Zobrazeno 1 - 10
of 80
pro vyhledávání: '"Toru Yago"'
Autor:
Yuya Fujita, Toru Yago, Haruki Matsumoto, Tomoyuki Asano, Naoki Matsuoka, Jumpei Temmoku, Shuzo Sato, Makiko Yashiro-Furuya, Eiji Suzuki, Hiroshi Watanabe, Atsushi Kawakami, Kiyoshi Migita
Publikováno v:
Arthritis Research & Therapy, Vol 23, Iss 1, Pp 1-9 (2021)
Abstract Background Gout is an autoinflammatory disease driven by interleukin-1 (IL-1) induction in response to uric acid crystals. IL-1β production is dependent on inflammasome activation, which requires a priming signal, followed by an activating
Externí odkaz:
https://doaj.org/article/ad9fca7d9beb4f92820865628f269011
Autor:
Yuya Fujita, Toru Yago, Tomoyuki Asano, Haruki Matsumoto, Naoki Matsuoka, Jumpei Temmoku, Shuzo Sato, Makiko Yashiro-Furuya, Eiji Suzuki, Hiroshi Watanabe, Atsushi Kawakami, Kiyoshi Migita
Publikováno v:
PLoS ONE, Vol 16, Iss 8 (2021)
Background Adult-onset Still’s disease (AOSD) is a systemic autoinflammatory disease in which danger-associated molecular patterns (DAMPs)-mediated inflammasome activation seems to be involved in the disease pathogenesis. Cold-inducible RNA-binding
Externí odkaz:
https://doaj.org/article/c8dd3996888f43329b1587110a58a8d8
Autor:
Haruki Matsumoto, Yuya Fujita, Tomoyuki Asano, Naoki Matsuoka, Jumpei Temmoku, Shuzo Sato, Makiko Yashiro–Furuya, Kohei Yokose, Shuhei Yoshida, Eiji Suzuki, Toru Yago, Hiroshi Watanabe, Atsushi Kawakami, Kiyoshi Migita
Publikováno v:
PLoS ONE, Vol 16, Iss 11 (2021)
Background Anti-citrullinated peptide antibodies (ACPA) and inflammatory cytokines play important roles in the development of rheumatoid arthritis (RA). T cell immunoglobulin and mucin–domain containing–3 (TIM–3) is an immune-checkpoint molecul
Externí odkaz:
https://doaj.org/article/5146651f887140efad76d4db9c829101
Publikováno v:
Pharmaceuticals, Vol 3, Iss 5, Pp 1394-1410 (2010)
Osteoclasts play a critical role in both normal bone metabolism and bone resorption in the joints of patients with rheumatoid arthritis. It has been reported that non-steroidal anti-inflammatory drugs (NSAIDs) inhibit murine osteoclastogenesis in vit
Externí odkaz:
https://doaj.org/article/4796d7a15d6d4892b0e3881d16c8ca4b
Autor:
Toru Yago, Yuki Nanke, Manabu Kawamoto, Tsuyoshi Kobashigawa, Hisashi Yamanaka, Shigeru Kotake
Publikováno v:
Journal of Clinical Medicine, Vol 6, Iss 9, p 81 (2017)
IL-23, which is composed of p19 and p40 subunits, is a proinflammatory cytokine that contributes to the formation and maintenance of Th17 cells in inflammatory autoimmune diseases. IL-23 is a human osteoclastogenic cytokine and anti-IL-23 antibody at
Externí odkaz:
https://doaj.org/article/e16817c576834880b1c551fffc953a44
Publikováno v:
Journal of Clinical Medicine, Vol 6, Iss 7, p 74 (2017)
Behcet’s disease (BD) is a polysymptomatic and recurrent systemic vasculitis with a chronic course and unknown cause. The pathogenesis of BD has not been fully elucidated; however, BD has been considered to be a typical Th1-mediated inflammatory di
Externí odkaz:
https://doaj.org/article/bc058d7d2a16491b9e84d32198442d8f
Publikováno v:
Journal of Clinical Medicine, Vol 6, Iss 7, p 67 (2017)
Helper T (Th) cells play an important role in the pathogenesis of autoimmune diseases, including rheumatoid arthritis (RA). It has been revealed that Th17 cells can shift to Th1 cells (i.e., “nonclassic Th1 cells”), which are reported to be more
Externí odkaz:
https://doaj.org/article/7116cd36330948b1951669bbcb9a7a37
Autor:
Kenji Saito, Jumpei Temmoku, Yuya Sumichika, Shuhei Yoshida, Haruki Matsumoto, Yuya Fujita, Naoki Matsuoka, Tomoyuki Asano, Toru Yago, Shuzo Sato, Kiyoshi Migita
Publikováno v:
Internal Medicine; 2024, Vol. 63 Issue 10, p1473-1478, 6p
Autor:
Haruki Matsumoto, Hironori Ohashi, Yuya Fujita, Shuhei Yoshida, Kohei Yokose, Jumpei Temmoku, Naoki Matsuoka, Yumetaka Shinden, Keigo Kusano, Tatsuru Sonobe, Yohei Nakamoto, Makiko Yashiro-Furuya, Tomoyuki Asano, Shuzo Sato, Eiji Suzuki, Toru Yago, Hiroshi Watanabe, Kiyoshi Migita
Publikováno v:
The Tohoku Journal of Experimental Medicine. 256:169-174
Familial Mediterranean fever (FMF) is a hereditary autoinflammatory disease characterized by recurrent episodes of fever and serositis. Periodic febrile attack can be managed with biologic medication in colchicine-resistant FMF patients, however, no
Autor:
Shuhei Yoshida, Haruki Matsumoto, Yuya Fujita, Kohei Yokose, Jumpei Temmoku, Naoki Matsuoka, Makiko Yashiro-Furuya, Tomoyuki Asano, Shuzo Sato, Eiji Suzuki, Toru Yago, Takae Yaguchi, Tetsuro Aita, Misaki Kusano, Toshiyuki Yamamoto, Hiroshi Watanabe, Kiyoshi Migita
Publikováno v:
The Tohoku Journal of Experimental Medicine. 256:303-308