Zobrazeno 1 - 10
of 19
pro vyhledávání: '"Torben, Ørntoft"'
Autor:
Nkerorema Djodji Damas, Michela Marcatti, Christophe Côme, Lise Lotte Christensen, Morten Muhlig Nielsen, Roland Baumgartner, Helene Maria Gylling, Giulia Maglieri, Carsten Friis Rundsten, Stefan E. Seemann, Nicolas Rapin, Simon Thézenas, Søren Vang, Torben Ørntoft, Claus Lindbjerg Andersen, Jakob Skou Pedersen, Anders H. Lund
Publikováno v:
Nature Communications, Vol 7, Iss 1, Pp 1-14 (2016)
Several lncRNAs have been linked to cancer. Here, the authors identify SNHG5 as a long non-coding RNA promoting proliferation and survival of colorectal cancer cells by protecting specific mRNAs from STAU1-mediated degradation.
Externí odkaz:
https://doaj.org/article/b798c3c5b5f543a9b6ab76d138075453
Supplementary Figure 2 from Retinoblastoma Pathway Defects Show Differential Ability to Activate the Constitutive DNA Damage Response in Human Tumorigenesis
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a467068587b1aafce0a553d1d80ba5d0
https://doi.org/10.1158/0008-5472.22366514
https://doi.org/10.1158/0008-5472.22366514
Autor:
Torben Ørntoft, Lars Dyrskjøt, Klaus Møller-Ernst Jensen, Guido Sauter, Thomas Thykjaer, Flemming Brandt Sørensen, Carsten Wiuf, Karin Birkenkamp-Demtroder, Mads Aaboe
The human transcription factor SOX4 was 5-fold up-regulated in bladder tumors compared with normal tissue based on whole-genome expression profiling of 166 clinical bladder tumor samples and 27 normal urothelium samples. Using a SOX4-specific antibod
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5d3edfda062bc64d1f24514d3e1ce0da
https://doi.org/10.1158/0008-5472.c.6495374.v1
https://doi.org/10.1158/0008-5472.c.6495374.v1
Supplementary Figure Legends 1-4 from Retinoblastoma Pathway Defects Show Differential Ability to Activate the Constitutive DNA Damage Response in Human Tumorigenesis
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::76fa04bc5b85436c16c255b25987b8c1
https://doi.org/10.1158/0008-5472.22366505
https://doi.org/10.1158/0008-5472.22366505
Supplementary Figure 1 from Retinoblastoma Pathway Defects Show Differential Ability to Activate the Constitutive DNA Damage Response in Human Tumorigenesis
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0bb67dd68eea2b407c326e2e0092f4a7
https://doi.org/10.1158/0008-5472.22366517.v1
https://doi.org/10.1158/0008-5472.22366517.v1
Autor:
Torben Ørntoft, Lars Dyrskjøt, Klaus Møller-Ernst Jensen, Guido Sauter, Thomas Thykjaer, Flemming Brandt Sørensen, Carsten Wiuf, Karin Birkenkamp-Demtroder, Mads Aaboe
In addition to the link to the pdf supplementary information, microarray data for this article is available at: www.mdl.dk -OR- www.ncbi.nlm.nih.gov/geo/
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::00809ee02b8a0b53beb5854874141c2c
https://doi.org/10.1158/0008-5472.22366994.v1
https://doi.org/10.1158/0008-5472.22366994.v1
Loss of G1-S control and aberrations of the p16Ink4a-cyclin D1/cyclin-dependent kinase (CDK) 4(6)-pRb-E2F-cyclin E/CDK2 pathway are common in human cancer. Previous studies showed that oncogene-induced aberrant proliferation, such as on cyclin E over
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1f60f5ad06bbba9b421acad23b00b522
https://doi.org/10.1158/0008-5472.c.6495170.v1
https://doi.org/10.1158/0008-5472.c.6495170.v1
Supplementary Figure 3 from Retinoblastoma Pathway Defects Show Differential Ability to Activate the Constitutive DNA Damage Response in Human Tumorigenesis
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::604b09c14ee39f8eed435af287d28c7d
https://doi.org/10.1158/0008-5472.22366511
https://doi.org/10.1158/0008-5472.22366511
Supplementary Figure 4 from Retinoblastoma Pathway Defects Show Differential Ability to Activate the Constitutive DNA Damage Response in Human Tumorigenesis
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::40d9f9022a1d5ce7c762d8b710027dff
https://doi.org/10.1158/0008-5472.22366508.v1
https://doi.org/10.1158/0008-5472.22366508.v1
Autor:
Marianne T, Bjerre, Siri H, Strand, Maibritt, Nørgaard, Helle, Kristensen, Anne Ki, Rasmussen, Martin Mørck, Mortensen, Jacob, Fredsøe, Peter, Mouritzen, Benedicte, Ulhøi, Torben, Ørntoft, Michael, Borre, Karina D, Sørensen
Publikováno v:
International Journal of Molecular Sciences
Prostate cancer (PCa) is a clinically heterogeneous disease and currently, accurate diagnostic and prognostic molecular biomarkers are lacking. This study aimed to identify novel DNA hypermethylation markers for PCa with future potential for blood-ba
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::b56944a7c1401013330613394bf787b7
https://pubmed.ncbi.nlm.nih.gov/30866497
https://pubmed.ncbi.nlm.nih.gov/30866497