Zobrazeno 1 - 10
of 13
pro vyhledávání: '"Tomasz J. Slebioda"'
Autor:
Tomasz J. Ślebioda, Marcin Stanisławowski, Lucyna Kaszubowska, Jan M. Zaucha, Michał A. Żmijewski
Publikováno v:
Biomedicines, Vol 10, Iss 11, p 2912 (2022)
Chimeric antigen receptor T (CAR-T) cells are genetically modified autologous T cells that have revolutionized the treatment of relapsing and refractory haematological malignancies. In this review we present molecular pathways involved in the activat
Externí odkaz:
https://doaj.org/article/f1b29ecfbd5d46f984d7f67d37b311e9
Autor:
Tomasz J. Ślebioda, Zbigniew Kmieć
Publikováno v:
Mediators of Inflammation, Vol 2014 (2014)
Externí odkaz:
https://doaj.org/article/964ecc41afdd44a08215fd1e0601b7c7
Autor:
Dorota Zurawa-Janicka, Jarek Kobiela, Marcin Stanisławowski, Andrzej J. Lachinski, Tomasz J. Slebioda, Zbigniew Sledzinski, Tomasz Wenta, Wojciech Biernat, Piotr M. Wierzbicki, Rafał Pęksa, Zbigniew Kmieć, Barbara Lipinska
Publikováno v:
International Journal of Molecular Sciences, Vol 21, Iss 3947, p 3947 (2020)
International Journal of Molecular Sciences
Volume 21
Issue 11
International Journal of Molecular Sciences
Volume 21
Issue 11
HtrA proteases regulate cellular homeostasis and cell death. Their dysfunctions have been correlated with oncogenesis and response to therapeutic treatment. We investigated the relation between HtrA1-3 expression and clinicopathological, and survival
Autor:
Krzysztof Celiński, Piotr Landowski, Barbara Kamińska, Tomasz J. Slebioda, Marta Cyman, Agnieszka Bojarska-Junak, Zbigniew Kmieć
Publikováno v:
Cytometry Part B: Clinical Cytometry. 92:165-169
Background Interaction between TL1A and death receptor 3 (DR3) is associated with the pathogenesis of inflammatory bowel disease (IBD), although their role in the development of this disease remains not fully explained. Some studies showed elevated e
Autor:
Sonya James, Aymen Al-Shamkhani, Tomasz J. Slebioda, Adam F. Cunningham, Vadim Y. Taraban, Sarah L. Buchan
Publikováno v:
European Journal of Immunology. 42:580-588
The TNF receptor superfamily member death receptor 3 (DR3) exacerbates Th2- and Th17-cell-mediated inflammatory and autoimmune conditions, yet no role in host defence has been reported. Here, we examined the role of DR3 during infection with Salmonel
Autor:
Jane E. Willoughby, Aymen Al-Shamkhani, Tomasz J. Slebioda, Vadim Y. Taraban, Tania F. Rowley, Sarah L. Buchan, John R. Ferdinand
Publikováno v:
European Journal of Immunology. 41:2606-2611
TNFRSF25 is a member of the TNF receptor superfamily (TNFRSF) that binds to the TNF-like protein TL1A. Although recent studies have demonstrated a role for TNFRSF25 in regulating CD4(+) T-cell responses, it remains to be determined if TNFRSF25 functi
Autor:
Edward Chung Yern Wang, Tomasz J. Slebioda, Vadim Y. Taraban, Aymen Al-Shamkhani, Sarah L. Buchan, Neil Smyth, Sonya James, Jane E. Willoughby, Gareth J. Thomas, Bhavwanti Sheth
Publikováno v:
Europe PubMed Central
The tumor necrosis factor (TNF) superfamily protein TNF-like 1A (TL1A) is the ligand for death receptor 3 (DR3). TL1A is induced on activated dendritic cells (DCs) and its expression has been linked to human inflammatory bowel disease. To address how
Autor:
Tomasz J, Slebioda, Agnieszka, Bojarska-Junak, Marta, Cyman, Piotr, Landowski, Barbara, Kaminska, Krzysztof, Celinski, Zbigniew, Kmiec
Publikováno v:
Cytometry. Part B, Clinical cytometry. 92(2)
Interaction between TL1A and death receptor 3 (DR3) is associated with the pathogenesis of inflammatory bowel disease (IBD), although their role in the development of this disease remains not fully explained. Some studies showed elevated expression o
Autor:
Marcin Stanisławowski, Marcin Matuszewski, Agata Wronska, Piotr M. Wierzbicki, Agata Olejniczak, Zbigniew Kmieć, Jakub Klacz, Agnieszka Rybarczyk, Tomasz J. Slebioda
Publikováno v:
International Journal of Oncology
Clear-cell renal cell carcinoma (ccRCC) is the most common subtype of RCC (70–80%) and is associated with poor prognosis in 40% of cases mainly due to metastasis in the course of the disease. RASSF1, with its isoforms RASSF1A and RASSF1C, is a tumo
Autor:
Sarah L. Buchan, Vadim Y. Taraban, Tomasz J. Slebioda, Sonya James, Adam F. Cunningham, Aymen Al-Shamkhani
Publikováno v:
European Journal of Immunology.