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Cells with surface expression of CD133highCD71low are enriched for tripotent colony-forming progenitor cells in the adult murine pancreas

Autor: Jin, Liang, Gao, Dan, Feng, Tao, Tremblay, Jacob R., Ghazalli, Nadiah, Luo, Angela, Rawson, Jeffrey, Quijano, Janine C., Chai, Jing, Wedeken, Lena, Hsu, Jasper, LeBon, Jeanne, Walker, Stephanie, Shih, Hung-Ping, Mahdavi, Alborz, Tirrell, David A., Riggs, Arthur D., Ku, H. Teresa

Publikováno v: In Stem Cell Research January 2016 16(1):40-53

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In Vitro Colony Assays for Characterizing Tri-potent Progenitor Cells Isolated from the Adult Murine Pancreas

Autor: Tremblay, Jacob R., LeBon, Jeanne M., Luo, Angela, Quijano, Janine C., Wedeken, Lena, Jou, Kevin, Riggs, Arthur D., Tirrell, David A., Ku, H. Teresa

Stem and progenitor cells from the adult pancreas could be a potential source of therapeutic beta-like cells for treating patients with type 1 diabetes. However, it is still unknown whether stem and progenitor cells exist in the adult pancreas. Resea

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=od________38::4e07ef3abe4a8d87ff1e0deb8efcc07e
https://resolver.caltech.edu/CaltechAUTHORS:20160705-152413540

Breast cancer-secreted miR-122 reprograms glucose metabolism in pre-metastatic niche to promote metastasis

Autor: Fong, Miranda Y., Zhou, Weiying, Liu, Liang, Alontaga, Aileen Y., Chandra, Manasa, Ashby, Jonathan, Chow, Amy, O’Connor, Sean Francis, Li, Shasha, Chin, Andrew R., Somlo, George, Palomares, Melanie, Li, Zhuo, Tremblay, Jacob R., Tsuyada, Akihiro, Sun, Guoqiang, Reid, Michael A., Wu, Xiwei, Swiderski, Piotr, Ren, Xiubao, Shi, Yanhong, Kong, Mei, Zhong, Wenwan, Chen, Yuan, Wang, Shizhen Emily

Publikováno v: Fong, Miranda Y.; Zhou, Weiying; Liu, Liang; Alontaga, Aileen Y.; Chandra, Manasa; Ashby, Jonathan; et al.(2015). Breast cancer-secreted miR-122 reprograms glucose metabolism in pre-metastatic niche to promote metastasis. Nature cell biology, 17(2), 183-194. UC Office of the President: California Breast Cancer Research Program. Retrieved from: http://www.escholarship.org/uc/item/3sg6m9qp

Reprogrammed glucose metabolism as a result of increased glycolysis and glucose uptake is a hallmark of cancer. Here we show that cancer cells can suppress glucose uptake by non-tumour cells in the pre-metastatic niche, by secreting vesicles that car

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=od_______325::b892df65f80624d7c9d154966b7aabd6
http://www.escholarship.org/uc/item/3sg6m9qp