Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Tianying Bian"'
Publikováno v:
Mediators of Inflammation, Vol 2017 (2017)
The aim of this study was to investigate the role of human β-defensin 3 (hBD3) in the initiation stage of atherosclerosis with human umbilical vein endothelial cells (HUVECs) triggered by tumor necrosis factor- (TNF-) α. The effects of hBD3 on TNF-
Externí odkaz:
https://doaj.org/article/11692686ab5744ff963d55fc858b3fba
Publikováno v:
BIOCELL. 46:2281-2289
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::35fb0acca902a1b39b3ee7a7e05729c2
https://doi.org/10.32604/biocell.2022.020697
https://doi.org/10.32604/biocell.2022.020697
Autor:
Fuhua Yan, Jing Zhou, Yin Xiao, Tianying Bian, Yangheng Zhang, Wenrong Yang, Xiaofeng Huang, Na Kong, Can Ni
Publikováno v:
Biomaterials. 206:115-132
The regeneration of lost periodontal apparatus in periodontitis treatment remains a clinical challenge due to the limited regenerative capacity of cementum, periodontal ligament and alveolar bone in periodontitis condition. For periodontal tissue reg
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::174ce335048c4a880cd41bd3ab7f8c1f
https://doi.org/10.1016/j.biomaterials.2019.03.039
https://doi.org/10.1016/j.biomaterials.2019.03.039
Publikováno v:
Molecular Immunology. 91:65-74
Human β-defensin 3 (hBD3) is a cationic peptide with immunomodulatory effects on both innate and acquired immune responses. Periodontitis, an inflammatory disease that extends deep into periodontal tissues, causes the loss of supporting structures a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a58532e189046ba8ec93de876761e987
https://doi.org/10.1016/j.molimm.2017.08.012
https://doi.org/10.1016/j.molimm.2017.08.012
Publikováno v:
Mediators of Inflammation, Vol 2017 (2017)
Mediators of Inflammation
Mediators of Inflammation
The aim of this study was to investigate the role of humanβ-defensin 3 (hBD3) in the initiation stage of atherosclerosis with human umbilical vein endothelial cells (HUVECs) triggered by tumor necrosis factor- (TNF-)α. The effects of hBD3 on TNF-α
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::91bd541cc2721293279e59f1a6893add
Publikováno v:
Cytokine. 92
β-defensin 3, a multifunctional antimicrobial peptide, has immuno-regulatory activities. We investigated the modulatory mechanism of human β-defensin 3 (hBD3) on acute inflammatory response resulted from Porphyromonas gingivalis lipopolysaccharide
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::396519cf0ec87042ff16108949ef8c20
https://pubmed.ncbi.nlm.nih.gov/28092794
https://pubmed.ncbi.nlm.nih.gov/28092794
Publikováno v:
International immunopharmacology. 38
Background and aim Porphyromonas gingivalis ( P. gingivalis ) lipopolysaccharide (LPS) is reported to be associated with the progression of atherosclerosis (AS). In this study, we explored the potential of human β-defensin-3 (hBD3), an antimicrobial
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::db3ef70f410597de973e499b66a76803
https://pubmed.ncbi.nlm.nih.gov/27289299
https://pubmed.ncbi.nlm.nih.gov/27289299
Publikováno v:
Peptides. 82
Human beta-defensin 3 (hBD3) is an antimicrobial peptide showing immunomodulatory effect on both innate and acquired immune response. Atherosclerosis is an inflammatory disease characterized by accumulation of lipids in the vascular wall. In this stu
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6368e553ae1688bb5fc2c9ad0244007c
https://pubmed.ncbi.nlm.nih.gov/27298203
https://pubmed.ncbi.nlm.nih.gov/27298203