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of 3
pro vyhledávání: '"Tim Trobisch"'
Autor:
Celia Lerma-Martin, Pau Badia-i-Mompel, Ricardo O. Ramirez Flores, Patricia Sekol, Annika Hofmann, Thomas Thäwel, Christian J. Riedl, Florian Wünnemann, Miguel A. Ibarra-Arellano, Tim Trobisch, Philipp Eisele, Denis Schapiro, Maximilian Haeussler, Simon Hametner, Julio Saez-Rodriguez, Lucas Schirmer
Multiple sclerosis (MS) is a prototypic chronic-inflammatory disease of the central nervous system. After initial lesion formation during active demyelination, inflammation is gradually compartmentalized and restricted to specific tissue areas such a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::f11b1146c16cff385def94c52c8b78cf
https://doi.org/10.1101/2022.11.03.514906
https://doi.org/10.1101/2022.11.03.514906
Autor:
Tim Trobisch, Amel Zulji, Nikolas A. Stevens, Sophia Schwarz, Sven Wischnewski, Mikail Öztürk, Javier Perales-Patón, Maximilian Haeussler, Julio Saez-Rodriguez, Dmitry Velmeshev, Lucas Schirmer
Publikováno v:
Acta neuropathologica, vol 144, iss 5
Multiple sclerosis (MS) is a multifocal and progressive inflammatory disease of the central nervous system (CNS). However, the compartmentalized pathology of the disease affecting various anatomical regions including gray and white matter and lack of
Autor:
Philipp C. Münch, Lukas Bunse, Camila Fernández-Zapata, Josef Priller, Oliver Schnell, Sabine Heiland, Jürgen Beck, Edward W. Green, Chotima Böttcher, Denis Abu-Sammour, Francisco J. Quintana, Roman Sankowski, Felix Sahm, Carina Ramallo Guevara, Wolfgang Wick, Khwab Sanghvi, Tobias Kessler, Tim Trobisch, Frederik Cichon, Carsten Hopf, Lucas Schirmer, Michael Platten, Dieter Henrik Heiland, Miriam Ratliff, Anna von Landenberg, Theresa Bunse, Jana K. Sonner, Daniel Schrimpf, Mirco Friedrich, Marco Prinz, Hagen M. Gegner, Ilona Gutcher, Stefan Pusch, Michael Kilian, Gernot Poschet, Katrin Aslan, Markus Hahn, Andreas von Deimling
Publikováno v:
Nature cancer 2(7), 723-740 (2021). doi:10.1038/s43018-021-00201-z
The dynamics and phenotypes of intratumoral myeloid cells during tumor progression are poorly understood. Here we define myeloid cellular states in gliomas by longitudinal single-cell profiling and demonstrate their strict control by the tumor genoty