Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Timothy Fabisiak"'
Autor:
Ashwini Sri Hari, Rajeswari Banerji, Li-Ping Liang, Ruth E. Fulton, Christopher Quoc Huynh, Timothy Fabisiak, Pallavi Bhuyan McElroy, James R. Roede, Manisha Patel
Publikováno v:
Redox Biology, Vol 67, Iss , Pp 102895- (2023)
Glutathione (GSH) depletion, and impaired redox homeostasis have been observed in experimental animal models and patients with epilepsy. Pleiotropic strategies that elevate GSH levels via transcriptional regulation have been shown to significantly de
Externí odkaz:
https://doaj.org/article/ed4713f353534d5c94fce6e8d5efe468
Autor:
Timothy Fabisiak, Manisha Patel
Publikováno v:
Frontiers in Cell and Developmental Biology, Vol 10 (2022)
The roles of both neuroinflammation and oxidative stress in the pathophysiology of epilepsy have begun to receive considerable attention in recent years. However, these concepts are predominantly studied as separate entities despite the evidence that
Externí odkaz:
https://doaj.org/article/17ef0460a64f4a60afd8ba3e20179e9e
Autor:
Ruth E. Fulton, Jennifer N. Pearson-Smith, Christopher Q. Huynh, Timothy Fabisiak, Li-Ping Liang, Stefanos Aivazidis, Brigit A. High, Georgia Buscaglia, Timothy Corrigan, Robert Valdez, Takahiko Shimizu, Manisha N. Patel
Publikováno v:
Neurobiology of Disease, Vol 158, Iss , Pp 105470- (2021)
Mitochondrial superoxide (O2−) production is implicated in aging, neurodegenerative disease, and most recently epilepsy. Yet the specific contribution of neuronal O2− to these phenomena is unclear. Here, we selectively deleted superoxide dismutas
Externí odkaz:
https://doaj.org/article/05ca107846c64e9d90c8cc9f4faa05a3
Autor:
Brigit A. High, Ruth Fulton, Timothy Fabisiak, Georgia Buscaglia, Jennifer N. Pearson-Smith, Stefanos Aivazidis, Christopher Q Huynh, Li-Ping Liang, Takahiko Shimizu, Timothy Corrigan, Manisha Patel, Robert Valdez
Publikováno v:
Neurobiology of Disease, Vol 158, Iss, Pp 105470-(2021)
Neurobiol Dis
Neurobiol Dis
Mitochondrial superoxide (O(2)(.−)) production is implicated in aging, neurodegenerative disease, and most recently epilepsy. Yet the specific contribution of neuronal O(2)(.−) to these phenomena is unclear. Here, we selectively deleted superoxid