Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Tinglong Jin"'
Publikováno v:
Frontiers in Endocrinology, Vol 15 (2024)
ObjectiveDiabetic nephropathy (DN) is a major microvascular complication of diabetes and the leading cause of end-stage renal disease. Early detection and prevention of DN are important. Retinol-binding protein 4 (RBP4) has been considered as a singl
Externí odkaz:
https://doaj.org/article/4db4f15a0e3c450b8219a4d37be650c8
Publikováno v:
Artificial Cells, Nanomedicine, and Biotechnology, Vol 47, Iss 1, Pp 2379-2388 (2019)
We, the Editors and Publisher of the journal Artificial Cells, Nanomedicine, and Biotechnology, have retracted the following article:Xiabo Chen, Renxiong Wei, Tinglong Jin & Hanguang Du (2019) Notoginsenoside R1 alleviates TNF-α-induced pancreatic
Externí odkaz:
https://doaj.org/article/eaecc337f76c4ee0ae29991920a91dba
Publikováno v:
BMC Complementary and Alternative Medicine, Vol 19, Iss 1, Pp 1-9 (2019)
Abstract Background Wenshen Jianpi recipe (WSJPR), a blended traditional Chinese medicine, is considered to have the possible beneficial effect on the progression of diabetic nephropathy (DN). This present study was designed to elucidate this protect
Externí odkaz:
https://doaj.org/article/db8a656883c94203a5ac19d7c5a9042b
Publikováno v:
Frontiers in Endocrinology; 5/6/2024, p1-10, 10p
Publikováno v:
BMC Complementary and Alternative Medicine
BMC Complementary and Alternative Medicine, Vol 19, Iss 1, Pp 1-9 (2019)
BMC Complementary and Alternative Medicine, Vol 19, Iss 1, Pp 1-9 (2019)
Background Wenshen Jianpi recipe (WSJPR), a blended traditional Chinese medicine, is considered to have the possible beneficial effect on the progression of diabetic nephropathy (DN). This present study was designed to elucidate this protective activ
Publikováno v:
Artificial cells, nanomedicine, and biotechnology. 47(1)
Diabetes mellitus (DM) is a metabolic disorder that seriously harms human health. Notoginsenoside R1 (NGR1) can be used in various diseases. We explored consequents of NGR1 on tumour necrosis factor (TNF)-α-stimulated Min6 and rat primary islets β