Zobrazeno 1 - 10 of 1 109 pro vyhledávání: '"Todiras M"'
1
Akademický článek
Angiotensin deficient FVB/N mice are normotensive.
Autor: Rodrigues AF; Max-Delbrück-Center for Molecular Medicine in the Helmholtz Association, Berlin, Germany.; Department of Biology, Chemistry and Pharmacy, Free University of Berlin, Berlin, Germany.; German Center for Cardiovascular Research (DZHK), Partner Site Berlin, Berlin, Germany., Todiras M; Max-Delbrück-Center for Molecular Medicine in the Helmholtz Association, Berlin, Germany.; Nicolae Testemițanu State University of Medicine and Pharmacy, Chisinau, Moldova., Qadri F; Max-Delbrück-Center for Molecular Medicine in the Helmholtz Association, Berlin, Germany., Alenina N; Max-Delbrück-Center for Molecular Medicine in the Helmholtz Association, Berlin, Germany.; German Center for Cardiovascular Research (DZHK), Partner Site Berlin, Berlin, Germany., Bader M; Max-Delbrück-Center for Molecular Medicine in the Helmholtz Association, Berlin, Germany.; German Center for Cardiovascular Research (DZHK), Partner Site Berlin, Berlin, Germany.; Charité Universitätsmedizin Berlin, Berlin, Germany.; Institute for Biology, University of Lübeck, Lübeck, Germany.
Publikováno v: British journal of pharmacology [Br J Pharmacol] 2023 Jul; Vol. 180 (14), pp. 1843-1861. Date of Electronic Publication: 2023 Feb 22.
Zobrazit plný text záznamu
2
Inhibition of the NLRP3/IL-1β axis protects against sepsis-induced cardiomyopathy
Autor: Busch, K., Kny, M., Huang, N., Klassert, T.E., Stock, M., Hahn, A., Graeger, S., Todiras, M., Schmidt, S., Chamling, B., Willenbrock, M., Groß, S., Biedenweg, D., Heuser, A., Scheidereit, C., Butter, C., Felix, S.B., Otto, O., Luft, F.C., Slevogt, H., Fielitz, J.
BACKGROUND: Septic cardiomyopathy worsens the prognosis of critically ill patients. Clinical data suggest that interleukin-1β (IL-1β), activated by the NLRP3 inflammasome, compromises cardiac function. Whether or not deleting Nlrp3 would prevent ca
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=mdc______med::c88bcab0c4befc738bf93eccfac9fe94
http://edoc.mdc-berlin.de/20902/7/20902suppl.zip
Zobrazit plný text záznamu
3
Akademický článek
G-protein-coupled receptor MAS deletion produces a preeclampsia-like phenotype in FVB/N mice.
Autor: Vieira Pimentel RL; Department of Physiology and Biophysics, National Institute of Science and Technology in Nanobiopharmaceutics, Federal University of Minas Gerais, Belo Horizonte, MG, Brazil., Braga JF; Department of Physiology and Biophysics, National Institute of Science and Technology in Nanobiopharmaceutics, Federal University of Minas Gerais, Belo Horizonte, MG, Brazil., Velloso EPP; Department of Physiology and Biophysics, National Institute of Science and Technology in Nanobiopharmaceutics, Federal University of Minas Gerais, Belo Horizonte, MG, Brazil., Lautner RQ; Department of Physiology and Biophysics, National Institute of Science and Technology in Nanobiopharmaceutics, Federal University of Minas Gerais, Belo Horizonte, MG, Brazil., de Oliveira ML; Department of Physiology and Biophysics, National Institute of Science and Technology in Nanobiopharmaceutics, Federal University of Minas Gerais, Belo Horizonte, MG, Brazil., Todiras M; Max-Delbrück-Center for Molecular Medicine (MDC), Berlin-Buch, Germany., Alenina N; Department of Physiology and Biophysics, National Institute of Science and Technology in Nanobiopharmaceutics, Federal University of Minas Gerais, Belo Horizonte, MG, Brazil.; Max-Delbrück-Center for Molecular Medicine (MDC), Berlin-Buch, Germany.; German Center for Cardiovascular Research (DZHK) site Berlin, Berlin, Germany., Bader M; Department of Physiology and Biophysics, National Institute of Science and Technology in Nanobiopharmaceutics, Federal University of Minas Gerais, Belo Horizonte, MG, Brazil.; Max-Delbrück-Center for Molecular Medicine (MDC), Berlin-Buch, Germany.; German Center for Cardiovascular Research (DZHK) site Berlin, Berlin, Germany.; Charité University Medicine Berlin, Germany.; Institute for Biology, University of Lübeck, Lübeck, Germany., de Sousa FG; Department of Physiology and Biophysics, National Institute of Science and Technology in Nanobiopharmaceutics, Federal University of Minas Gerais, Belo Horizonte, MG, Brazil., Beier SL; Department of Physiology and Biophysics, National Institute of Science and Technology in Nanobiopharmaceutics, Federal University of Minas Gerais, Belo Horizonte, MG, Brazil., Santos R; Department of Physiology and Biophysics, National Institute of Science and Technology in Nanobiopharmaceutics, Federal University of Minas Gerais, Belo Horizonte, MG, Brazil.
Publikováno v: Clinical science (London, England : 1979) [Clin Sci (Lond)] 2023 Aug 31; Vol. 137 (16), pp. 1249-1263.
Zobrazit plný text záznamu
4
Akademický článek
Quantifying the impact of gut microbiota on inflammation and hypertensive organ damage.
Autor: Avery EG; Experimental and Clinical Research Center, A Cooperation of Charité-Universitätsmedizin Berlin and Max-Delbrück-Center for Molecular Medicine, Berlin, Germany.; Max-Delbrück-Center for Molecular Medicine in the Helmholtz Association, Berlin, Germany.; DZHK (German Centre for Cardiovascular Research), partner site Berlin, Germany.; Department of Biology, Chemistry, and Pharmacy, Freie Universität Berlin, Berlin, Germany., Bartolomaeus H; Experimental and Clinical Research Center, A Cooperation of Charité-Universitätsmedizin Berlin and Max-Delbrück-Center for Molecular Medicine, Berlin, Germany.; Max-Delbrück-Center for Molecular Medicine in the Helmholtz Association, Berlin, Germany.; DZHK (German Centre for Cardiovascular Research), partner site Berlin, Germany.; Department of Nephrology and Internal Intensive Care Medicine, Charité-Universitätsmedizin Berlin, Corporate member of Freie Universität Berlin and Humboldt-Universität zu Berlin, Berlin, Germany., Rauch A; Experimental and Clinical Research Center, A Cooperation of Charité-Universitätsmedizin Berlin and Max-Delbrück-Center for Molecular Medicine, Berlin, Germany.; Max-Delbrück-Center for Molecular Medicine in the Helmholtz Association, Berlin, Germany.; DZHK (German Centre for Cardiovascular Research), partner site Berlin, Germany.; Department of Nephrology and Internal Intensive Care Medicine, Charité-Universitätsmedizin Berlin, Corporate member of Freie Universität Berlin and Humboldt-Universität zu Berlin, Berlin, Germany., Chen CY; Experimental and Clinical Research Center, A Cooperation of Charité-Universitätsmedizin Berlin and Max-Delbrück-Center for Molecular Medicine, Berlin, Germany.; Max-Delbrück-Center for Molecular Medicine in the Helmholtz Association, Berlin, Germany.; DZHK (German Centre for Cardiovascular Research), partner site Berlin, Germany.; Charité-Universitätsmedizin Berlin, Corporate member of Freie Universität Berlin and Humboldt-Universität zu Berlin, Berlin, Germany., N'Diaye G; Experimental and Clinical Research Center, A Cooperation of Charité-Universitätsmedizin Berlin and Max-Delbrück-Center for Molecular Medicine, Berlin, Germany.; Max-Delbrück-Center for Molecular Medicine in the Helmholtz Association, Berlin, Germany.; Charité-Universitätsmedizin Berlin, Corporate member of Freie Universität Berlin and Humboldt-Universität zu Berlin, Berlin, Germany., Löber U; Experimental and Clinical Research Center, A Cooperation of Charité-Universitätsmedizin Berlin and Max-Delbrück-Center for Molecular Medicine, Berlin, Germany.; Max-Delbrück-Center for Molecular Medicine in the Helmholtz Association, Berlin, Germany.; DZHK (German Centre for Cardiovascular Research), partner site Berlin, Germany., Bartolomaeus TUP; Experimental and Clinical Research Center, A Cooperation of Charité-Universitätsmedizin Berlin and Max-Delbrück-Center for Molecular Medicine, Berlin, Germany.; Max-Delbrück-Center for Molecular Medicine in the Helmholtz Association, Berlin, Germany.; DZHK (German Centre for Cardiovascular Research), partner site Berlin, Germany.; Charité-Universitätsmedizin Berlin, Corporate member of Freie Universität Berlin and Humboldt-Universität zu Berlin, Berlin, Germany., Fritsche-Guenther R; Max-Delbrück-Center for Molecular Medicine in the Helmholtz Association, Berlin, Germany.; Metabolomics Platform, Berlin Institute of Health at Charité-Universitätsmedizin Berlin, Berlin, Germany., Rodrigues AF; Max-Delbrück-Center for Molecular Medicine in the Helmholtz Association, Berlin, Germany.; DZHK (German Centre for Cardiovascular Research), partner site Berlin, Germany.; Department of Biology, Chemistry, and Pharmacy, Freie Universität Berlin, Berlin, Germany., Yarritu A; Experimental and Clinical Research Center, A Cooperation of Charité-Universitätsmedizin Berlin and Max-Delbrück-Center for Molecular Medicine, Berlin, Germany.; Max-Delbrück-Center for Molecular Medicine in the Helmholtz Association, Berlin, Germany.; DZHK (German Centre for Cardiovascular Research), partner site Berlin, Germany.; Department of Nephrology and Internal Intensive Care Medicine, Charité-Universitätsmedizin Berlin, Corporate member of Freie Universität Berlin and Humboldt-Universität zu Berlin, Berlin, Germany., Zhong C; Institute of Translational Physiology, Charité-Universitätsmedizin Berlin, Corporate Member of Freie Universität Berlin and Humboldt-Universität zu Berlin, Charitéplatz 1, 10117 Berlin, Germany., Fei L; Institute of Translational Physiology, Charité-Universitätsmedizin Berlin, Corporate Member of Freie Universität Berlin and Humboldt-Universität zu Berlin, Charitéplatz 1, 10117 Berlin, Germany., Tsvetkov D; Experimental and Clinical Research Center, A Cooperation of Charité-Universitätsmedizin Berlin and Max-Delbrück-Center for Molecular Medicine, Berlin, Germany.; DZHK (German Centre for Cardiovascular Research), partner site Berlin, Germany.; Charité-Universitätsmedizin Berlin, Corporate member of Freie Universität Berlin and Humboldt-Universität zu Berlin, Berlin, Germany.; Department of Geriatrics, University of Greifswald, University District Hospital Wolgast, Greifswald, Germany., Todiras M; Max-Delbrück-Center for Molecular Medicine in the Helmholtz Association, Berlin, Germany.; Nicolae Testemianu State University of Medicine and Pharmacy, Chisinau, Moldova., Park JK; Hannover Medical School, Hannover, Germany., Markó L; Experimental and Clinical Research Center, A Cooperation of Charité-Universitätsmedizin Berlin and Max-Delbrück-Center for Molecular Medicine, Berlin, Germany.; Max-Delbrück-Center for Molecular Medicine in the Helmholtz Association, Berlin, Germany.; DZHK (German Centre for Cardiovascular Research), partner site Berlin, Germany.; Charité-Universitätsmedizin Berlin, Corporate member of Freie Universität Berlin and Humboldt-Universität zu Berlin, Berlin, Germany., Maifeld A; Experimental and Clinical Research Center, A Cooperation of Charité-Universitätsmedizin Berlin and Max-Delbrück-Center for Molecular Medicine, Berlin, Germany.; Max-Delbrück-Center for Molecular Medicine in the Helmholtz Association, Berlin, Germany.; DZHK (German Centre for Cardiovascular Research), partner site Berlin, Germany.; Charité-Universitätsmedizin Berlin, Corporate member of Freie Universität Berlin and Humboldt-Universität zu Berlin, Berlin, Germany., Patzak A; Institute of Translational Physiology, Charité-Universitätsmedizin Berlin, Corporate Member of Freie Universität Berlin and Humboldt-Universität zu Berlin, Charitéplatz 1, 10117 Berlin, Germany., Bader M; Max-Delbrück-Center for Molecular Medicine in the Helmholtz Association, Berlin, Germany.; DZHK (German Centre for Cardiovascular Research), partner site Berlin, Germany.; Charité-Universitätsmedizin Berlin, Corporate member of Freie Universität Berlin and Humboldt-Universität zu Berlin, Berlin, Germany., Kempa S; Max-Delbrück-Center for Molecular Medicine in the Helmholtz Association, Berlin, Germany.; Integrative Proteomics and Metabolomics Platform, Berlin Institute for Medical Systems Biology BIMSB, Berlin, Germany., Kirwan JA; Max-Delbrück-Center for Molecular Medicine in the Helmholtz Association, Berlin, Germany.; Metabolomics Platform, Berlin Institute of Health at Charité-Universitätsmedizin Berlin, Berlin, Germany., Forslund SK; Experimental and Clinical Research Center, A Cooperation of Charité-Universitätsmedizin Berlin and Max-Delbrück-Center for Molecular Medicine, Berlin, Germany.; Max-Delbrück-Center for Molecular Medicine in the Helmholtz Association, Berlin, Germany.; DZHK (German Centre for Cardiovascular Research), partner site Berlin, Germany.; Charité-Universitätsmedizin Berlin, Corporate member of Freie Universität Berlin and Humboldt-Universität zu Berlin, Berlin, Germany., Müller DN; Experimental and Clinical Research Center, A Cooperation of Charité-Universitätsmedizin Berlin and Max-Delbrück-Center for Molecular Medicine, Berlin, Germany.; Max-Delbrück-Center for Molecular Medicine in the Helmholtz Association, Berlin, Germany.; DZHK (German Centre for Cardiovascular Research), partner site Berlin, Germany.; Charité-Universitätsmedizin Berlin, Corporate member of Freie Universität Berlin and Humboldt-Universität zu Berlin, Berlin, Germany., Wilck N; Experimental and Clinical Research Center, A Cooperation of Charité-Universitätsmedizin Berlin and Max-Delbrück-Center for Molecular Medicine, Berlin, Germany.; Max-Delbrück-Center for Molecular Medicine in the Helmholtz Association, Berlin, Germany.; DZHK (German Centre for Cardiovascular Research), partner site Berlin, Germany.; Department of Nephrology and Internal Intensive Care Medicine, Charité-Universitätsmedizin Berlin, Corporate member of Freie Universität Berlin and Humboldt-Universität zu Berlin, Berlin, Germany.
Publikováno v: Cardiovascular research [Cardiovasc Res] 2023 Jun 13; Vol. 119 (6), pp. 1441-1452.
Zobrazit plný text záznamu
5
Akademický článek
Tento výsledek nelze pro nepřihlášené uživatele zobrazit.
K zobrazení výsledku je třeba se přihlásit.
6
Increased angiotensin II formation in the brain modulates cardiovascular homeostasis and erythropoiesis
Autor: Rodrigues, A.F., Todiras, M., Qadri, F., Campagnole-Santos, M.J., Alenina, N., Bader, M.
In spite of the fact that the modulatory effects of angiotensin II (Ang II) on the sympathetic nerve activity to targeted organs involved in blood pressure (BP) regulation is well acknowledged, the local production of this peptide in the brain and th
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=mdc______med::c313037dd2ba453ac4a8bc5e99ae3e0d
http://edoc.mdc-berlin.de/20298/1/20298oa.pdf
Zobrazit plný text záznamu
7
Phenylalanine-hydroxylase contributes to serotonin synthesis in mice
Autor: Mordhorst, A., Dhandapani, P., Matthes, S., Mosienko, V., Rothe, M., Todiras, M., Self, J., Schunck, W.H., Schütz, A., Bader, M., Alenina, N.
Serotonin is an important signaling molecule in the periphery and in the brain. The hydroxylation of tryptophan is the first and rate‐limiting step of its synthesis. In most vertebrates, two enzymes have been described to catalyze this step, trypto
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=mdc______med::caa9b4ac5110b7d4c72d2173e85a71c9
http://edoc.mdc-berlin.de/20219/1/20219oa.pdf
Zobrazit plný text záznamu
8
Akademický článek
Characterisation of the Antibody Response in Sinopharm (BBIBP-CorV) Recipients and COVID-19 Convalescent Sera from the Republic of Moldova.
Autor: Ulinici M; National Institute for Health and Medical Research, Faculty of Medicine, Nicolae Testemitanu State University of Medicine and Pharmacy, 2004 Chisinau, Moldova.; Alfa Diagnostica Laboratory, 2021 Chisinau, Moldova., Suljič A; Institute of Microbiology and Immunology, Faculty of Medicine, University of Ljubljana, SI-1000 Ljubljana, Slovenia., Poggianella M; Laboratory of Molecular Virology, International Centre for Genetic Engineering and Biotechnology (ICGEB), 34149 Trieste, Italy., Milan Bonotto R; Laboratory of Molecular Virology, International Centre for Genetic Engineering and Biotechnology (ICGEB), 34149 Trieste, Italy., Resman Rus K; Institute of Microbiology and Immunology, Faculty of Medicine, University of Ljubljana, SI-1000 Ljubljana, Slovenia., Paraschiv A; National Institute for Health and Medical Research, Faculty of Medicine, Nicolae Testemitanu State University of Medicine and Pharmacy, 2004 Chisinau, Moldova., Bonetti AM; Laboratory of Molecular Virology, International Centre for Genetic Engineering and Biotechnology (ICGEB), 34149 Trieste, Italy., Todiras M; National Institute for Health and Medical Research, Faculty of Medicine, Nicolae Testemitanu State University of Medicine and Pharmacy, 2004 Chisinau, Moldova., Corlateanu A; National Institute for Health and Medical Research, Faculty of Medicine, Nicolae Testemitanu State University of Medicine and Pharmacy, 2004 Chisinau, Moldova., Groppa S; National Institute for Health and Medical Research, Faculty of Medicine, Nicolae Testemitanu State University of Medicine and Pharmacy, 2004 Chisinau, Moldova., Ceban E; National Institute for Health and Medical Research, Faculty of Medicine, Nicolae Testemitanu State University of Medicine and Pharmacy, 2004 Chisinau, Moldova., Petrovec M; Institute of Microbiology and Immunology, Faculty of Medicine, University of Ljubljana, SI-1000 Ljubljana, Slovenia., Marcello A; Laboratory of Molecular Virology, International Centre for Genetic Engineering and Biotechnology (ICGEB), 34149 Trieste, Italy.
Publikováno v: Vaccines [Vaccines (Basel)] 2023 Mar 13; Vol. 11 (3). Date of Electronic Publication: 2023 Mar 13.
Zobrazit plný text záznamu
9
Angiotensin‐II receptor type Ia does not contribute to cardiac atrophy following high‐thoracic spinal cord injury in mice
Autor: Järve, A., Qadri, F., Todiras, M., Schmolke, S., Bader, M.
NEW FINDINGS: What is the central question of this study? Spinal cord injury leads to cardiac atrophy and the role of the renin-angiotensin system with angiotensin II acting via its receptor AT1a in this effect has not been previously explored. What
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=mdc______med::cb8d752f57bf55b74cf173ff9f6b90a5
http://edoc.mdc-berlin.de/19046/1/19046oa.pdf
Zobrazit plný text záznamu
10
The absence of serotonin in the brain alters acute stress responsiveness by interfering with the genomic function of the glucocorticoid receptors
Autor: Sbrini, G., Brivio, P., Peeva, P.M., Todiras, M., Bader, M., Alenina, N., Calabrese, F.
Alterations in serotonergic transmission have been related to a major predisposition to develop psychiatric pathologies, such as depression. We took advantage of tryptophan hydroxylase (TPH) 2 deficient rats, characterized by a complete absence of se
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=mdc______med::a1b489c15351a0a813d32cdfa3677c37
http://edoc.mdc-berlin.de/19068/1/19068oa.pdf
Zobrazit plný text záznamu

Vyhledávací nástroje:

Upřesnit hledání

Omezení vyhledávání
Plný text Recenzováno Digitální knihovna AV ČR
Zdroje