
Publikováno v:
Digestion. 59:314-320
Background/Aims: Ethanol is metabolized by alcohol dehydrogenase in the human stomach. This metabolism contributes to the so-called first-pass metabolism of ethanol which is affected by gender, medication, and morphological alterations of the gastric
Publikováno v:
Alcohol. 12:111-115
Chronic ethanol consumption is a major risk factor for oropharyngeal, esophageal, and rectal cancer. Because hyperregenerative gastrointestinal mucosa has an increased susceptibility towards chemical carcinogens and thus influences carcinogenesis, va
Autor:
Rüdiger Waldherr, Robert M. Russell, Ulrich A. Simanowski, Felix Stickel, Heinz Maier, Helmut K. Seitz, Don Smith, Paolo M. Suter
Publikováno v:
JNCI Journal of the National Cancer Institute. 85:2030-2033
Autor:
R. Eckey, H. W. Goedde, G. Egerer, D P Agarwal, Helmut K. Seitz, Ulrich A. Simanowski, Rüdiger Waldherr, J P von Wartburg
Publikováno v:
Gut. 34:1433-1437
As various isoenzymes of gastric alcohol dehydrogenase exist and as the effect of sex and age on these enzymes is unknown, this study measured the activity of gastric alcohol dehydrogenase at high and low ethanol concentrations in endoscopic biopsy s
Publikováno v:
Research in Experimental Medicine. 192:205-212
It has been reported that aging strikingly decreases in vivo ethanol metabolism in F344 rats without major effects on hepatic alcohol dehydrogenase (ADH) activity. Because hepatic ADH activity is not always rate limiting in the oxidation of ethanol,