Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Thomas P Pierce"'
Autor:
Nancy Wang, Timothy A Scott, Andreas Kupz, Meghanashree M Shreenivas, Newton G Peres, Dianna M Hocking, Chenying Yang, Leila Jebeli, Lynette Beattie, Joanna R Groom, Thomas P Pierce, Linda M Wakim, Sammy Bedoui, Richard A Strugnell
Publikováno v:
PLoS Pathogens, Vol 19, Iss 9, p e1011666 (2023)
Prior infection can generate protective immunity against subsequent infection, although the efficacy of such immunity can vary considerably. Live-attenuated vaccines (LAVs) are one of the most effective methods for mimicking this natural process, and
Externí odkaz:
https://doaj.org/article/f3d56009cb644d75a2d9d9ab1727aad4
Publikováno v:
The Scientific World Journal, Vol 1, Pp 356-362 (2001)
Significant uncertainty exists in magnitude and variability of ammonia (NH3) emissions, which are needed for air quality modeling of aerosols and deposition of nitrogen compounds. Approximately 85% of NH3 emissions are estimated to come from agricult
Externí odkaz:
https://doaj.org/article/e2d238c9bcc949ca9d63a46013d79561
Autor:
Mariah G, Alorro, Thomas P, Pierce, Moritz F, Eissmann, Christine, Dijkstra, Ross A, Dickins, Matthias, Ernst, Michael, Buchert, Frederic, Masson
Publikováno v:
Genesis (New York, N.Y. : 2000). 55(4)
Signal transducer and activator of transcription 3 (Stat3) is a transcription factor that has many essential roles during inflammation, development and cancer. Stat3 is therefore an attractive therapeutic target in many diseases. While current Stat3
Autor:
Christopher G. Love, Zhengdeng Lei, Steve Rozen, Andrew G. Jarnicki, Michelle Palmieri, Adele Preaudet, Fred Schaper, Michael Hallek, Patrick Tan, Alex Boussioutas, Matthias Ernst, Tracy L Putoczki, Ryan Farid, Thomas P. Pierce, Stefan Thiem, Veena Gopalakrishnan, Bruno Catimel, Michael Buchert
Publikováno v:
The Journal of clinical investigation. 123(2)
Gastrointestinal cancers are frequently associated with chronic inflammation and excessive secretion of IL-6 family cytokines, which promote tumorigenesis through persistent activation of the GP130/JAK/STAT3 pathway. Although tumor progression can be
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