Zobrazeno 1 - 3
of 3
pro vyhledávání: '"Tim Putzka"'
Autor:
Nicole Schröder, Anja Schaffrath, Josua A. Welter, Tim Putzka, Angelika Griep, Patrick Ziegler, Elisa Brandt, Sebastian Samer, Michael T. Heneka, Hannes Kaddatz, Jiangshan Zhan, Eugenia Kipp, Thomas Pufe, Simone C. Tauber, Markus Kipp, Lars-Ove Brandenburg
Publikováno v:
Journal of Neuroinflammation, Vol 17, Iss 1, Pp 1-15 (2020)
Abstract Background An important hallmark of Alzheimer’s disease (AD) is the increase of Aβ1-42 burden and its accumulation to senile plaques, leading the reactive gliosis and neurodegeneration. The modulation of glia cell function represents an a
Externí odkaz:
https://doaj.org/article/b44e213a449e4510a06c4e89b88026b5
Autor:
Sebastian Samer, Thomas Pufe, EF Brandt, Patrick Ziegler, Jiangshan Zhan, Hannes Kaddatz, Josua A. Welter, Eugenia Kipp, Nicole Schröder, Lars-Ove Brandenburg, Markus Kipp, Michael T. Heneka, Simone C. Tauber, Tim Putzka, Anja Schaffrath, Angelika Griep
Publikováno v:
Journal of Neuroinflammation, Vol 17, Iss 1, Pp 1-15 (2020)
Journal of Neuroinflammation
Journal of neuroinflammation 17(1), 131 (2020). doi:10.1186/s12974-020-01816-2
Journal of Neuroinflammation
Journal of neuroinflammation 17(1), 131 (2020). doi:10.1186/s12974-020-01816-2
Background An important hallmark of Alzheimer’s disease (AD) is the increase of Aβ1-42 burden and its accumulation to senile plaques, leading the reactive gliosis and neurodegeneration. The modulation of glia cell function represents an attractive
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::03de95900e2208fc29ed388125b58cc8
http://link.springer.com/article/10.1186/s12974-020-01816-2
http://link.springer.com/article/10.1186/s12974-020-01816-2
Autor:
Nicole Schröder, Anja Schaffrath, Josua A. Welter, Tim Putzka, Angelika Griep, Patrick Ziegler, Elisa Brandt, Sebastian Samer, Michael T. Heneka, Eugenia Kipp, Thomas Pufe, Simone C. Tauber, Markus Kipp, Lars-Ove Brandenburg
Background: An important hallmark of Alzheimer's disease (AD) is the increase of Aβ1-42 burden and its accumulation to senile plaques, leading the reactive gliosis and neurodegeneration. The modulation of glia cell function represent an attractive t
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::71806d5294a4e88528370234313357bf
https://doi.org/10.21203/rs.2.17967/v1
https://doi.org/10.21203/rs.2.17967/v1
