Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Tomoko Katono"'
Autor:
Masao Maeno, Takayuki Kawato, Kumiko Nakai, Tomoko Katono-Tani, Sakurako Iwata, Ning Zhao, Akemi Kimura
Publikováno v:
Journal of Hard Tissue Biology. 21:231-236
Autor:
Fan Zhang, Hideki Tanaka, Natsuko Tanabe, Yoshiyuki Yonehara, Satoshi Kitami, Masao Maeno, Takayuki Kawato, Naoto Suzuki, Tomoko Katono-Tani
Publikováno v:
Biochimie. 92:398-404
Interleukin-17 (IL-17) is produced exclusively by activated T cells and neutrophils, and stimulates osteoclastic bone resorption via osteoblasts by inducing the expression of "receptor activator of NF-kappaB (RANK) ligand" (RANKL). However, the direc
Publikováno v:
Oral Microbiology and Immunology. 23:14-20
Background/aims: The cell surface protein antigen (PAg) and glucosyltransferases (GTFs) produced by Streptococcus sobrinus are considered to be major colonization factors of the organism. Methods: We constructed a fusion gene encoding a protein compo
Autor:
Maiko Shoji, Osamu Takeichi, Masao Maeno, Akira Morozumi, Tomoko Katono, Natsuko Tanabe, Naoto Suzuki, Narihiro Mitsui
Publikováno v:
Acta Biochimica et Biophysica Sinica. 39:163-172
Previous studies have indicated that lipopolysaccharide (LPS) from Gram-negative bacteria in plaque induces the release of prostaglandin E2 (PGE2), which promotes alveolar bone resorption in periodontitis, and that tobacco smoking might be an importa
Autor:
Tomoko Katono, Masao Maeno, Naoto Suzuki, Hitoshi Oka, Natsuko Tanabe, Kazuhiro Yamanaka, Masafumi Motohashi, Takayuki Kawato
Publikováno v:
Acta Biochimica et Biophysica Sinica. 38:874-882
Tobacco smoking is an important risk factor for the development of severe periodontitis. Recently, we showed that nicotine affected mineralized nodule formation, and that nicotine and lipopolysaccharide stimulated the formation of osteoclast-like cel
Autor:
Masafumi Motohashi, Maiko Shoji, Natsuko Tanabe, Hideki Tanaka, Tomoko Katono, Setsuko Sato, Masao Maeno, Naoto Suzuki
Publikováno v:
Life Sciences. 78:1733-1740
Several studies have indicated that one of the causes of alveolar bone destruction with periodontitis is lipopolysaccharide (LPS) from the cell wall of Gram-negative bacteria in plaque and that tobacco smoking may be an important risk factor for the
Autor:
Maiko, Shoji, Natsuko, Tanabe, Narihiro, Mitsui, Naoto, Suzuki, Osamu, Takeichi, Tomoko, Katono, Akira, Morozumi, Masao, Maeno
Publikováno v:
Acta biochimica et biophysica Sinica. 39(3)
Previous studies have indicated that lipopolysaccharide (LPS) from Gram-negative bacteria in plaque induces the release of prostaglandin E(2) (PGE(2)), which promotes alveolar bone resorption in periodontitis, and that tobacco smoking might be an imp