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A Role for the Non-Receptor Tyrosine Kinase Abl2/Arg in Experimental Neuroinflammation

Autor: Freja Aksel Jacobsen, Jeppe Christian Mouritsen, Dan Holmberg, B Thomas Bäckström, Alexander N. Scherer, Samra Sardar, Anthony J. Koleske, Åsa Andersson, Hera Bragadóttir

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Journal of Neuroimmune Pharmacology

Multiple sclerosis is a neuroinflammatory degenerative disease, caused by activated immune cells infiltrating the CNS. The disease etiology involves both genetic and environmental factors. The mouse genetic locus, Eae27, linked to disease development

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ae1664598bbfb38486b0226028e3e079
https://curis.ku.dk/ws/files/234566403/11481_2018_Article_9783.pdf