Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Ulandt Kim"'
Publikováno v:
PLoS Pathogens, Vol 2, Iss 10 (2006)
Externí odkaz:
https://doaj.org/article/0f78c957bcb54c5ca62c6a42cb82c1c0
Publikováno v:
PLoS Pathogens, Vol 2, Iss 5, p e43 (2006)
By manipulating arthropod reproduction worldwide, the heritable endosymbiont Wolbachia has spread to pandemic levels. Little is known about the microbial basis of cytoplasmic incompatibility (CI) except that bacterial densities and percentages of inf
Externí odkaz:
https://doaj.org/article/d846c883799d4bf893bb88bbd74ddb99
Autor:
David S. Reiner, Scott C. Dawson, Hilary G. Morrison, Anjali Prabhu, Heidi G. Elmendorf, Ulandt Kim, Mitchell L. Sogin, Adrian B. Hehl, Claudia I. Reich, Erica Lasek-Nesselquist, Rodney D. Adam, Michael Holder, Gerard Manning, Susan M. Huse, Michael J. Cipriano, Feng Chen, Staffan G. Svärd, Nora Q.E. Passamaneck, Anuranjini Nigam, John Samuelson, Aaron A. Best, W. Zacheus Cande, Stephen B. Aley, Daniel Palm, Barbara J. Davids, Frances D. Gillin, Gary J. Olsen, Julie E. J. Nixon, Andrew G. McArthur
Publikováno v:
Science. 317:1921-1926
The genome of the eukaryotic protist Giardia lamblia, an important human intestinal parasite, is compact in structure and content, contains few introns or mitochondrial relics, and has simplified machinery for DNA replication, transcription, RNA proc
Publikováno v:
Genome Research. 9:325-333
Arabidopsis thaliana BAC T07M07 encoding the abscisic acid-insensitive 4 (ABI4) locus has been sequenced completely. It contains a 95,713-bp insert and 24 predicted genes. Most putative genes were confirmed by gel-based RNA profiling and a cluster of
Publikováno v:
PLoS Pathogens
In PLoS Pathogens, volume 2, issue 5:10.1371/journal.ppat.0020043 The length of the inset bar in Figure 5C should equal 50 nm, not 100 nm as originally stated.
Publikováno v:
PLoS Pathogens, Vol 2, Iss 5, p e43 (2006)
PLoS Pathogens
PLoS Pathogens
By manipulating arthropod reproduction worldwide, the heritable endosymbiont Wolbachia has spread to pandemic levels. Little is known about the microbial basis of cytoplasmic incompatibility (CI) except that bacterial densities and percentages of inf