Progestogen hypersensitivity: successful use of progesterone desensitisation and omalizumab to facilitate in vitro fertilisation.

Autor: Xu G; Department of Immunology, Concord Hospital, Sydney, New South Wales, Australia., Yuson R; Department of Immunology, Concord Hospital, Sydney, New South Wales, Australia., Rafferty M; Department of Immunology, Concord Hospital, Sydney, New South Wales, Australia., Thai TL; Pharmacy Department, Concord Hospital, Sydney, New South Wales, Australia., Limaye S; Department of Immunology, Concord Hospital, Sydney, New South Wales, Australia.; Concord Clinical School, University of Sydney, Sydney, New South Wales, Australia.

Publikováno v: Internal medicine journal [Intern Med J] 2024 Oct; Vol. 54 (10), pp. 1749-1752. Date of Electronic Publication: 2024 Sep 17.

Lovastatin attenuates hypertension induced by renal tubule-specific knockout of ATP-binding cassette transporter A1, by inhibiting epithelial sodium channels.

Autor: Wu MM; Departments of Cardiology and Clinic Pharmacy, Institute of Metabolic Disease, Heilongjiang Academy of Medical Science, Key Laboratories of Education Ministry for Myocardial Ischemia Mechanism and Treatment, Harbin Medical University Cancer Hospital, Harbin, China.; Department of Physiology, Emory University School of Medicine, Atlanta, Georgia., Liang C; Departments of Cardiology and Clinic Pharmacy, Institute of Metabolic Disease, Heilongjiang Academy of Medical Science, Key Laboratories of Education Ministry for Myocardial Ischemia Mechanism and Treatment, Harbin Medical University Cancer Hospital, Harbin, China., Yu XD; Departments of Cardiology and Clinic Pharmacy, Institute of Metabolic Disease, Heilongjiang Academy of Medical Science, Key Laboratories of Education Ministry for Myocardial Ischemia Mechanism and Treatment, Harbin Medical University Cancer Hospital, Harbin, China., Song BL; Departments of Cardiology and Clinic Pharmacy, Institute of Metabolic Disease, Heilongjiang Academy of Medical Science, Key Laboratories of Education Ministry for Myocardial Ischemia Mechanism and Treatment, Harbin Medical University Cancer Hospital, Harbin, China.; Department of Physiology, Emory University School of Medicine, Atlanta, Georgia., Yue Q; Department of Physiology, Emory University School of Medicine, Atlanta, Georgia., Zhai YJ; Department of Physiology, Emory University School of Medicine, Atlanta, Georgia., Linck V; Department of Physiology, Emory University School of Medicine, Atlanta, Georgia., Cai YX; Departments of Cardiology and Clinic Pharmacy, Institute of Metabolic Disease, Heilongjiang Academy of Medical Science, Key Laboratories of Education Ministry for Myocardial Ischemia Mechanism and Treatment, Harbin Medical University Cancer Hospital, Harbin, China., Niu N; Departments of Cardiology and Clinic Pharmacy, Institute of Metabolic Disease, Heilongjiang Academy of Medical Science, Key Laboratories of Education Ministry for Myocardial Ischemia Mechanism and Treatment, Harbin Medical University Cancer Hospital, Harbin, China., Yang X; Departments of Cardiology and Clinic Pharmacy, Institute of Metabolic Disease, Heilongjiang Academy of Medical Science, Key Laboratories of Education Ministry for Myocardial Ischemia Mechanism and Treatment, Harbin Medical University Cancer Hospital, Harbin, China., Zhang BL; Departments of Cardiology and Clinic Pharmacy, Institute of Metabolic Disease, Heilongjiang Academy of Medical Science, Key Laboratories of Education Ministry for Myocardial Ischemia Mechanism and Treatment, Harbin Medical University Cancer Hospital, Harbin, China., Wang QS; Departments of Cardiology and Clinic Pharmacy, Institute of Metabolic Disease, Heilongjiang Academy of Medical Science, Key Laboratories of Education Ministry for Myocardial Ischemia Mechanism and Treatment, Harbin Medical University Cancer Hospital, Harbin, China., Zou L; Department of Physiology, Emory University School of Medicine, Atlanta, Georgia., Zhang S; Department of Physiology, Emory University School of Medicine, Atlanta, Georgia., Thai TL; Department of Physiology, Emory University School of Medicine, Atlanta, Georgia., Ma J; Division of Pulmonary, Allergy, Critical Care and Sleep Medicine, Department of Medicine, Atlanta Veterans Affairs Medical Center, Decatur, Georgia., Sutliff RL; Division of Pulmonary, Allergy, Critical Care and Sleep Medicine, Department of Medicine, Atlanta Veterans Affairs Medical Center, Decatur, Georgia., Zhang ZR; Departments of Cardiology and Clinic Pharmacy, Institute of Metabolic Disease, Heilongjiang Academy of Medical Science, Key Laboratories of Education Ministry for Myocardial Ischemia Mechanism and Treatment, Harbin Medical University Cancer Hospital, Harbin, China., Ma HP; Department of Physiology, Emory University School of Medicine, Atlanta, Georgia.

Publikováno v: British journal of pharmacology [Br J Pharmacol] 2019 Sep; Vol. 176 (18), pp. 3695-3711. Date of Electronic Publication: 2019 Jul 30.

ENaC activity is regulated by calpain-2 proteolysis of MARCKS proteins.

Autor: Montgomery DS; Department of Physiology and Functional Genomics and Department of Medicine Division of Nephrology, Hypertension, and Renal Transplantation, University of Florida College of Medicine, Gainesville, Florida., Yu L; College of Resources and Environmental Sciences, Nanjing Agricultural University, Nanjing, China; and., Ghazi ZM; Department of Physiology, Emory University School of Medicine, Atlanta, Georgia., Thai TL; Department of Physiology, Emory University School of Medicine, Atlanta, Georgia., Al-Khalili O; Department of Physiology, Emory University School of Medicine, Atlanta, Georgia., Ma HP; Department of Physiology, Emory University School of Medicine, Atlanta, Georgia., Eaton DC; Department of Physiology, Emory University School of Medicine, Atlanta, Georgia., Alli AA; Department of Physiology and Functional Genomics and Department of Medicine Division of Nephrology, Hypertension, and Renal Transplantation, University of Florida College of Medicine, Gainesville, Florida; aalli@ufl.edu.

Publikováno v: American journal of physiology. Cell physiology [Am J Physiol Cell Physiol] 2017 Jul 01; Vol. 313 (1), pp. C42-C53. Date of Electronic Publication: 2017 May 03.

Inhibition of TRPC6 reduces non-small cell lung cancer cell proliferation and invasion.

Autor: Yang LL; Department of Radiology, The Fourth Affiliated Hospital of Harbin Medical University, Harbin, Heilongjiang, China.; Molecular Imaging Research Center of Harbin Medical University, Harbin, Heilongjiang, China.; Department of Physiology, Emory University School of Medicine, Atlanta, Georgia, USA., Liu BC; Department of Radiology, The Fourth Affiliated Hospital of Harbin Medical University, Harbin, Heilongjiang, China.; Molecular Imaging Research Center of Harbin Medical University, Harbin, Heilongjiang, China.; Department of Physiology, Emory University School of Medicine, Atlanta, Georgia, USA., Lu XY; Department of Physiology, Emory University School of Medicine, Atlanta, Georgia, USA., Yan Y; Department of Radiology, The Fourth Affiliated Hospital of Harbin Medical University, Harbin, Heilongjiang, China.; Molecular Imaging Research Center of Harbin Medical University, Harbin, Heilongjiang, China., Zhai YJ; Department of Radiology, The Fourth Affiliated Hospital of Harbin Medical University, Harbin, Heilongjiang, China.; Molecular Imaging Research Center of Harbin Medical University, Harbin, Heilongjiang, China.; Department of Physiology, Emory University School of Medicine, Atlanta, Georgia, USA., Bao Q; Department of Physiology, Emory University School of Medicine, Atlanta, Georgia, USA., Doetsch PW; Department of Radiation Oncology, and Biochemistry and Winship Cancer Institute of Emory University, Atlanta, Georgia, USA., Deng X; Department of Radiation Oncology, and Biochemistry and Winship Cancer Institute of Emory University, Atlanta, Georgia, USA., Thai TL; Department of Physiology, Emory University School of Medicine, Atlanta, Georgia, USA.; Center for Cell and Molecular Signaling, Emory University School of Medicine, Atlanta, Georgia, USA., Alli AA; Department of Physiology, Emory University School of Medicine, Atlanta, Georgia, USA.; Center for Cell and Molecular Signaling, Emory University School of Medicine, Atlanta, Georgia, USA., Eaton DC; Department of Physiology, Emory University School of Medicine, Atlanta, Georgia, USA.; Center for Cell and Molecular Signaling, Emory University School of Medicine, Atlanta, Georgia, USA., Shen BZ; Department of Radiology, The Fourth Affiliated Hospital of Harbin Medical University, Harbin, Heilongjiang, China.; Molecular Imaging Research Center of Harbin Medical University, Harbin, Heilongjiang, China., Ma HP; Department of Physiology, Emory University School of Medicine, Atlanta, Georgia, USA.; Center for Cell and Molecular Signaling, Emory University School of Medicine, Atlanta, Georgia, USA.

Publikováno v: Oncotarget [Oncotarget] 2017 Jan 17; Vol. 8 (3), pp. 5123-5134.

The Lectin-like Domain of TNF Increases ENaC Open Probability through a Novel Site at the Interface between the Second Transmembrane and C-terminal Domains of the α-Subunit.

Autor: Lucas R; From the Vascular Biology Center, rlucas@augusta.edu.; the Department of Pharmacology and Toxicology, and.; the Division of Pulmonary and Critical Care Medicine, Medical College of Georgia, Augusta, Georgia 30912., Yue Q; the Department of Physiology, Emory University, Atlanta, Georgia 30322., Alli A; the Department of Physiology, Emory University, Atlanta, Georgia 30322.; the Department of Physiology and Functional Genomics, University of Florida College of Medicine, Gainesville, Florida 32610., Duke BJ; the Department of Physiology, Emory University, Atlanta, Georgia 30322., Al-Khalili O; the Department of Physiology, Emory University, Atlanta, Georgia 30322., Thai TL; the Department of Physiology, Emory University, Atlanta, Georgia 30322., Hamacher J; the Department of Pulmonology, Saarland University, D-66421 Homburg, Germany., Sridhar S; From the Vascular Biology Center., Lebedyeva I; the Department of Chemistry, Augusta University, Augusta, Georgia 30912., Su H; From the Vascular Biology Center., Tzotzos S; Apeptico Research and Development, 1150 Vienna, Austria., Fischer B; Apeptico Research and Development, 1150 Vienna, Austria., Gameiro AF; the Department of Chemistry, Augusta University, Augusta, Georgia 30912., Loose M; the Institute for Medical Microbiology, Justus-Liebig University, 35390 Giessen, Germany, and., Chakraborty T; the Institute for Medical Microbiology, Justus-Liebig University, 35390 Giessen, Germany, and., Shabbir W; the Department of Pharmacology and Toxicology, University Vienna, 1010 Vienna, Austria., Aufy M; the Department of Pharmacology and Toxicology, University Vienna, 1010 Vienna, Austria., Lemmens-Gruber R; the Department of Pharmacology and Toxicology, University Vienna, 1010 Vienna, Austria., Eaton DC; the Department of Physiology, Emory University, Atlanta, Georgia 30322, deaton@emory.edu., Czikora I; From the Vascular Biology Center, iczikora@augusta.edu.

Publikováno v: The Journal of biological chemistry [J Biol Chem] 2016 Nov 04; Vol. 291 (45), pp. 23440-23451. Date of Electronic Publication: 2016 Sep 19.