Zobrazeno 1 - 10
of 144
pro vyhledávání: '"Thomas C. Westfall"'
Autor:
Thomas C. Westfall, Martin C. Michel, Dan Larhammar, Herbert Herzog, William F. Colmers, Thue W. Schwartz, Rémi Quirion, Helen M. Cox, Annette G. Beck-Sickinger, Henri Doods
Publikováno v:
IUPHAR/BPS Guide to Pharmacology CITE. 2019
Neuropeptide Y (NPY) receptors (nomenclature as agreed by the NC-IUPHAR Subcommittee on Neuropeptide Y Receptors [156]) are activated by the endogenous peptides neuropeptide Y, neuropeptide Y-(3-36), peptide YY, PYY-(3-36) and pancreatic polypeptide
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::54f49275fd771dedaabf2a5c0f5e9851
https://doi.org/10.2218/gtopdb/f46/2019.4
https://doi.org/10.2218/gtopdb/f46/2019.4
Publikováno v:
Acta Physiologica. 203:37-45
Noradrenaline, neuropeptide Y and adenosine triphosphate are co-stored in, and co-released from, sympathetic nerves. Each transmitter modulates its own release as well as the release of one another; thus, anything affecting the release of one of thes
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::fad591ac1b1054a4681e9f8e0c71f419
https://doi.org/10.1111/j.1748-1716.2010.02242.x
https://doi.org/10.1111/j.1748-1716.2010.02242.x
Publikováno v:
American Journal of Physiology-Heart and Circulatory Physiology. 298:H457-H465
Neuropeptide Y (NPY) is a cotransmitter with norepinephrine (NE) and ATP in sympathetic nerves. There is evidence for increased activity of the sympathetic nervous system and the renin-angiotensin system (RAS), as well as a role for NPY in the develo
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bdb0bb641b0b9655440d445d388cf925
https://doi.org/10.1152/ajpheart.00400.2009
https://doi.org/10.1152/ajpheart.00400.2009
Publikováno v:
NeuroToxicology. 30:1030-1035
Parkinson disease is a specific form of neurodegeneration characterized by a loss of nigra-striatal dopaminergic neurons in the midbrain of humans. The disease is also characterized by an increase in oxidative stress and a loss of glutathione in the
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3566752e25dc9ac0770ec832f95470d5
https://doi.org/10.1016/j.neuro.2009.07.006
https://doi.org/10.1016/j.neuro.2009.07.006
Publikováno v:
American Journal of Physiology-Heart and Circulatory Physiology. 295:H2188-H2197
The sympathetic nervous system and renin-angiotensin system are both thought to contribute to the development and maintenance of hypertension in experimental models such as the spontaneously hypertensive rat (SHR). We demonstrated that periarterial n
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1fc9ea86796d735baf85b5911c7ccec5
https://doi.org/10.1152/ajpheart.00384.2008
https://doi.org/10.1152/ajpheart.00384.2008
Publikováno v:
Biochemical Pharmacology. 73:1446-1454
In rat pheochromocytoma (PC12) cells the dopamine D(2) receptor agonists apomorphine (APO) and n-propylnorapomorphine (NPA) produced a concentration dependent inhibition of K(+)-evoked neuropeptide Y release (NPY-ir). The effect of APO was blocked by
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7fc3c9b0898ff65b3f222db634b5ef28
https://doi.org/10.1016/j.bcp.2007.01.003
https://doi.org/10.1016/j.bcp.2007.01.003
Publikováno v:
Neuropharmacology. 52:1396-1402
The purpose of the present study was to determine whether or not activation of neuropeptide Y (NPY) receptors resulted in an enhancement or attenuation of the KCl (50 mM) evoked release of [3H]dopamine newly synthesized from [3H]tyrosine in superfuse
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bf5a03096e86c9f1e7517aa0a28de281
https://doi.org/10.1016/j.neuropharm.2007.01.018
https://doi.org/10.1016/j.neuropharm.2007.01.018
Publikováno v:
Journal of Cardiovascular Pharmacology. 47:723-728
The effect of neuropeptide Y (NPY) on the basal and nerve stimulation-induced increase in norepinephrine synthesis was studied in the isolated and perfused mesenteric arterial bed of the rat. Tyrosine hydroxylation, the rate-limiting step in catechol
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1b15d7ef2479e1ea7fc219c90707eb05
https://doi.org/10.1097/01.fjc.0000211761.06271.15
https://doi.org/10.1097/01.fjc.0000211761.06271.15
Publikováno v:
American Journal of Physiology-Heart and Circulatory Physiology. 286:H296-H303
Nitric oxide (NO) reacts with catecholamines resulting in their deactivation. In this study, we demonstrated that coincubation of NO donors with sympathetic neurotransmitters decreased the amount of norepinephrine detected but not ATP or neuropeptide
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::246a7fc31b5ed6436cb422eb67ee6fe9
https://doi.org/10.1152/ajpheart.00668.2003
https://doi.org/10.1152/ajpheart.00668.2003
Publikováno v:
Autonomic and Autacoid Pharmacology. 23:141-147
Summary 1 A variety of prostanoids were examined for their ability to alter the periarterial nerve stimulation-induced release of noradrenaline (NA) and neuropeptide Y immunoreactive compounds (NPY-ir) from the perfused mesenteric arterial bed of the
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2b1ff7a31ecf779329a7dfc14fb8109b
https://doi.org/10.1046/j.1474-8673.2003.00288.x
https://doi.org/10.1046/j.1474-8673.2003.00288.x