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pro vyhledávání: '"Tzu-Yue Shiu"'
Publikováno v:
PLoS ONE, Vol 12, Iss 7, p e0181299 (2017)
[This corrects the article DOI: 10.1371/journal.pone.0061089.].
Externí odkaz:
https://doaj.org/article/c6a04452d43c47ea94b93638928b099f
Publikováno v:
PLoS ONE, Vol 8, Iss 4, p e61089 (2013)
Hepatitis C virus (HCV) has been reported to regulate cellular microRNAs. The HCV core protein is considered to be a potential oncoprotein in HCV-related hepatocellular carcinoma, but HCV core-modulated cellular microRNAs are unknown. The HCV core pr
Externí odkaz:
https://doaj.org/article/4a8c848556374ee08270134777bbc078
Autor:
Tzu-Yue Shiu, 徐祖岳
101
Chapter I. The Study of Cellular MicroRNA Expression Regulated by Hepatitis C Virus Core Protein in Human Hepatoma Cell Lines Chronic hepatitis C virus (HCV) infection contributes to chronic hepatitis, liver cirrhosis, and hepatocellular car
Chapter I. The Study of Cellular MicroRNA Expression Regulated by Hepatitis C Virus Core Protein in Human Hepatoma Cell Lines Chronic hepatitis C virus (HCV) infection contributes to chronic hepatitis, liver cirrhosis, and hepatocellular car
Externí odkaz:
http://ndltd.ncl.edu.tw/handle/41998307147172616671
Autor:
Tsai-Yuan Hsieh, Chung-Bao Hsieh, Hsin-Hung Huang, An-Chieh Feng, Wei-Kuo Chang, Tien-Yu Huang, Hsuan-Hwai Lin, Tzu-Yue Shiu, Yu-Lueng Shih
Publikováno v:
Life Sciences. 284:119708
Hepatocellular carcinoma (HCC) is a primary malignancy of the hepatocyte. Interleukin enhancer binding factor 2 (ILF2) plays a role in the development of HCC. However, the regulatory mechanisms of ILF2 expression in HCC remain unclear. In this study,
Autor:
Tsai-Yuan Hsieh, Shih-Ming Huang, An-Chieh Feng, Yu-Lueng Shih, Wei-Kuo Chang, Chung-Bao Hsieh, Hsuan-Hwai Lin, Tien-Yu Huang, Tzu-Yue Shiu
Publikováno v:
Journal of Molecular Medicine. 95:629-639
Hepatitis C virus (HCV) infection is a major cause of chronic hepatitis, liver cirrhosis, and hepatocellular carcinoma (HCC). HCV core protein is considered as a positive regulator of telomerase activity. In this study, we focused on the deregulated
Autor:
Hsuan Hwai Lin, Tsai Yuan Hsieh, Heng Cheng Chu, Wei-Kuo Chang, Hsin-Hung Huang, Tzu Yue Shiu, Yu-Lueng Shih
Publikováno v:
Liver International. 35:2050-2056
Background & Aims Gilbert's syndrome causes pharmacological variation in drug glucuronidation and unexpected toxicity from therapeutic agents. The two common genotypes of Gilbert's syndrome are a dinucleotide polymorphism (TA)7 in TATA-Box as well as
Autor:
N. F. Chu, Heng-Cheng Chu, Tsai-Yuan Hsieh, Wei-Kuo Chang, Tsu Yi Chao, Hsin-Hung Huang, Tzu-Yue Shiu, Y. C. Chao
Publikováno v:
Genetics and Molecular Research. 13:670-679
Gilbert's syndrome is suspected in patients with unconjugated hyperbilirubinemia caused by decreased activity of the UDP-glucuronosyltransferase 1A1 (UGT1A1) gene in the absence of abnormal liver function and hemolysis. The major genetic variants und
Autor:
King-Song Jeng, Tzu-Yue Shiu, Michael M. C. Lai, Tsai-Yuan Hsieh, Wei-Kuo Chang, Peng-Jen Chen, Heng-Cheng Chu, You-Chen Chao, Hsuan-Hwai Lin, Shih-Ming Huang, Tai-Chi Lee
Publikováno v:
Pharmacogenetics and Genomics. 17:229-236
Gilbert's syndrome is a congenital, nonhemolytic, unconjugated hyperbilirubinemia. The most common genotype of Gilbert's syndrome is the homozygous polymorphism, A(TA)7TAA, in the promoter of the gene for UDP-glucuronosyltransferase 1A1 (UGT1A1), wit
Publikováno v:
PLoS ONE
PLoS ONE, Vol 12, Iss 7, p e0181299 (2017)
PLoS ONE, Vol 8, Iss 4, p e61089 (2013)
PLoS ONE, Vol 12, Iss 7, p e0181299 (2017)
PLoS ONE, Vol 8, Iss 4, p e61089 (2013)
Background Hepatitis C virus (HCV) has been reported to regulate cellular microRNAs. The HCV core protein is considered to be a potential oncoprotein in HCV-related hepatocellular carcinoma, but HCV core-modulated cellular microRNAs are unknown. The
Autor:
Tien-Yu Huang, Heng-Cheng Chu, Yu-Lueng Shih, Wei-Kuo Chang, Tzu-Yue Shiu, Tsai-Yuan Hsieh, Shih-Ming Huang
Publikováno v:
The Biochemical journal. 449(3)
Jaundice or hyperbilirubinaemia is a common complication of sepsis. UGT1A1 (UDP-glucuronosyltransferase 1A1) is a critical gene for bilirubin metabolism and irinotecan detoxification. However, the molecular pathogenesis of hyperbilirubinaemia during