Zobrazeno 1 - 10
of 336
pro vyhledávání: '"Theodore J, Standiford"'
Autor:
Jose Victor Jimenez, Elizabeth Munroe, Andrew J. Weirauch, Kelly Fiorino, Christopher A. Culter, Kristine Nelson, Wassim W. Labaki, Philip J. Choi, Ivan Co, Theodore J. Standiford, Hallie C. Prescott, Robert C. Hyzy
Publikováno v:
Critical Care, Vol 27, Iss 1, Pp 1-7 (2023)
Abstract Background In patients with acute respiratory distress syndrome undergoing mechanical ventilation, positive end-expiratory pressure (PEEP) can lead to recruitment or overdistension. Current strategies utilized for PEEP titration do not permi
Externí odkaz:
https://doaj.org/article/2c873f9ab5af41baa0be54452a217946
Autor:
Filiz T. Korkmaz, Anukul T. Shenoy, Elise M. Symer, Lillia A. Baird, Christine V. Odom, Emad I. Arafa, Ernest L. Dimbo, Elim Na, William Molina-Arocho, Matthew Brudner, Theodore J. Standiford, Jawahar L. Mehta, Tatsuya Sawamura, Matthew R. Jones, Joseph P. Mizgerd, Katrina E. Traber, Lee J. Quinton
Publikováno v:
JCI Insight, Vol 7, Iss 23 (2022)
Identifying host factors that contribute to pneumonia incidence and severity are of utmost importance to guiding the development of more effective therapies. Lectin-like oxidized low-density lipoprotein receptor 1 (LOX-1, encoded by OLR1) is a scaven
Externí odkaz:
https://doaj.org/article/b320f241dc9942bba46957a1d10f8c08
Autor:
Yuzi Tian, Patrick Li, Zhenyu Wu, Qiufang Deng, Baihong Pan, Kathleen A. Stringer, Hasan B. Alam, Theodore J. Standiford, Yongqing Li
Publikováno v:
Frontiers in Immunology, Vol 12 (2021)
Sepsis is a life-threatening organ dysfunction caused by dysregulated host response to infection that often results in acute lung injury (ALI)/acute respiratory distress syndrome (ARDS). An emerging mechanism of sepsis-induced ARDS involves neutrophi
Externí odkaz:
https://doaj.org/article/69193551dfae406087cb329a85749787
Publikováno v:
Frontiers in Immunology, Vol 12 (2021)
The IL-36 family of cytokines were identified in the early 2000’s as a new subfamily of the IL-1 cytokine family, and since then, the role of IL-36 cytokines during various inflammatory processes has been characterized. While most of the research h
Externí odkaz:
https://doaj.org/article/7515a4e4023b479f9acd42feaa6c8494
Autor:
Hernán F. Peñaloza, Tolani F. Olonisakin, William G. Bain, Yanyan Qu, Rick van der Geest, Jill Zupetic, Mei Hulver, Zeyu Xiong, Michael W. Newstead, Chunbin Zou, Jonathan K. Alder, Joel A. Ybe, Theodore J. Standiford, Janet S. Lee
Publikováno v:
mBio, Vol 12, Iss 2 (2021)
Pseudomonas aeruginosa
Externí odkaz:
https://doaj.org/article/c3f646f87d284e3eadcfc706deab5378
Autor:
Yuzi Tian, Shibin Qu, Hasan B. Alam, Aaron M. Williams, Zhenyu Wu, Qiufang Deng, Baihong Pan, Jing Zhou, Baoling Liu, Xiuzhen Duan, Jianjie Ma, Santanu Mondal, Paul R. Thompson, Kathleen A. Stringer, Theodore J. Standiford, Yongqing Li
Publikováno v:
JCI Insight, Vol 5, Iss 20 (2020)
Peptidylarginine deiminases (PADs) are a family of calcium-dependent enzymes that are involved in a variety of human disorders, including cancer and autoimmune diseases. Although targeting PAD4 has shown no benefit in sepsis, the role of PAD2 remains
Externí odkaz:
https://doaj.org/article/0014eeb9c2ae473c8d0d4eee0554907d
Autor:
Michael D. Maile, Theodore J. Standiford, Milo C. Engoren, Kathleen A. Stringer, Elizabeth S. Jewell, Thekkelnaycke M. Rajendiran, Tanu Soni, Charles F. Burant
Publikováno v:
Respiratory Research, Vol 19, Iss 1, Pp 1-8 (2018)
Abstract Background It is unknown if the plasma lipidome is a useful tool for improving our understanding of the acute respiratory distress syndrome (ARDS). Therefore, we measured the plasma lipidome of individuals with ARDS at two time-points to det
Externí odkaz:
https://doaj.org/article/e88796dac5aa4fc391bd95413d6a5319
Autor:
Shailesh Agarwal, Shawn J. Loder, David Cholok, John Li, Guowu Bian, Srilakshmi Yalavarthi, Shuli Li, William F. Carson, Charles Hwang, Simone Marini, Chase Pagani, Nicole Edwards, Matthew J. Delano, Theodore J. Standiford, Jason S. Knight, Steven L. Kunkel, Yuji Mishina, Peter A. Ward, Benjamin Levi
Publikováno v:
Frontiers in Immunology, Vol 10 (2019)
Inflammation after trauma is both critical to normal wound healing and may be highly detrimental when prolonged or unchecked with the potential to impair physiologic healing and promote de novo pathology. Mechanical strain after trauma is associated
Externí odkaz:
https://doaj.org/article/2677d734533846c3970bc16d46f04165
Autor:
Venkateshwar G. Keshamouni, Theodore J. Standiford, Jun Chen, Guido Bommer, Venkata R. Narala, Himabindu Kurapati, Ajaya Kumar Reka
Supplementary Video 1 from Peroxisome Proliferator-Activated Receptor-γ Activation Inhibits Tumor Metastasis by Antagonizing Smad3-Mediated Epithelial-Mesenchymal Transition
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::024e0e9e51e388d547de4bdd5a327617
https://doi.org/10.1158/1535-7163.22484984.v1
https://doi.org/10.1158/1535-7163.22484984.v1
Autor:
Venkateshwar G. Keshamouni, Theodore J. Standiford, Jun Chen, Guido Bommer, Venkata R. Narala, Himabindu Kurapati, Ajaya Kumar Reka
Epithelial-mesenchymal transition (EMT) was shown to confer tumor cells with abilities essential for metastasis, including migratory phenotype, invasiveness, resistance to apoptosis, evading immune surveillance, and tumor stem cell traits. Therefore,
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4497336e2a5889b88e47638a0ae00bbe
https://doi.org/10.1158/1535-7163.c.6531692.v1
https://doi.org/10.1158/1535-7163.c.6531692.v1