Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Twinkle Chowdhury"'
Autor:
Pete Clarner, Shukkwan K. Lau, Twinkle Chowdhury, Edward Guilmette, Patrick Trapa, Shih-Ching Lo, Shen Shen
Publikováno v:
Molecular Therapy: Methods & Clinical Development, Vol 23, Iss , Pp 68-77 (2021)
Robust assays to quantify adeno-associated virus (AAV) vector expression and potency are essential for gene therapy development. These assays inform the efficacy, safety, and pharmacodynamic profiles of AAV development candidates. Additionally, for g
Externí odkaz:
https://doaj.org/article/26fd4b183fad4cf4a1ac7b008a0a48b6
Publikováno v:
Antioxidants, Vol 7, Iss 8, p 100 (2018)
Interleukin-1β (IL-1β), a key cytokine that drives neuroinflammation in the Central Nervous System (CNS), is enhanced in many neurological diseases/disorders. Although IL-1β contributes to and/or sustains pathophysiological processes in the CNS, w
Externí odkaz:
https://doaj.org/article/1f6f70bc191b4542b00e91289273a20c
Autor:
Edward Guilmette, Patrick Trapa, Shen Shen, Shih-Ching Lo, Pete Clarner, Shukkwan K. Lau, Twinkle Chowdhury
Publikováno v:
Molecular Therapy. Methods & Clinical Development
Molecular Therapy: Methods & Clinical Development, Vol 23, Iss, Pp 68-77 (2021)
Molecular Therapy: Methods & Clinical Development, Vol 23, Iss, Pp 68-77 (2021)
Robust assays to quantify adeno-associated virus (AAV) vector expression and potency are essential for gene therapy development. These assays inform the efficacy, safety, and pharmacodynamic profiles of AAV development candidates. Additionally, for g
Publikováno v:
Antioxidants, Vol 7, Iss 8, p 100 (2018)
Antioxidants
Volume 7
Issue 8
Antioxidants
Volume 7
Issue 8
Interleukin-1&beta
(IL-1&beta
), a key cytokine that drives neuroinflammation in the Central Nervous System (CNS), is enhanced in many neurological diseases/disorders. Although IL-1&beta
contributes to and/or sustains pathophysiologica
(IL-1&beta
), a key cytokine that drives neuroinflammation in the Central Nervous System (CNS), is enhanced in many neurological diseases/disorders. Although IL-1&beta
contributes to and/or sustains pathophysiologica