Zobrazeno 1 - 10
of 20
pro vyhledávání: '"Tulika Mittal"'
Autor:
Avshesh Mishra, Anshika Srivastava, Surendra Kumar, Tulika Mittal, Naveen Garg, Surendra Kumar Agarwal, Shantanu Pande, Balraj Mittal
Publikováno v:
Indian Heart Journal, Vol 67, Iss 3, Pp 214-221 (2015)
Background: Left ventricular dysfunction (LVD) with subsequent congestive heart failure (CHF) constitutes the final common pathway for a host of cardiac disorders. The impaired LV function develops in response to an ischemic insult followed by a fall
Externí odkaz:
https://doaj.org/article/866f7494bcf744c5b1bc458ba930ea90
Autor:
Saurabh Singh Rathore, Surendra Kumar Agarwal, Shantanu Pande, Sushil Kumar Singh, Tulika Mittal, Balraj Mittal
Publikováno v:
PLoS ONE, Vol 7, Iss 5, p e37844 (2012)
OBJECTIVES: To develop a population specific pharmacogenetic acenocoumarol dosing algorithm for north Indian patients and show its efficiency in dosage prediction. METHODS: Multiple and linear stepwise regression analyses were used to include age, se
Externí odkaz:
https://doaj.org/article/c88d077a97a3437da34db3373ced538e
Autor:
Anshika Srivastava, Naveen Garg, Tulika Mittal, Roopali Khanna, Shipra Gupta, Prahlad Kishore Seth, Balraj Mittal
Publikováno v:
PLoS ONE, Vol 6, Iss 9, p e24123 (2011)
RATIONALE: Mutations in MYBPC3 encoding cardiac myosin binding protein C are common genetic cause of hereditary cardiac myopathies. An intronic 25-bp deletion in MYBPC3 at 3' region is associated with dilated (DCM) and hypertrophic (HCM) cardiomyopat
Externí odkaz:
https://doaj.org/article/363144b524e3461084f88d25dac9f2a5
Autor:
Balraj Mittal, Tulika Mittal, Shantanu Pande, Avshesh Mishra, Surendra K Agarwal, Anshika Srivastava, Surendra Kumar, Naveen Garg
Publikováno v:
Indian Heart Journal, Vol 67, Iss 3, Pp 214-221 (2015)
BackgroundLeft ventricular dysfunction (LVD) with subsequent congestive heart failure (CHF) constitutes the final common pathway for a host of cardiac disorders. The impaired LV function develops in response to an ischemic insult followed by a fall i
Publikováno v:
Gene. 546:309-317
Left ventricular dysfunction (LVD) is a complex, multifactorial condition, caused by mechanical, neurohormonal, and genetic factors. We have previously observed association of renin-angiotensin-aldosterone system (RAAS), matrix metalloproteinases (MM
Autor:
Saurabh Singh Rathore, Surendra Kumar Agarwal, Shantanu Pande, Sushil Kumar Singh, Tulika Mittal, Balraj Mittal
Publikováno v:
Indian Journal of Medical Research, Vol 139, Iss 4, Pp 572-578 (2014)
Background & objectives: CYP4F2 and γ-glutamyl carboxylase (GGCX) have small but significant roles in the maintenance dose of coumarinic oral anticoagulants (COAs). CYP4F2 1347 G > A and GGCX 12970 C > G polymorphisms have been used in the pharmacog
Publikováno v:
DNA and Cell Biology. 30:401-406
MicroRNAs (miRNA) are a class of small noncoding RNA molecules that have been implicated in a wide variety of cellular functions through post-transcriptional regulations on target genes. Common genetic variants (single-nucleotide polymorphisms, SNPs)
Autor:
Saurabh Singh, Rathore, Surendra Kumar, Agarwal, Shantanu, Pande, Sushil Kumar, Singh, Tulika, Mittal, Balraj, Mittal
Publikováno v:
The Indian Journal of Medical Research
Background & objectives: CYP4F2 and γ-glutamyl carboxylase (GGCX) have small but significant roles in the maintenance dose of coumarinic oral anticoagulants (COAs). CYP4F2 1347 G>A and GGCX 12970 C>G polymorphisms have been used in the pharmacogenet
Autor:
Surendra Kumar, Balraj Mittal, Anshika Srivastava, Naveen Garg, Tulika Mittal, Avshesh Mishra
Publikováno v:
Coronary artery disease. 25(1)
BACKGROUND: Coronary artery disease (CAD) is the most common type of heart disease and cause of heart attacks. It has been proposed that both the susceptibility to disease and the interindividual variability in response to treatment relates in part t
Publikováno v:
Cytokine. 61(3)
Rationale Inflammation exacerbates a number of deleterious effects on the heart, most notable being left ventricular dysfunction (LVD). A promoter polymorphism of the NFKB1 gene (encodes p50 subunit) results in lower protein levels of NFkB p50 subuni