Zobrazeno 1 - 10
of 15
pro vyhledávání: '"Tibor Glant"'
Autor:
Ádám Horváth, Éva Borbély, Kata Bölcskei, Nikolett Szentes, Tamás Kiss, Mátyás Belák, Tibor Rauch, Tibor Glant, Róza Zákány, Tamás Juhász, Edina Karanyicz, Ferenc Boldizsár, Zsuzsanna Helyes, Bálint Botz
Publikováno v:
Journal of Neuroinflammation, Vol 15, Iss 1, Pp 1-12 (2018)
Abstract Objective The regulatory role of capsaicin-sensitive peptidergic sensory nerves has been shown in acute inflammation, but little is known about their involvement in T/B-cell driven autoimmune arthritis. This study integratively characterized
Externí odkaz:
https://doaj.org/article/caf56b2373f0485180b48577b7338bc7
Autor:
Tibor Glant
Publikováno v:
Gerundium, Vol 11, Iss 1-2 (2020)
Ma Magyarországon vajmi keveset tudunk az amerikai felsőoktatásról, annak történetéről és problémáiról. Ez nagymértékben a kommunista örökségnek tudható be, hiszen 1945/47 és 1989 között az amerikanisztika a „tűrt” es a „t
Externí odkaz:
https://doaj.org/article/17f3e96889e141f2bc3c24d31fad514c
Autor:
Tibor Glant
Publikováno v:
Hungarian Cultural Studies, Vol 9, Iss 0, Pp 184-196 (2016)
1966, the tenth anniversary of the 1956 Revolution, was a key year in US–Hungarian relations. Diplomatic relations were raised from the lowest to the highest level, but suspicion and tension remained. Neither side knew what to expect from the other
Externí odkaz:
https://doaj.org/article/8a8f2be8f8dd490e95fc88eaab90c155
Autor:
Daniel H Zimmerman, Katalin Mikecz, Kenneth S Rosenthal, Adrienn Markovics, Roy Carambula, Jason Ciemielewski, Tibor Glant
Publikováno v:
The Journal of Immunology. 202:133.4-133.4
Background Development of a therapeutic vaccine for rheumatoid arthritis (RA) requires modulating antigen specific inflammatory T cell responses that drive disease. RA can be initiated and driven by responses to multiple antigens but peptide vaccines
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::ed48fd0fd79d1b59efd86c2ef3d6362c
https://doi.org/10.4049/jimmunol.202.supp.133.4
https://doi.org/10.4049/jimmunol.202.supp.133.4
Autor:
Tibor Glant
Publikováno v:
Studies in Travel Writing. 16:287-301
The written account of Count Bela Szechenyi's 1862 trip to the United States, published in 1863, represents a unique chapter in Hungarian travel writing on the New World. The author was the first-born son of Count Istvan Szechenyi, an iconic figure o
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::38e29f61c4ccb334c114432b2dc60e00
https://doi.org/10.1080/13645145.2012.701068
https://doi.org/10.1080/13645145.2012.701068
Autor:
Charlotte, de Wolf, Ruurd, van der Zee, Ineke, den Braber, Tibor, Glant, Bernard, Maillère, Emmanuel, Favry, Menno, van Lummel, Frits, Koning, Aad, Hoek, Irene, Ludwig, Willem, van Eden, Femke, Broere
Publikováno v:
Arthritisrheumatology (Hoboken, N.J.). 68(3)
We previously showed that mycobacterial Hsp70-derived peptide B29 induced B29-specific Treg cells that suppressed experimental arthritis in mice via cross-recognition of their mammalian Hsp70 homologs. The aim of the current study was to characterize
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::d83d3e73575b07dee5557799c9bbca76
https://pubmed.ncbi.nlm.nih.gov/26414917
https://pubmed.ncbi.nlm.nih.gov/26414917
Autor:
Daniel H Zimmerman, Kenneth Rosenthal, Roy Carambula, Steve Cress, Adrienn Markovics, Tibor Glant, Katalin Mikecz
Publikováno v:
The Journal of Immunology. 198:224.14-224.14
Background Pro-inflammatory T helper (Th)17 or Th1 cell responses (IL17, IFNγ, TNFα) drive autoimmune pathology in rheumatoid arthritis (RA) at the expense of anti-inflammatory/regulatory (IL4, IL10, TGFβ) T cell responses. Ligand Epitope Antigen
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::c50c5dc1a50c62252785fd34ecee0f91
https://doi.org/10.4049/jimmunol.198.supp.224.14
https://doi.org/10.4049/jimmunol.198.supp.224.14
Publikováno v:
The Journal of Immunology. 190:67.6-67.6
Introduction. A major therapeutic goal in autoimmune inflammatory diseases such as rheumatoid arthritis (RA) is to suppress responses against (auto)antigens present in the target organs. Antigen-specific therapies such as the Ligand Epitope Antigen P
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::e2e6f0be48c8ad6eccf2b947389be451
https://doi.org/10.4049/jimmunol.190.supp.67.6
https://doi.org/10.4049/jimmunol.190.supp.67.6
Publikováno v:
The Journal of Immunology. 186:167.21-167.21
Joint damage in rheumatoid arthritis (RA) is largely mediated by synovial fluid (SF) neutrophils. In proteoglycan (PG)-induced arthritis (PGIA), an autoimmune mouse model of RA, SF cells show high expression of the myeloid lineage markers CD11b and G
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::dce30028fc18105de3bb9ff421817257
https://doi.org/10.4049/jimmunol.186.supp.167.21
https://doi.org/10.4049/jimmunol.186.supp.167.21