Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Thea Reinkens"'
Autor:
Yvonne Lisa Behrens, Gudrun Göhring, Laura Gaschler, Ronny Nienhold, Thea Reinkens, Elke Schirmer, Sabine Lukat, Sabine Knöß, Renate Strasser, Stephanie Sembill, Zofia Wotschofsky, Meinolf Suttorp, Manuela Krumbholz, Brigitte Schlegelberger, Markus Metzler, Axel Karow
Publikováno v:
EJC Paediatric Oncology, Vol 2, Iss , Pp 100067- (2023)
Externí odkaz:
https://doaj.org/article/54bcb69c90bd43ab9dc33c418b839f96
Autor:
Yvonne Lisa Behrens, Laura Gaschler, Ronny Nienhold, Thea Reinkens, Elke Schirmer, Sabine Knöß, Renate Strasser, Stephanie Sembill, Zofia Wotschofsky, Meinolf Suttorp, Manuela Krumbholz, Brigitte Schlegelberger, Markus Metzler, Gudrun Göhring, Axel Karow
Publikováno v:
Haematologica, Vol 109, Iss 3 (2023)
Externí odkaz:
https://doaj.org/article/2991f0a209f34f849c803225ef909f31
Autor:
Nicole Huge, Thea Reinkens, Reena Buurman, Maria Sandbothe, Anke Bergmann, Hannah Wallaschek, Beate Vajen, Amelie Stalke, Melanie Decker, Marlies Eilers, Vera Schäffer, Oliver Dittrich-Breiholz, Engin Gürlevik, Florian Kühnel, Brigitte Schlegelberger, Thomas Illig, Britta Skawran
Publikováno v:
Cancer Cell International, Vol 22, Iss 1, Pp 1-16 (2022)
Abstract Background In hepatocellular carcinoma (HCC), histone deacetylases (HDACs) are frequently overexpressed. This results in chromatin compaction and silencing of tumor-relevant genes and microRNAs. Modulation of microRNA expression is a potenti
Externí odkaz:
https://doaj.org/article/3da413d23a3548bfa6c3093759f16cd4
Autor:
Amelie Stalke, Annika Behrendt, Finja Hennig, Holger Gohlke, Nicole Buhl, Thea Reinkens, Ulrich Baumann, Brigitte Schlegelberger, Thomas Illig, Eva‐Doreen Pfister, Britta Skawran
Publikováno v:
Clinical Genetics.
Autor:
Thea, Reinkens, Amelie, Stalke, Nicole, Huge, Beate, Vajen, Marlies, Eilers, Vera, Schäffer, Oliver, Dittrich-Breiholz, Brigitte, Schlegelberger, Thomas, Illig, Britta, Skawran
Publikováno v:
Journal of Cancer
BACKGROUND: Patients with hepatocellular carcinoma (HCC) have very limited treatment options. For the last fourteen years, the multi-tyrosine kinase inhibitor sorafenib has been used as standard-of-care therapeutic agent in advanced HCC. Unfortunatel
Autor:
Beate Vajen, Rahul Bhowmick, Luisa Greiwe, Vera Schäffer, Marlies Eilers, Thea Reinkens, Amelie Stalke, Gunnar Schmidt, Jan Fiedler, Thomas Thum, David S. DeLuca, Ian D. Hickson, Brigitte Schlegelberger, Thomas Illig, Britta Skawran
Publikováno v:
Vajen, B, Bhowmick, R, Greiwe, L, Schäffer, V, Eilers, M, Reinkens, T, Stalke, A, Schmidt, G, Fiedler, J, Thum, T, Deluca, D S, Hickson, I D, Schlegelberger, B, Illig, T & Skawran, B 2022, ' MicroRNA-449a Inhibits Triple Negative Breast Cancer by Disturbing DNA Repair and Chromatid Separation ', International Journal of Molecular Sciences, vol. 23, no. 9, 5131 . https://doi.org/10.3390/ijms23095131
International Journal of Molecular Sciences; Volume 23; Issue 9; Pages: 5131
International Journal of Molecular Sciences; Volume 23; Issue 9; Pages: 5131
Chromosomal instability (CIN) can be a driver of tumorigenesis but is also a promising therapeutic target for cancer associated with poor prognosis such as triple negative breast cancer (TNBC). The treatment of TNBC cells with defects in DNA repair g
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::36680480b1121792b02815cad038ce59
https://curis.ku.dk/portal/da/publications/microrna449a-inhibits-triple-negative-breast-cancer-by-disturbing-dna-repair-and-chromatid-separation(f96841cc-72d5-4901-a7e6-1c26fdd32596).html
https://curis.ku.dk/portal/da/publications/microrna449a-inhibits-triple-negative-breast-cancer-by-disturbing-dna-repair-and-chromatid-separation(f96841cc-72d5-4901-a7e6-1c26fdd32596).html
Autor:
Nicole Huge, Thea Reinkens, Reena Buurman, Maria Sandbothe, Anke Bergmann, Hannah Wallaschek, Beate Vajen, Amelie Stalke, Melanie Decker, Marlies Eilers, Vera Schäffer, Oliver Dittrich-Breiholz, Engin Gürlevik, Florian Kühnel, Brigitte Schlegelberger, Thomas Illig, Britta Skawran
Publikováno v:
Cancer cell international. 22(1)
Background In hepatocellular carcinoma (HCC), histone deacetylases (HDACs) are frequently overexpressed. This results in chromatin compaction and silencing of tumor-relevant genes and microRNAs. Modulation of microRNA expression is a potential treatm