Zobrazeno 1 - 10
of 1 319
pro vyhledávání: '"Thomas G Scott"'
Autor:
Vohs,, Paul A.
Publikováno v:
Wildlife Society Bulletin (1973-2006), 1998 Oct 01. 26(3), 640-642.
Externí odkaz:
https://www.jstor.org/stable/3783781
Publikováno v:
PLoS Genetics, Vol 20, Iss 2, p e1011159 (2024)
Common genetic variants in the repressive GATA-family transcription factor (TF) TRPS1 locus are associated with breast cancer risk, and luminal breast cancer cell lines are particularly sensitive to TRPS1 knockout. We introduced an inducible degron t
Externí odkaz:
https://doaj.org/article/1ea7745be3074667b30911fbb237b595
Autor:
Allen, Durward L.
Publikováno v:
The Journal of Wildlife Management, 1956 Apr 01. 20(2), 196-197.
Externí odkaz:
https://www.jstor.org/stable/3797428
Autor:
Goodnight, Clarence J.
Publikováno v:
The American Biology Teacher, 1962 May 01. 24(5), 375-376.
Externí odkaz:
https://www.jstor.org/stable/4440009
Autor:
Thomas G. Scott
Publikováno v:
An evaluation of the red fox / Thomas G. Scott.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::9f377c94e5f73cea1b6bc34d805ce9ee
https://doi.org/10.5962/bhl.title.15124
https://doi.org/10.5962/bhl.title.15124
Autor:
Sylwia Hasterok, Thomas G. Scott, Devin G. Roller, Adam Spencer, Arun B. Dutta, Kizhakke M Sathyan, Daniel E. Frigo, Michael J. Guertin, Daniel Gioeli
The clinical success of combined androgen deprivation therapy (ADT) and radiation therapy (RT) in prostate cancer (PCa) created interest in understanding the mechanistic links between androgen receptor (AR) signaling and the DNA damage response (DDR)
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::c389664a3c8cb66537286b0269663bc4
https://doi.org/10.1101/2023.05.13.540653
https://doi.org/10.1101/2023.05.13.540653
Autor:
James E. Bradner, George D. Demetri, Scott A. Armstrong, Ewa Sicinska, Chandrajit P. Raut, Thomas G. Scott, Otari Chipashvili, Timothy Hagan, Jessica L. Andersen, Matthew A. Lawlor, Matthew L. Hemming
Gastrointestinal stromal tumor (GIST) is a mesenchymal neoplasm characterized by activating mutations in the related receptor tyrosine kinases KIT and PDGFRA. GIST relies on expression of these unamplified receptor tyrosine kinase (RTK) genes through
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::892a37a37a39293056fb095a998fdb8b
https://doi.org/10.1158/0008-5472.c.6511217
https://doi.org/10.1158/0008-5472.c.6511217