Zobrazeno 1 - 10
of 12
pro vyhledávání: '"Tomoyuki Matsusaka"'
Autor:
Tomoyuki Matsusaka, Takayuki Fujino, Kenjiro Kikuchi, Naoyuki Hasebe, Masahiko Morihira, Erdenechimeg Baljinnyam, Jun Fukuzawa, Kazuhiro Sumitomo, Kazuma Izawa
Publikováno v:
American Journal of Physiology-Heart and Circulatory Physiology. 290:H577-H583
Reactive oxygen species (ROS) enhance myocardial ischemia-reperfusion (I/R) injury. Ischemic preconditioning (PC) provides potent cardioprotective effects in I/R. However, it has not been elucidated whether PC diminishes ROS stress in I/R and whether
Autor:
Naoyuki Hasebe, Hironobu Matsuhashi, Shunsuke Natori, Kenjiro Kikuchi, Tadashi Ihara, Yin-Tie Jin, Akira Ido, Tomoyuki Matsusaka
Publikováno v:
American Journal of Respiratory and Critical Care Medicine. 167:895-901
Nitric oxide (NO) inhalation therapy has been widely used in several diseases with pulmonary hypertension. However, application of NO inhalation therapy remains controversial in heart failure. Cardiovascular effects of inhaled NO (iNO) were evaluated
Autor:
Masahiko Morihira, Tomoyuki Matsusaka, Hideki Nakamura, Kenjiro Kikuchi, Shunsuke Natori, Takafumi Ohta, Hironobu Matsuhashi, Akira Ido, Yin-Tie Jin, Naoyuki Hasebe, Shiro Tsuji
Publikováno v:
Journal of Cardiovascular Pharmacology. 36:S55-S60
We examined the dose responses to continuous infusion of isoproterenol (ISO) and norepinephrine (NE) in normal (control) and procainamide-induced heart failure dogs with or without inhalation of 70 ppm nitric oxide (NO). Inhaled NO affected neither l
Autor:
Masahiko Morihira, Erdenechimeg Baljinnyam, Jun Fukuzawa, Kazuhiro Sumitomo, Naoyuki Hasebe, Tomoyuki Matsusaka, Takayuki Fujino, Fumitaka Ushikubi, Kenjiro Kikuchi
Publikováno v:
Hypertension research : official journal of the Japanese Society of Hypertension. 29(11)
Reactive oxygen species (ROS) enhance myocardial ischemia-reperfusion (I/R) injury. Ebselen, a seleno-organic glutathione peroxidase (GPx) mimetic, has a protective effect against tissue injury induced by ROS. However, the cardio-protective effect of
Autor:
Kenjiro Kikuchi, Tomoyuki Matsusaka, Takafumi Ohta, Naoyuki Hasebe, Shiro Tsuji, Yin-Tie Jin, Shunsuke Natori
Publikováno v:
American journal of physiology. Heart and circulatory physiology. 292(3)
Sympathetic nervous activation is a crucial compensatory mechanism in heart failure. However, excess catecholamine may induce cardiac dysfunction and β-adrenergic desensitization. Although magnesium is known to be a cardioprotective agent, its benef
Publikováno v:
Cardiovascular research. 54(3)
Objectives: Clinical impact of magnesium (Mg) therapy remains controversial in acute myocardial infarction. We investigated the infarct size limiting effects of Mg and its mechanism in rabbits. Methods: Anesthetized rabbits underwent 30 min coronary
Autor:
Masahiko Morihira, Hironobu Matsuhashi, Yin-Tie Jin, Tomoyuki Matsusaka, Shunsuke Natori, Naoyuki Hasebe, Shiro Tsuji, Takafumi Ohta, Hideki Nakamura, Kenjiro Kikuchi
Publikováno v:
Journal of Cardiac Failure. 5:68
Autor:
Jun-ichi Kawabe, Shunsuke Natori, Tomoyuki Matsusaka, Naoyuki Hasebe, Yin-Tie Jin, Kenjiro Kikuchi
Publikováno v:
Journal of Cardiac Failure. 4:71
Autor:
Shunsuke Natori, Hironobu Matsuhashi, Naoyuki Hasebe, Tomoyuki Matsusaka, Kenjiro Kikuchi, Akira Ido, Takafumi Ohta, Hideki Nakamura M.D., Yin-Tie Jin, Masahiko Morihira
Publikováno v:
Journal of Cardiac Failure. 4:69
Autor:
Shunsuke Natori, Naoyuki Hasebe, Yin-Tie Jin, Tomoyuki Matsusaka, Akira Ido, Hironobu Matsuhashi, Tadashi Ihara, Kenjiro Kikuchi
Publikováno v:
American Journal of Respiratory & Critical Care Medicine; 2003, Vol. 167 Issue 6, p895-901, 7p